Conversion of metaplastic Barrett's epithelium into post-mitotic goblet cells by gamma-secretase inhibition.
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Signaling pathways in the molecular pathogenesis of adenocarcinomas of the esophagus and gastroesophageal junctionThe Origins of Gastric Cancer From Gastric Stem Cells: Lessons From Mouse Models.Hedgehog signaling regulates FOXA2 in esophageal embryogenesis and Barrett's metaplasia.Unexplored potentials of epigenetic mechanisms of plants and animals-theoretical considerations.Biology of Barrett's esophagus and esophageal adenocarcinomaNotch signaling modulates MUC16 biosynthesis in an in vitro model of human corneal and conjunctival epithelial cell differentiationA multiparametric approach to monitor the effects of γ-secretase inhibition along the whole intestinal tract.High Goblet Cell Count Is Inversely Associated with Ploidy Abnormalities and Risk of Adenocarcinoma in Barrett's Esophagus.Inhibition of Notch signaling enhances transdifferentiation of the esophageal squamous epithelium towards a Barrett's-like metaplasia via KLF4.Barrett esophagus: what a mouse model can teach us about human diseaseMolecular mechanisms of Barrett's esophagus.Barrett's esophagus: cancer and molecular biology.Mini-review: Does Notch promote or suppress cancer? New findings and old controversies.NOTCH Signaling and ATOH1 in Colorectal Cancers.Goblet Cell Ratio in Combination with Differentiation and Stem Cell Markers in Barrett Esophagus Allow Distinction of Patients with and without Esophageal Adenocarcinoma.Transcommitment: Paving the Way to Barrett's Metaplasia.Inflammation and Notch signaling: a crosstalk with opposite effects on tumorigenesisThe Barrett's Gland in Phenotype Space.Notch signaling pathway and Cdx2 expression in the development of Barrett's esophagus.Math1/Atoh1 contributes to intestinalization of esophageal keratinocytes by inducing the expression of Muc2 and Keratin-20.Risk prediction in Barrett's esophagus - aspects of a combination of molecular and epidemiologic biomarkers reflecting alterations of the microenvironment.Dysfunctional transforming growth factor-β signaling with constitutively active Notch signaling in Barrett's esophageal adenocarcinoma.NOTCH1 and NOTCH3 coordinate esophageal squamous differentiation through a CSL-dependent transcriptional network.Bile acid and inflammation activate gastric cardia stem cells in a mouse model of Barrett-like metaplasia.Bile acids induce Delta-like 1 expression via Cdx2-dependent pathway in the development of Barrett's esophagus.Notch Signaling Pathway Is Inhibited in the Development of Barrett's Esophagus: An In Vivo and In Vitro Study.
P2860
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P2860
Conversion of metaplastic Barrett's epithelium into post-mitotic goblet cells by gamma-secretase inhibition.
description
2010 nî lūn-bûn
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2010年の論文
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2010年学术文章
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2010年学术文章
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2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
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2010年學術文章
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2010年學術文章
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name
Conversion of metaplastic Barr ...... by gamma-secretase inhibition.
@en
Conversion of metaplastic Barr ...... by gamma-secretase inhibition.
@nl
type
label
Conversion of metaplastic Barr ...... by gamma-secretase inhibition.
@en
Conversion of metaplastic Barr ...... by gamma-secretase inhibition.
@nl
prefLabel
Conversion of metaplastic Barr ...... by gamma-secretase inhibition.
@en
Conversion of metaplastic Barr ...... by gamma-secretase inhibition.
@nl
P2093
P50
P356
P1476
Conversion of metaplastic Barr ...... by gamma-secretase inhibition.
@en
P2093
Hans Clevers
Johan H van Es
Johannes G Kusters
Peter D Siersema
Ron W F de Bruin
Vivianda Menke
Wim de Lau
P304
P356
10.1242/DMM.003012
P577
2010-01-01T00:00:00Z