Porphyromonas gingivalis lipopolysaccharide is poorly recognized by molecular components of innate host defense in a mouse model of early inflammation
about
Strain-dependent activation of monocytes and inflammatory macrophages by lipopolysaccharide of Porphyromonas gingivalisCell adhesion molecules in the pathogenesis of and host defence against microbial infection.Porphyromonas gingivalis GroEL induces osteoclastogenesis of periodontal ligament cells and enhances alveolar bone resorption in rats.Life below the gum line: pathogenic mechanisms of Porphyromonas gingivalis.Differential host response to LPS variants in amniochorion and the TLR4/MD-2 system in Macaca nemestrina.Porphyromonas gingivalis lipopolysaccharide is both agonist and antagonist for p38 mitogen-activated protein kinase activation.GRIM-19: A Double-edged Sword that Regulates Anti-Tumor and Innate Immune Responses.The hemoglobin receptor protein of porphyromonas gingivalis inhibits receptor activator NF-kappaB ligand-induced osteoclastogenesis from bone marrow macrophagesTetra- and penta-acylated lipid A structures of Porphyromonas gingivalis LPS differentially activate TLR4-mediated NF-κB signal transduction cascade and immuno-inflammatory response in human gingival fibroblastsPorphyromonas gingivalis lipopolysaccharide antagonizes Escherichia coli lipopolysaccharide at toll-like receptor 4 in human endothelial cells.Nucleotide sequence of the Porphyromonas gingivalis W83 recA homolog and construction of a recA-deficient mutantDifferential cytokine patterns in mouse macrophages and gingival fibroblasts after stimulation with porphyromonas gingivalis or Escherichia coli lipopolysaccharide.Association between Periodontopathogens and CRP Levels in Patients with Periodontitis in Serbia.The GroEL protein of Porphyromonas gingivalis regulates atherogenic phenomena in endothelial cells mediated by upregulating toll-like receptor 4 expression.Lipid A structural modifications in extreme conditions and identification of unique modifying enzymes to define the Toll-like receptor 4 structure-activity relationship.Escherichia coli msbB gene as a virulence factor and a therapeutic targetLocal chemokine paralysis, a novel pathogenic mechanism for Porphyromonas gingivalis.Bacterium-dependent induction of cytokines in mononuclear cells and their pathologic consequences in vivo.Identification of proteins differentially expressed in human monocytes exposed to Porphyromonas gingivalis and its purified components by high-throughput immunoblotting.Importance of heterogeneity in Porhyromonas gingivalis lipopolysaccharide lipid A in tissue specific inflammatory signalling.
P2860
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P2860
Porphyromonas gingivalis lipopolysaccharide is poorly recognized by molecular components of innate host defense in a mouse model of early inflammation
description
1995 nî lūn-bûn
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1995年の論文
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1995年学术文章
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1995年学术文章
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1995年学术文章
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1995年学术文章
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1995年学术文章
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1995年學術文章
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1995年學術文章
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name
Porphyromonas gingivalis lipop ...... se model of early inflammation
@en
Porphyromonas gingivalis lipop ...... e model of early inflammation.
@nl
type
label
Porphyromonas gingivalis lipop ...... se model of early inflammation
@en
Porphyromonas gingivalis lipop ...... e model of early inflammation.
@nl
prefLabel
Porphyromonas gingivalis lipop ...... se model of early inflammation
@en
Porphyromonas gingivalis lipop ...... e model of early inflammation.
@nl
P2093
P2860
P921
P1476
Porphyromonas gingivalis lipop ...... se model of early inflammation
@en
P2093
P2860
P304
P407
P577
1995-12-01T00:00:00Z