Early matrix metalloproteinase-12 inhibition worsens post-myocardial infarction cardiac dysfunction by delaying inflammation resolution.
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Synthesis and in Vitro and in Vivo Evaluation of MMP-12 Selective Optical ProbesCD36 Is a Matrix Metalloproteinase-9 Substrate That Stimulates Neutrophil Apoptosis and Removal During Cardiac Remodeling.The crossroads of inflammation, fibrosis, and arrhythmia following myocardial infarctionMatrix metalloproteinases as input and output signals for post-myocardial infarction remodelingTemporal neutrophil polarization following myocardial infarctionUsing the laws of thermodynamics to understand how matrix metalloproteinases coordinate the myocardial response to injuryLeukocyte diversity in resolving and nonresolving mechanisms of cardiac remodeling.Matrix Metalloproteinases in Myocardial Infarction and Heart Failure.Can heart function lost to disease be regenerated by therapeutic targeting of cardiac scar tissue?Aging is protective against pressure overload cardiomyopathy via adaptive extracellular matrix remodeling.Optical imaging of MMP-12 active form in inflammation and aneurysm.Defining the sham environment for post-myocardial infarction studies in miceIL-10 improves cardiac remodeling after myocardial infarction by stimulating M2 macrophage polarization and fibroblast activation.Early matrix metalloproteinase-9 inhibition post-myocardial infarction worsens cardiac dysfunction by delaying inflammation resolution.PARP-1 promotes tumor recurrence after warm ischemic liver graft transplantation via neutrophil recruitment and polarization.Proteomic analysis of the cardiac extracellular matrix: clinical research applications.Macrophage overexpression of matrix metalloproteinase-9 in aged mice improves diastolic physiology and cardiac wound healing after myocardial infarction.Understanding cardiac extracellular matrix remodeling to develop biomarkers of myocardial infarction outcomes.Guidelines for Experimental Models of Myocardial Ischemia and Infarction.Mapping macrophage polarization over the myocardial infarction time continuum.Expression of adhesion molecules on granulocytes and monocytes following myocardial infarction in rats drinking white wine.LXR/RXR signaling and neutrophil phenotype following myocardial infarction classify sex differences in remodeling
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P2860
Early matrix metalloproteinase-12 inhibition worsens post-myocardial infarction cardiac dysfunction by delaying inflammation resolution.
description
2015 nî lūn-bûn
@nan
2015年の論文
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2015年学术文章
@wuu
2015年学术文章
@zh-cn
2015年学术文章
@zh-hans
2015年学术文章
@zh-my
2015年学术文章
@zh-sg
2015年學術文章
@yue
2015年學術文章
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2015年學術文章
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name
Early matrix metalloproteinase ...... aying inflammation resolution.
@en
Early matrix metalloproteinase ...... aying inflammation resolution.
@nl
type
label
Early matrix metalloproteinase ...... aying inflammation resolution.
@en
Early matrix metalloproteinase ...... aying inflammation resolution.
@nl
prefLabel
Early matrix metalloproteinase ...... aying inflammation resolution.
@en
Early matrix metalloproteinase ...... aying inflammation resolution.
@nl
P2093
P2860
P1476
Early matrix metalloproteinase ...... aying inflammation resolution.
@en
P2093
Fouad A Zouein
Nicolle L Patterson
Rugmani Padmanabhan Iyer
Vincent Dive
Yonggang Ma
P2860
P304
P356
10.1016/J.IJCARD.2015.03.054
P407
P577
2015-03-05T00:00:00Z