Tumor necrosis factor-alpha antagonism protects from myocardial inflammation and fibrosis in experimental diabetic cardiomyopathy. - Wikidata - awgldk - TriplyDB

Tumor necrosis factor-alpha antagonism protects from myocardial inflammation and fibrosis in experimental diabetic cardiomyopathy.

Tumor necrosis factor-alpha antagonism protects from myocardial inflammation and fibrosis in experimental diabetic cardiomyopathy.

http://www.wikidata.org/entity/Q39815739

about

Diabetic Cardiovascular Disease Induced by Oxidative Stress Diabetic cardiomyopathy: bench to bedside Interplay of oxidative, nitrosative/nitrative stress, inflammation, cell death and autophagy in diabetic cardiomyopathy Antibody therapy can enhance AngiotensinII-induced myocardial fibrosis.The effect of Astragalus polysaccharides on attenuation of diabetic cardiomyopathy through inhibiting the extrinsic and intrinsic apoptotic pathways in high glucose -stimulated H9C2 cells.Serum albumin concentration and heart failure risk The Health, Aging, and Body Composition Study Prevention of vasa vasorum neovascularization attenuates early neointima formation in experimental hypercholesterolemia.Cannabidiol attenuates cardiac dysfunction, oxidative stress, fibrosis, and inflammatory and cell death signaling pathways in diabetic cardiomyopathy.Cardioprotective effects of tanshinone IIA pretreatment via kinin B2 receptor-Akt-GSK-3β dependent pathway in experimental diabetic cardiomyopathy Long-term in vivo resistin overexpression induces myocardial dysfunction and remodeling in rats.Genistein ameliorates cardiac inflammation and oxidative stress in streptozotocin-induced diabetic cardiomyopathy in rats.Anthrax lethal toxin suppresses high glucose induced VEGF over secretion through a post-translational mechanism.Inflammatory markers and incident heart failure risk in older adults: the Health ABC (Health, Aging, and Body Composition) study.Role of the innate immune system in acute viral myocarditis.Renal Denervation Findings on Cardiac and Renal Fibrosis in Rats with Isoproterenol Induced Cardiomyopathy.Diabetes-associated cardiac fibrosis: Cellular effectors, molecular mechanisms and therapeutic opportunities.Macrophages modulate cardiac function in lipotoxic cardiomyopathy.Anti-inflammatory effects of triptolide improve left ventricular function in a rat model of diabetic cardiomyopathy.Unique mechanistic insights into the beneficial effects of soluble epoxide hydrolase inhibitors in the prevention of cardiac fibrosis.Macrophage depletion suppresses sympathetic hyperinnervation following myocardial infarction.Low-dose radiation prevents type 1 diabetes-induced cardiomyopathy via activation of AKT mediated anti-apoptotic and anti-oxidant effects.Sitagliptin attenuates cardiomyopathy by modulating the JAK/STAT signaling pathway in experimental diabetic rats.Effects of cyclooxygenase-2 gene inactivation on cardiac autonomic and left ventricular function in experimental diabetes Gene deletion of the kinin receptor B1 attenuates cardiac inflammation and fibrosis during the development of experimental diabetic cardiomyopathy.The role of inflammatory cytokines in endothelial dysfunction Development of diabetic cardiomyopathy and the kallikrein-kinin system--new insights from B1 and B2 receptor signaling.Maturation-induces endothelial dysfunction via vascular inflammation in diabetic mice Oxidative stress in diabetes: implications for vascular and other complications.Magnolia extract (BL153) protection of heart from lipid accumulation caused cardiac oxidative damage, inflammation, and cell death in high-fat diet fed mice.Molecular mechanisms of diabetic cardiomyopathy.Oxidative stress: a key contributor to diabetic cardiomyopathy.Diabetic Cardiomyopathy: An Immunometabolic Perspective.Diabetes, oxidative stress, molecular mechanism, and cardiovascular disease--an overview.Do multiple nuclear factor kappa B activation mechanisms explain its varied effects in the heart?Role of tumor necrosis factor alpha in the pathogenesis of atrial fibrillation: A novel potential therapeutic target?Inflammation - Cause or Consequence of Heart Failure or Both?Pathophysiological Fundamentals of Diabetic Cardiomyopathy.An overview of the inflammatory signalling mechanisms in the myocardium underlying the development of diabetic cardiomyopathy.Inflammation and metabolic cardiomyopathy.IL-1β Inhibition in Cardiovascular Complications Associated to Diabetes Mellitus.

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Tumor necrosis factor-alpha antagonism protects from myocardial inflammation and fibrosis in experimental diabetic cardiomyopathy.

http://www.wikidata.org/entity/Q39815739

description

2007 nî lūn-bûn

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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2007年學術文章

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2007年學術文章

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name

Tumor necrosis factor-alpha an ...... ental diabetic cardiomyopathy.

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Tumor necrosis factor-alpha an ...... ental diabetic cardiomyopathy.

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type

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Tumor necrosis factor-alpha an ...... ental diabetic cardiomyopathy.

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Tumor necrosis factor-alpha an ...... ental diabetic cardiomyopathy.

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Tumor necrosis factor-alpha an ...... ental diabetic cardiomyopathy.

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Tumor necrosis factor-alpha an ...... ental diabetic cardiomyopathy.

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Basic Research in Cardiology

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Alexander Riad

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Andre Schmidt

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Carsten Tschöpe

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Dirk Westermann

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Sophie Van Linthout

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Xi-Yong Song

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Cardiac failure in transgenic mice with myocardial expression of tumor necrosis factor-alpha.

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s00395-007-0673-0

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500-507

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Michel Noutsias

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