Inhibition of SOC/Ca2+/NFAT pathway is involved in the anti-proliferative effect of sildenafil on pulmonary artery smooth muscle cells. - Wikidata - awgldk - TriplyDB

Inhibition of SOC/Ca2+/NFAT pathway is involved in the anti-proliferative effect of sildenafil on pulmonary artery smooth muscle cells.

Inhibition of SOC/Ca2+/NFAT pathway is involved in the anti-proliferative effect of sildenafil on pulmonary artery smooth muscle cells.

http://www.wikidata.org/entity/Q39911609

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TRP-ing up heart and vessels: canonical transient receptor potential channels and cardiovascular disease The protective effects of PCPA against monocrotaline-induced pulmonary arterial hypertension are mediated through the downregulation of NFAT-1 and NF-κB The Role of Transient Receptor Potential Channel 6 Channels in the Pulmonary Vasculature MicroRNA-124 suppresses the transactivation of nuclear factor of activated T cells by targeting multiple genes and inhibits the proliferation of pulmonary artery smooth muscle cells Chronic hypoxia increases TRPC6 expression and basal intracellular Ca2+ concentration in rat distal pulmonary venous smooth muscle L type Ca²+ channel blockers prevent oxaliplatin-induced cold hyperalgesia and TRPM8 overexpression in rats Chronic hypoxia upregulates pulmonary arterial ASIC1: a novel mechanism of enhanced store-operated Ca2+ entry and receptor-dependent vasoconstriction New mechanisms of pulmonary arterial hypertension: role of Ca²⁺ signaling.Nuclear factor of activated T-cell isoform expression and regulation in human myometrium Altered vasoreactivity in neonatal rats with pulmonary hypertension associated with bronchopulmonary dysplasia: Implication of both eNOS phosphorylation and calcium signaling Pathogenic role of store-operated and receptor-operated ca(2+) channels in pulmonary arterial hypertension.Regulation of ca(2+) signaling in pulmonary hypertension.Silencing of STIM1 attenuates hypoxia-induced PASMCs proliferation via inhibition of the SOC/Ca2+/NFAT pathway The Sur1-Trpm4 channel regulates NOS2 transcription in TLR4-activated microglia.Ginsenoside Rg1 attenuates hypoxia and hypercapnia-induced vasoconstriction in isolated rat pulmonary arterial rings by reducing the expression of p38.ASIC1-mediated calcium entry stimulates NFATc3 nuclear translocation via PICK1 coupling in pulmonary arterial smooth muscle cells Gravitational force modulates G2/M phase exit in mechanically unloaded myoblasts.Intermittent hypoxia-induced increases in reactive oxygen species activate NFATc3 increasing endothelin-1 vasoconstrictor reactivity.New therapies for pulmonary arterial hypertension: an update on current bench to bedside translation.Regulation of cardiovascular TRP channel functions along the NO-cGMP-PKG axis.Emerging roles for native Orai Ca2+ channels in cardiovascular disease Molecular pathogenesis and current pathology of pulmonary hypertension.The role of nuclear factor of activated T cells in pulmonary arterial hypertension.Dysfunction of volume-sensitive chloride channels contributes to cisplatin resistance in human lung adenocarcinoma cells.Inhibition of phosphodiesterase-5 suppresses calcineurin/NFAT- mediated TRPC6 expression in pulmonary artery smooth muscle cells Effect of hypoxia on TRPV1 and TRPV4 channels in rat pulmonary arterial smooth muscle cells.Store-operated channels in the pulmonary circulation of high- and low-altitude neonatal lambs.Notoginsenoside R1 attenuates hypoxia and hypercapnia-induced vasoconstriction in isolated rat pulmonary arterial rings by reducing the expression of ERK.Calcium influx and postsynaptic proteins coordinate the dendritic filopodium-spine transition.Gene therapy for pulmonary arterial hypertension: is a cure in sight?Impedance flow cytometry gauges proliferative capacity by detecting TRPC1 expression.NFATc3 pathway participates in the process that 15-LO/15-HETE protects pulmonary artery smooth muscle cells against apoptosis during hypoxia.mTORC1 is involved in hypoxia-induced pulmonary hypertension through the activation of Notch3.Revisiting the Role of TRP, Orai, and ASIC Channels in the Pulmonary Arterial Response to Hypoxia.

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Inhibition of SOC/Ca2+/NFAT pathway is involved in the anti-proliferative effect of sildenafil on pulmonary artery smooth muscle cells.

http://www.wikidata.org/entity/Q39911609

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2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年學術文章

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2009年學術文章

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2009年學術文章

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name

Inhibition of SOC/Ca2+/NFAT pa ...... ry artery smooth muscle cells.

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Inhibition of SOC/Ca2+/NFAT pa ...... ry artery smooth muscle cells.

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type

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Inhibition of SOC/Ca2+/NFAT pa ...... ry artery smooth muscle cells.

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Inhibition of SOC/Ca2+/NFAT pa ...... ry artery smooth muscle cells.

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Inhibition of SOC/Ca2+/NFAT pa ...... ry artery smooth muscle cells.

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Inhibition of SOC/Ca2+/NFAT pa ...... ry artery smooth muscle cells.

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Chen Wang

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Cong Wang

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Ji-Feng Li

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Jie Liu

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Jun Wan

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Jun Wang

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Lan Zhao

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Yue Xiu Wang

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P2860

A new generation of Ca2+ indicators with greatly improved fluorescence properties

Transcription factors of the NFAT family: regulation and function

NFAT proteins: key regulators of T-cell development and function

NFATc3 mediates chronic hypoxia-induced pulmonary arterial remodeling with alpha-actin up-regulation

The nuclear factor of activated T cells in pulmonary arterial hypertension can be therapeutically targeted

Mouse trp2, the homologue of the human trpc2 pseudogene, encodes mTrp2, a store depletion-activated capacitative Ca2+ entry channel

Store-operated calcium channels

Transcriptional regulation by calcium, calcineurin, and NFAT

Upregulated TRP and enhanced capacitative Ca(2+) entry in human pulmonary artery myocytes during proliferation.

Inhibition of calcineurin-NFAT hypertrophy signaling by cGMP-dependent protein kinase type I in cardiac myocytes.

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