Sequence identity of the terminal redundancies on the minus-strand DNA template is necessary but not sufficient for the template switch during hepadnavirus plus-strand DNA synthesis
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Mechanism for CCC DNA synthesis in hepadnaviruses.Effects of genomic length on translocation of hepatitis B virus polymerase-linked oligomer.Infection of ducklings with virus particles containing linear double-stranded duck hepatitis B virus DNA: illegitimate replication and reversionIdentification of an essential molecular contact point on the duck hepatitis B virus reverse transcriptaseA novel cis-acting element facilitates minus-strand DNA synthesis during reverse transcription of the hepatitis B virus genome.Base pairing among three cis-acting sequences contributes to template switching during hepadnavirus reverse transcription.cis-Acting sequences 5E, M, and 3E interact to contribute to primer translocation and circularization during reverse transcription of avian hepadnavirus DNAThree novel cis-acting elements required for efficient plus-strand DNA synthesis of the hepatitis B virus genome.Complementarity between epsilon and phi sequences in pregenomic RNA influences hepatitis B virus replication efficiency.The conformation of the 3' end of the minus-strand DNA makes multiple contributions to template switches during plus-strand DNA synthesis of duck hepatitis B virus.Template switches during plus-strand DNA synthesis of duck hepatitis B virus are influenced by the base composition of the minus-strand terminal redundancy.Small DNA hairpin negatively regulates in situ priming during duck hepatitis B virus reverse transcription.Changing the site of initiation of plus-strand DNA synthesis inhibits the subsequent template switch during replication of a hepadnavirus.The binding site of transcription factor YY1 is required for intramolecular recombination between terminally repeated sequences of linear replicative hepatitis B virus DNA.Mutations that increase in situ priming also decrease circularization for duck hepatitis B virus.Analysis of duck hepatitis B virus reverse transcription indicates a common mechanism for the two template switches during plus-strand DNA synthesis.cis-Acting sequences in addition to donor and acceptor sites are required for template switching during synthesis of plus-strand DNA for duck hepatitis B virus.Insertions within epsilon affect synthesis of minus-strand DNA before the template switch for duck hepatitis B virusThe sequence of the RNA primer and the DNA template influence the initiation of plus-strand DNA synthesis in hepatitis B virus.cis-Acting sequences that contribute to the synthesis of relaxed-circular DNA of human hepatitis B virus.
P2860
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P2860
Sequence identity of the terminal redundancies on the minus-strand DNA template is necessary but not sufficient for the template switch during hepadnavirus plus-strand DNA synthesis
description
1997 nî lūn-bûn
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1997年の論文
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1997年学术文章
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1997年学术文章
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1997年学术文章
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name
Sequence identity of the termi ...... irus plus-strand DNA synthesis
@en
Sequence identity of the termi ...... rus plus-strand DNA synthesis.
@nl
type
label
Sequence identity of the termi ...... irus plus-strand DNA synthesis
@en
Sequence identity of the termi ...... rus plus-strand DNA synthesis.
@nl
prefLabel
Sequence identity of the termi ...... irus plus-strand DNA synthesis
@en
Sequence identity of the termi ...... rus plus-strand DNA synthesis.
@nl
P2093
P2860
P1433
P1476
Sequence identity of the termi ...... irus plus-strand DNA synthesis
@en
P2093
P2860
P304
P407
P577
1997-01-01T00:00:00Z