Increased levels of superoxide and H2O2 mediate the differential susceptibility of cancer cells versus normal cells to glucose deprivation. - Wikidata - awgldk - TriplyDB

Increased levels of superoxide and H2O2 mediate the differential susceptibility of cancer cells versus normal cells to glucose deprivation.

Increased levels of superoxide and H2O2 mediate the differential susceptibility of cancer cells versus normal cells to glucose deprivation.

http://www.wikidata.org/entity/Q39927250

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TIGAR regulates DNA damage and repair through pentosephosphate pathway and Cdk5-ATM pathway SIRT3 is a mitochondria-localized tumor suppressor required for maintenance of mitochondrial integrity and metabolism during stress Redox-directed cancer therapeutics: molecular mechanisms and opportunities A new view of carcinogenesis and an alternative approach to cancer therapy Redox regulation of FoxO transcription factors Depolarization Controls TRAIL-Sensitization and Tumor-Selective Killing of Cancer Cells: Crosstalk with ROS Oxidative stress induces mitochondrial DNA damage and cytotoxicity through independent mechanisms in human cancer cells Increasing Superoxide Production and the Labile Iron Pool in Tumor Cells may Sensitize Them to Extracellular Ascorbate Dietary and pharmacological modification of the insulin/IGF-1 system: exploiting the full repertoire against cancer ROS homeostasis and metabolism: a dangerous liason in cancer cells Promoter hypermethylation mediated downregulation of FBP1 in human hepatocellular carcinoma and colon cancer Regulation of pancreatic cancer growth by superoxide Redox environment in stem and differentiated cells: A quantitative approach.Mitochondrial calcium uniporter activity is dispensable for MDA-MB-231 breast carcinoma cell survival Paclitaxel combined with inhibitors of glucose and hydroperoxide metabolism enhances breast cancer cell killing via H2O2-mediated oxidative stress.Peroxyoxalate Chemiluminescent Reaction as a Tool for Elimination of Tumour Cells Under Oxidative Stress.2-Deoxy-D-glucose treatment of endothelial cells induces autophagy by reactive oxygen species-mediated activation of the AMP-activated protein kinase.Sirt3, mitochondrial ROS, ageing, and carcinogenesis.The ketogenic diet reverses gene expression patterns and reduces reactive oxygen species levels when used as an adjuvant therapy for glioma.Tumor bioenergetics: an emerging avenue for cancer metabolism targeted therapy Thioredoxin-1 redox signaling regulates cell survival in response to hyperoxia.Restricting carbohydrates to fight head and neck cancer-is this realistic?Reactive oxygen species in normal and tumor stem cells.Phosphorylation and subcellular localization of p27Kip1 regulated by hydrogen peroxide modulation in cancer cells Ketogenic diets as an adjuvant cancer therapy: History and potential mechanism Ketogenic diets: from cancer to mitochondrial diseases and beyond.Implication of snail in metabolic stress-induced necrosis.The ketogenic diet and hyperbaric oxygen therapy prolong survival in mice with systemic metastatic cancer.Sensitivity to low-dose/low-LET ionizing radiation in mammalian cells harboring mutations in succinate dehydrogenase subunit C is governed by mitochondria-derived reactive oxygen species Copper-zinc superoxide dismutase-mediated redox regulation of bortezomib resistance in multiple myeloma.Combined inhibition of glycolysis, the pentose cycle, and thioredoxin metabolism selectively increases cytotoxicity and oxidative stress in human breast and prostate cancer NAC, tiron and trolox impair survival of cell cultures containing glioblastoma tumorigenic initiating cells by inhibition of cell cycle progression.The Warburg effect: evolving interpretations of an established concept Homeobox gene Dlx-2 is implicated in metabolic stress-induced necrosis.Enhancement of carboplatin-mediated lung cancer cell killing by simultaneous disruption of glutathione and thioredoxin metabolism.Relationship between NADPH and Th1/Th2 ratio in patients with non-Hodgkin lymphoma who have been exposed to pesticides.Biphasic activity of chloroquine in human colorectal cancer cells.Relationship between expression of NADPH oxidase 2 and invasion and prognosis of human gastric cancer Inhibition of Neuroblastoma Tumor Growth by Ketogenic Diet and/or Calorie Restriction in a CD1-Nu Mouse Model.Non-Toxic Metabolic Management of Metastatic Cancer in VM Mice: Novel Combination of Ketogenic Diet, Ketone Supplementation, and Hyperbaric Oxygen Therapy.

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P2860

Increased levels of superoxide and H2O2 mediate the differential susceptibility of cancer cells versus normal cells to glucose deprivation.

http://www.wikidata.org/entity/Q39927250

description

2009 nî lūn-bûn

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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2009年论文

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2009年论文

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name

Increased levels of superoxide ...... cells to glucose deprivation.

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Increased levels of superoxide ...... cells to glucose deprivation.

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type

label

Increased levels of superoxide ...... cells to glucose deprivation.

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Increased levels of superoxide ...... cells to glucose deprivation.

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prefLabel

Increased levels of superoxide ...... cells to glucose deprivation.

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Increased levels of superoxide ...... cells to glucose deprivation.

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Biochemical Journal

P1476

Increased levels of superoxide ...... cells to glucose deprivation.

@en

P2093

Douglas R Spitz

http://www.w3.org/2001/XMLSchema#string

Iman M Ahmad

http://www.w3.org/2001/XMLSchema#string

Larry W Oberley

http://www.w3.org/2001/XMLSchema#string

Nùkhet Aykin-Burns

http://www.w3.org/2001/XMLSchema#string

Yueming Zhu

http://www.w3.org/2001/XMLSchema#string

P2860

Protein measurement with the Folin phenol reagent

On the Origin of Cancer Cells

Facile detection of mitochondrial DNA mutations in tumors and bodily fluids

H(2)O(2)-induced O(2) production by a non-phagocytic NAD(P)H oxidase causes oxidant injury

Catalase in vitro

Production of large amounts of hydrogen peroxide by human tumor cells

Attenuation of LDH-A expression uncovers a link between glycolysis, mitochondrial physiology, and tumor maintenance

Cytotoxicity and metabolism of 4-hydroxy-2-nonenal and 2-nonenal in H2O2-resistant cell lines. Do aldehydic by-products of lipid peroxidation contribute to oxidative stress?

Glucose deprivation-induced oxidative stress in human tumor cells. A fundamental defect in metabolism?

Mitochondrial DNA in human malignancy.

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29-37

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10.1042/BJ20081258

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1

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418

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18937644

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