Group B streptococcal beta-hemolysin/cytolysin activates neutrophil signaling pathways in brain endothelium and contributes to development of meningitis.
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Defense at the border: the blood-brain barrier versus bacterial foreignersPathogenic Triad in Bacterial Meningitis: Pathogen Invasion, NF-κB Activation, and Leukocyte Transmigration that Occur at the Blood-Brain BarrierInflammasome/IL-1β Responses to Streptococcal PathogensPathogens penetrating the central nervous system: infection pathways and the cellular and molecular mechanisms of invasionStructure ofStreptococcus agalactiaetip pilin GBS104: a model for GBS pili assembly and host interactionsCharacterization of Fibrinogen Binding by Glycoproteins Srr1 and Srr2 of Streptococcus agalactiaeHost-pathogen interactions in bacterial meningitisUnderstanding the regulation of Group B Streptococcal virulence factorsA streptococcal lipid toxin induces membrane permeabilization and pyroptosis leading to fetal injuryPertussis toxin transiently affects barrier integrity, organelle organization and transmigration of monocytes in a human brain microvascular endothelial cell barrier modelRecent advances in understanding the molecular basis of group B Streptococcus virulencePathogenesis of Streptococcus urinary tract infection depends on bacterial strain and β-hemolysin/cytolysin that mediates cytotoxicity, cytokine synthesis, inflammation and virulenceThreonine phosphorylation prevents promoter DNA binding of the Group B Streptococcus response regulator CovR.Regulation of CovR expression in Group B Streptococcus impacts blood-brain barrier penetration.Gene expression in cortex and hippocampus during acute pneumococcal meningitisBlood-brain barrier invasion by group B Streptococcus depends upon proper cell-surface anchoring of lipoteichoic acid.Group B streptococcal beta-hemolysin/cytolysin directly impairs cardiomyocyte viability and functionAnthrax toxins inhibit neutrophil signaling pathways in brain endothelium and contribute to the pathogenesis of meningitisIdentification of a group B streptococcal fibronectin binding protein, SfbA, that contributes to invasion of brain endothelium and development of meningitis.Induction and termination of inflammatory signaling in group B streptococcal sepsis.Impact of host age and parity on susceptibility to severe urinary tract infection in a murine model.Correlation between Group B Streptococcal Genotypes, Their Antimicrobial Resistance Profiles, and Virulence Genes among Pregnant Women in Lebanon.Analysis of two-component systems in group B Streptococcus shows that RgfAC and the novel FspSR modulate virulence and bacterial fitness.Dynamic changes in Mcl-1 expression regulate macrophage viability or commitment to apoptosis during bacterial clearanceSurface proteins of Streptococcus agalactiae and related proteins in other bacterial pathogens.Group B Streptococcus β-hemolysin/cytolysin breaches maternal-fetal barriers to cause preterm birth and intrauterine fetal demise in vivoThe GBS PI-2a pilus is required for virulence in mice neonates.Adherence to and invasion of human brain microvascular endothelial cells are promoted by fibrinogen-binding protein FbsA of Streptococcus agalactiae.Glycosaminoglycan binding facilitates entry of a bacterial pathogen into central nervous systemsActivation of brain endothelium by pneumococcal neuraminidase NanA promotes bacterial internalization.Glucosyltransferases of viridans group streptococci modulate interleukin-6 and adhesion molecule expression in endothelial cells and augment monocytic cell adherence.Role of pore-forming toxins in bacterial infectious diseases.Group B Streptococcus interactions with human meningeal cells and astrocytes in vitro.Interaction of Streptococcus agalactiae and Cellular Innate Immunity in Colonization and DiseaseBinding of glycoprotein Srr1 of Streptococcus agalactiae to fibrinogen promotes attachment to brain endothelium and the development of meningitis.Gene expression pattern in human brain endothelial cells in response to Neisseria meningitidisThe β-hemolysin and intracellular survival of Streptococcus agalactiae in human macrophagesInteraction of neonatal phagocytes with group B streptococcus: recognition and response.Infection of human coronary artery endothelial cells by group B streptococcus contributes to dysregulation of apoptosis, hemostasis, and innate immune responsesGroup B Streptococcus hijacks the host plasminogen system to promote brain endothelial cell invasion
P2860
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P2860
Group B streptococcal beta-hemolysin/cytolysin activates neutrophil signaling pathways in brain endothelium and contributes to development of meningitis.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Group B streptococcal beta-hem ...... to development of meningitis.
@en
Group B streptococcal beta-hem ...... to development of meningitis.
@nl
type
label
Group B streptococcal beta-hem ...... to development of meningitis.
@en
Group B streptococcal beta-hem ...... to development of meningitis.
@nl
prefLabel
Group B streptococcal beta-hem ...... to development of meningitis.
@en
Group B streptococcal beta-hem ...... to development of meningitis.
@nl
P2860
P356
P1476
Group B streptococcal beta-hem ...... to development of meningitis.
@en
P2093
George Y Liu
Kelly S Doran
P2860
P304
P356
10.1172/JCI200317335
P407
P50
P577
2003-09-01T00:00:00Z