BMP-2 decreases Mash1 stability by increasing Id1 expression. - Wikidata - awgldk - TriplyDB

BMP-2 decreases Mash1 stability by increasing Id1 expression.

BMP-2 decreases Mash1 stability by increasing Id1 expression.

http://www.wikidata.org/entity/Q39996530

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Transcriptional control of vertebrate neurogenesis by the proneural factor Ascl1 MASH1/Ascl1a leads to GAP43 expression and axon regeneration in the adult CNS The p38/MK2/Hsp25 pathway is required for BMP-2-induced cell migration Disruption of CK2beta in embryonic neural stem cells compromises proliferation and oligodendrogenesis in the mouse telencephalon Angiopoietin 2 mediates the differentiation and migration of neural progenitor cells in the subventricular zone after stroke.Discovery of transcriptional regulators and signaling pathways in the developing pituitary gland by bioinformatic and genomic approaches.Increased stathmin1 expression in the dentate gyrus of mice causes abnormal axonal arborizations The p200 family protein p204 as a modulator of cell proliferation and differentiation: a brief survey Modelling the Delta1/Notch1 pathway: in search of the mediator(s) of neural stem cell differentiation.E-proteins orchestrate the progression of neural stem cell differentiation in the postnatal forebrain.Protein kinase Cα signaling regulates inhibitor of DNA binding 1 in the intestinal epithelium A transient wave of BMP signaling in the retina is necessary for Müller glial differentiation.p204, a p200 family protein, as a multifunctional regulator of cell proliferation and differentiation Dimerization-driven degradation of C. elegans and human E proteins.Neuronal specification exploits the inherent flexibility of cell-cycle gap phases.Post-transcriptional down-regulation of Atoh1/Math1 by bone morphogenic proteins suppresses medulloblastoma development p53 regulates neural stem cell proliferation and differentiation via BMP-Smad1 signaling and Id1.Epigenomic enhancer annotation reveals a key role for NFIX in neural stem cell quiescence.Selective induction of neocortical GABAergic neurons by the PDK1-Akt pathway through activation of Mash1.Presenilin-1 acts via Id1 to regulate the function of muscle satellite cells in a gamma-secretase-independent manner.Global expression profiling of globose basal cells and neurogenic progression within the olfactory epithelium.Protein kinase CK2 in development and differentiation.Choices choices: regulation of precursor differentiation during enteric nervous system development.BMP signaling in telencephalic neural cell specification and maturation.Bone morphogenetic protein signaling in vertebrate motor neurons and neuromuscular communication.The roles of interferon-inducible p200 family members IFI16 and p204 in innate immune responses, cell differentiation and proliferation.bHLH proteins involved in Drosophila neurogenesis are mutually regulated at the level of stability Regulation of neurogenin stability by ubiquitin-mediated proteolysis.Cross-talk between Wnt and bone morphogenetic protein 2 (BMP-2) signaling in differentiation pathway of C2C12 myoblasts.Return to quiescence of mouse neural stem cells by degradation of a proactivation protein.Unraveling the differential dynamics of developmental fate in central and peripheral nervous systems Multi-site phospho-regulation of proneural transcription factors controls proliferation versus differentiation in development and reprogramming.Scleraxis modulates bone morphogenetic protein 4 (BMP4)-Smad1 protein-smooth muscle α-actin (SMA) signal transduction in diabetic nephropathy.Characterization of Wnt/β-catenin and BMP/Smad signaling pathways in an in vitro model of amyotrophic lateral sclerosis.Differential interactions between Notch and ID factors control neurogenesis by modulating Hes factor autoregulation.Bone morphogenetic protein 2 inhibits neurite outgrowth of motor neuron-like NSC-34 cells and up-regulates its type II receptor.

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P2860

BMP-2 decreases Mash1 stability by increasing Id1 expression.

http://www.wikidata.org/entity/Q39996530

description

2004 nî lūn-bûn

@nan

2004年の論文

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2004年学术文章

@wuu

2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

@zh-sg

2004年學術文章

@yue

2004年學術文章

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2004年學術文章

@zh-hant

name

BMP-2 decreases Mash1 stability by increasing Id1 expression.

@en

BMP-2 decreases Mash1 stability by increasing Id1 expression.

@nl

type

label

BMP-2 decreases Mash1 stability by increasing Id1 expression.

@en

BMP-2 decreases Mash1 stability by increasing Id1 expression.

@nl

prefLabel

BMP-2 decreases Mash1 stability by increasing Id1 expression.

@en

BMP-2 decreases Mash1 stability by increasing Id1 expression.

@nl

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P356

SJ.EMBOJ.7600360

P1433

The EMBO Journal

P1476

BMP-2 decreases Mash1 stability by increasing Id1 expression.

@en

P2093

Francesc Viñals

http://www.w3.org/2001/XMLSchema#string

Jose Luis Rosa

http://www.w3.org/2001/XMLSchema#string

Julia Reiriz

http://www.w3.org/2001/XMLSchema#string

Ramon Bartrons

http://www.w3.org/2001/XMLSchema#string

Santiago Ambrosio

http://www.w3.org/2001/XMLSchema#string

P2860

Synergistic signaling in fetal brain by STAT3-Smad1 complex bridged by p300

Notch signaling induces rapid degradation of achaete-scute homolog 1.

Alternative neural crest cell fates are instructively promoted by TGFbeta superfamily members

Phosphorylation of nuclear MyoD is required for its rapid degradation.

Mechanisms of TGF-beta signaling from cell membrane to the nucleus

Degradation of Id proteins by the ubiquitin-proteasome pathway

Id1 and Id3 are required for neurogenesis, angiogenesis and vascularization of tumour xenografts

Smad-dependent and Smad-independent pathways in TGF-beta family signalling

Modulation of basic helix-loop-helix transcription complex formation by Id proteins during neuronal differentiation

An achaete-scute homologue essential for neuroendocrine differentiation in the lung

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3527-3537

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scholarly article

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10.1038/SJ.EMBOJ.7600360

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17

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23

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Francesc Ventura

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2004-08-19T00:00:00Z

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8393773

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15318167

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516632

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