Second-site proviral enhancer alterations in lymphomas induced by enhancer mutants of SL3-3 murine leukemia virus: negative effect of nuclear factor 1 binding site.
about
Identification of novel Bach2 transcripts and protein isoforms through tagging analysis of retroviral integrations in B-cell lymphomas.Tandemization of a subregion of the enhancer sequences from SRS 19-6 murine leukemia virus associated with T-lymphoid but not other leukemias.Gene expression profiling of murine T-cell lymphoblastic lymphoma identifies deregulation of S-phase initiating genes.In vivo footprinting of the enhancer sequences in the upstream long terminal repeat of Moloney murine leukemia virus: differential binding of nuclear factors in different cell typesCore-binding factor influences the disease specificity of Moloney murine leukemia virusIncreased induction of osteopetrosis, but unaltered lymphomagenicity, by murine leukemia virus SL3-3 after mutation of a nuclear factor 1 site in the enhancerIkaros, a lymphoid-cell-specific transcription factor, contributes to the leukemogenic phenotype of a mink cell focus-inducing murine leukemia virusStability of AML1 (core) site enhancer mutations in T lymphomas induced by attenuated SL3-3 murine leukemia virus mutants.Increased lymphomagenicity and restored disease specificity of AML1 site (core) mutant SL3-3 murine leukemia virus by a second-site enhancer variant evolved in vivo.An SL3-3 murine leukemia virus enhancer variant more pathogenic than the wild type obtained by assisted molecular evolution in vivoControl of pathogenicity and disease specificity of a T-lymphomagenic gammaretrovirus by E-box motifs but not by an overlapping glucocorticoid response elementLoss of MicroRNA targets in the 3' untranslated region as a mechanism of retroviral insertional activation of growth factor independence 1.Activation of the brain-specific neurogranin gene in murine T-cell lymphomas by proviral insertional mutagenesis.Mutation of all Runx (AML1/core) sites in the enhancer of T-lymphomagenic SL3-3 murine leukemia virus unmasks a significant potential for myeloid leukemia induction and favors enhancer evolution toward induction of other disease patterns.Analysis of wild-type and mutant SL3-3 murine leukemia virus insertions in the c-myc promoter during lymphomagenesis reveals target site hot spots, virus-dependent patterns, and frequent error-prone gap repair.Selection of reversions and suppressors of a mutation in the CBF binding site of a lymphomagenic retrovirusSuppressor mutations within the core binding factor (CBF/AML1) binding site of a T-cell lymphomagenic retrovirus.CBF, Myb, and Ets binding sites are important for activity of the core I element of the murine retrovirus SL3-3 in T lymphocytes.B-Cell lymphoma induction by akv murine leukemia viruses harboring one or both copies of the tandem repeat in the U3 enhancer.
P2860
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P2860
Second-site proviral enhancer alterations in lymphomas induced by enhancer mutants of SL3-3 murine leukemia virus: negative effect of nuclear factor 1 binding site.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
1997年學術文章
@zh
1997年學術文章
@zh-hant
name
Second-site proviral enhancer ...... nuclear factor 1 binding site.
@en
Second-site proviral enhancer ...... nuclear factor 1 binding site.
@nl
type
label
Second-site proviral enhancer ...... nuclear factor 1 binding site.
@en
Second-site proviral enhancer ...... nuclear factor 1 binding site.
@nl
prefLabel
Second-site proviral enhancer ...... nuclear factor 1 binding site.
@en
Second-site proviral enhancer ...... nuclear factor 1 binding site.
@nl
P2093
P2860
P1433
P1476
Second-site proviral enhancer ...... nuclear factor 1 binding site.
@en
P2093
Ethelberg S
Grundström T
Hallberg B
Pedersen FS
P2860
P304
P407
P577
1997-02-01T00:00:00Z