Induction of apoptosis by cigarette smoke via ROS-dependent endoplasmic reticulum stress and CCAAT/enhancer-binding protein-homologous protein (CHOP).
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Interactive effect of cigarette smoke extract and World Trade Center dust particles on airway cell cytotoxicityNrf2 expression is increased in peripheral blood mononuclear cells derived from mild-moderate ex-smoker COPD patients with persistent oxidative stressQuantitative Profiling of Protein S-Glutathionylation Reveals Redox-Dependent Regulation of Macrophage Function during Nanoparticle-Induced Oxidative StressSelenoprotein N deficiency in mice is associated with abnormal lung developmentAberrant, differential and bidirectional regulation of the unfolded protein response towards cell survival by 3'-deoxyadenosineFrom structure to redox: The diverse functional roles of disulfides and implications in disease.β-lapachone significantly increases the effect of ionizing radiation to cause mitochondrial apoptosis via JNK activation in cancer cells.Heightened endoplasmic reticulum stress in the lungs of patients with chronic obstructive pulmonary disease: the role of Nrf2-regulated proteasomal activity.Precursor of advanced glycation end products mediates ER-stress-induced caspase-3 activation of human dermal fibroblasts through NAD(P)H oxidase 4Mitochondrial thioredoxin reductase regulates major cytotoxicity pathways of proteasome inhibitors in multiple myeloma cells.Thrombin increases lung fibroblast survival while promoting alveolar epithelial cell apoptosis via the endoplasmic reticulum stress marker, CCAAT enhancer-binding homologous proteinAkt kinase mediates the prosurvival effect of smoking compounds in pancreatic ductal cells.Cigarette smoking promotes inflammation in patients with COPD by affecting the polarization and survival of Th/Tregs through up-regulation of muscarinic receptor 3 and 5 expressionResveratrol exerts an anti-apoptotic effect on human bronchial epithelial cells undergoing cigarette smoke exposure.Induction of the unfolded protein response by cigarette smoke is primarily an activating transcription factor 4-C/EBP homologous protein mediated processCritical role of proteostasis-imbalance in pathogenesis of COPD and severe emphysemaActivation of the UPR protects against cigarette smoke-induced RPE apoptosis through up-regulation of Nrf2.Glucosamine induces cell death via proteasome inhibition in human ALVA41 prostate cancer cell.Ghrelin effects on neuropeptides in the rat hypothalamus depend on fatty acid metabolism actions on BSX but not on gender.Genetic ablation of the aryl hydrocarbon receptor causes cigarette smoke-induced mitochondrial dysfunction and apoptosisChronic SIV and morphine treatment increases heat shock protein 5 expression at the synapse.Ketamine-induced ulcerative cystitis and bladder apoptosis involve oxidative stress mediated by mitochondria and the endoplasmic reticulum.Effects of Cigarette Smoke Condensate on Oxidative Stress, Apoptotic Cell Death, and HIV Replication in Human Monocytic Cells.Screening and Characterization of Drugs That Protect Corneal Endothelial Cells Against Unfolded Protein Response and Oxidative StressAirway Exposure to E-Cigarette Vapors Impairs Autophagy and Induces Aggresome Formation.Selenoprotein R Protects Human Lens Epithelial Cells against D-Galactose-Induced Apoptosis by Regulating Oxidative Stress and Endoplasmic Reticulum StressAdverse Outcomes Associated with Cigarette Smoke Radicals Related to Damage to Protein-disulfide IsomeraseEndoplasmic reticulum stress-mediated hippocampal neuron apoptosis involved in diabetic cognitive impairment.Hyperoxia and interferon-γ-induced injury in developing lungs occur via cyclooxygenase-2 and the endoplasmic reticulum stress-dependent pathway.Mechanisms of radiation toxicity in transformed and non-transformed cellsIdentification of Target Genes Involved in the Antiproliferative Effect of Enzyme-Modified Ginseng Extract in HepG2 Hepatocarcinoma Cell.HIV protease inhibitor lopinavir-induced TNF-alpha and IL-6 expression is coupled to the unfolded protein response and ERK signaling pathways in macrophages.Alcoholic lung injury: metabolic, biochemical and immunological aspects.Cigarette smoke reversibly activates hypoxia-inducible factor 1 in a reactive oxygen species-dependent manner.Endoplasmic reticulum stress in bone marrow-derived cells prevents acute cardiac inflammation and injury in response to angiotensin II.Glutathione Peroxidase-1 Suppresses the Unfolded Protein Response upon Cigarette Smoke Exposure.Therapeutic potential of traditional chinese medicine on inflammatory diseasesOxidative stress induces mitochondrial dysfunction and a protective unfolded protein response in RPE cells.Genetics of childhood-onset inflammatory bowel disease.The unfolded protein response in lung disease.
P2860
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P2860
Induction of apoptosis by cigarette smoke via ROS-dependent endoplasmic reticulum stress and CCAAT/enhancer-binding protein-homologous protein (CHOP).
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Induction of apoptosis by ciga ...... ein-homologous protein (CHOP).
@en
Induction of apoptosis by ciga ...... ing protein-homologous protein
@nl
type
label
Induction of apoptosis by ciga ...... ein-homologous protein (CHOP).
@en
Induction of apoptosis by ciga ...... ing protein-homologous protein
@nl
prefLabel
Induction of apoptosis by ciga ...... ein-homologous protein (CHOP).
@en
Induction of apoptosis by ciga ...... ing protein-homologous protein
@nl
P2093
P1476
Induction of apoptosis by ciga ...... tein-homologous protein (CHOP)
@en
P2093
Ayumi Kasai
Maro Okamura
Masanori Kitamura
Nobuhiko Hiramatsu
Yasuhiro Tagawa
P356
10.1016/J.FREERADBIOMED.2008.03.003
P577
2008-03-18T00:00:00Z