The Escherichia coli K-12 gntP gene allows E. coli F-18 to occupy a distinct nutritional niche in the streptomycin-treated mouse large intestine.
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What's for dinner?: Entner-Doudoroff metabolism in Escherichia coli.Estimation of growth rates of Escherichia coli BJ4 in streptomycin-treated and previously germfree mice by in situ rRNA hybridization.Carbon nutrition of Escherichia coli in the mouse intestine.rpoS gene function is a disadvantage for Escherichia coli BJ4 during competitive colonization of the mouse large intestine.Microbial imbalance and intestinal pathologies: connections and contributionsGntP is the Escherichia coli Fructuronic acid transporter and belongs to the UxuR regulon.Molecular genetic characterization of the Escherichia coli gntT gene of GntI, the main system for gluconate metabolism.There is a specific response to pH by isolates of Haemophilus influenzae and this has a direct influence on biofilm formationCommensal and Pathogenic Escherichia coli Metabolism in the Gut.L-fucose stimulates utilization of D-ribose by Escherichia coli MG1655 DeltafucAO and E. coli Nissle 1917 DeltafucAO mutants in the mouse intestine and in M9 minimal medium.Streptomycin-induced inflammation enhances Escherichia coli gut colonization through nitrate respiration.Host-Derived Sialic Acids Are an Important Nutrient Source Required for Optimal Bacterial Fitness In VivoA temperature-regulated Campylobacter jejuni gluconate dehydrogenase is involved in respiration-dependent energy conservation and chicken colonization.Sucrose metabolism contributes to in vivo fitness of Streptococcus pneumoniae.A functional cra gene is required for Salmonella enterica serovar typhimurium virulence in BALB/c mice.An Escherichia coli MG1655 lipopolysaccharide deep-rough core mutant grows and survives in mouse cecal mucus but fails to colonize the mouse large intestine.Escherichia coli F-18 and E. coli K-12 eda mutants do not colonize the streptomycin-treated mouse large intestineGut microbiota: Role in pathogen colonization, immune responses, and inflammatory disease.Mouse intestine selects nonmotile flhDC mutants of Escherichia coli MG1655 with increased colonizing ability and better utilization of carbon sources.Glycolytic and gluconeogenic growth of Escherichia coli O157:H7 (EDL933) and E. coli K-12 (MG1655) in the mouse intestine.The streptomycin-treated mouse intestine selects Escherichia coli envZ missense mutants that interact with dense and diverse intestinal microbiota.Role of motility and the flhDC Operon in Escherichia coli MG1655 colonization of the mouse intestineThe evolution of restraint in bacterial biofilms under nontransitive competition.Escherichia coli O157:H7 and other E. coli strains share physiological properties associated with intestinal colonization.Precolonized human commensal Escherichia coli strains serve as a barrier to E. coli O157:H7 growth in the streptomycin-treated mouse intestine.
P2860
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P2860
The Escherichia coli K-12 gntP gene allows E. coli F-18 to occupy a distinct nutritional niche in the streptomycin-treated mouse large intestine.
description
1996 nî lūn-bûn
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1996年の論文
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1996年学术文章
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1996年学术文章
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1996年学术文章
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1996年学术文章
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name
The Escherichia coli K-12 gntP ...... treated mouse large intestine.
@en
The Escherichia coli K-12 gntP ...... treated mouse large intestine.
@nl
type
label
The Escherichia coli K-12 gntP ...... treated mouse large intestine.
@en
The Escherichia coli K-12 gntP ...... treated mouse large intestine.
@nl
prefLabel
The Escherichia coli K-12 gntP ...... treated mouse large intestine.
@en
The Escherichia coli K-12 gntP ...... treated mouse large intestine.
@nl
P2093
P2860
P1476
The Escherichia coli K-12 gntP ...... treated mouse large intestine.
@en
P2093
McCormick BA
Moller-Nielsen E
Schembri MA
Sweeney NJ
P2860
P304
P407
P577
1996-09-01T00:00:00Z