Primary cilia modulate balance of canonical and non-canonical Wnt signaling responses in the injured kidney.
about
DNMT1 and HDAC2 Cooperate to Facilitate Aberrant Promoter Methylation in Inorganic Phosphate-Induced Endothelial-Mesenchymal Transition.Therapeutic targets for polycystic kidney disease.Primary Cilia and Coordination of Receptor Tyrosine Kinase (RTK) and Transforming Growth Factor β (TGF-β) Signaling.Progression of chronic kidney disease: too much cellular talk causes damage.(Pro)renin receptor (ATP6AP2) depletion arrests As4.1 cells in the G0/G1 phase thereby increasing formation of primary cilia.Olfactory loss and dysfunction in ciliopathies: Molecular mechanisms and potential therapies.Can Tissue Cilia Lengths and Urine Cilia Proteins Be Markers of Kidney Diseases?Genetic defects in ciliary genes in autosomal dominant polycystic kidney disease.
P2860
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P2860
Primary cilia modulate balance of canonical and non-canonical Wnt signaling responses in the injured kidney.
description
2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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name
Primary cilia modulate balance ...... sponses in the injured kidney.
@en
Primary cilia modulate balance ...... sponses in the injured kidney.
@nl
type
label
Primary cilia modulate balance ...... sponses in the injured kidney.
@en
Primary cilia modulate balance ...... sponses in the injured kidney.
@nl
prefLabel
Primary cilia modulate balance ...... sponses in the injured kidney.
@en
Primary cilia modulate balance ...... sponses in the injured kidney.
@nl
P2093
P2860
P1476
Primary cilia modulate balance ...... sponses in the injured kidney.
@en
P2093
Gerhard A Müller
Michael Zeisberg
Shoji Saito
P2860
P2888
P356
10.1186/S13069-015-0024-Y
P50
P577
2015-04-16T00:00:00Z
P5875
P6179
1024951204