Stepwise transformation of rat embryo fibroblasts: c-Jun, JunB, or JunD can cooperate with Ras for focus formation, but a c-Jun-containing heterodimer is required for immortalization.
about
Kaposi's sarcoma-associated herpesvirus induction of AP-1 and interleukin 6 during primary infection mediated by multiple mitogen-activated protein kinase pathways.Distinct roles of Jun : Fos and Jun : ATF dimers in oncogenesisAP-1 in cell proliferation and survivalThe cancer/testis antigen CAGE with oncogenic potential stimulates cell proliferation by up-regulating cyclins D1 and E in an AP-1- and E2F-dependent manner.Inhibition of AP-1 by SARI negatively regulates transformation progression mediated by CCN1.Cloning and characterization of SARI (suppressor of AP-1, regulated by IFN).The c-Jun-induced transformation process involves complex regulation of tenascin-C expression.Activation mechanisms of the urokinase-type plasminogen activator promoter by hepatocyte growth factor/scatter factor.The role of neurofibromin in N-Ras mediated AP-1 regulation in malignant peripheral nerve sheath tumorsNovel mechanism of JNK pathway activation by adenoviral E1A.Overexpression of hTERT extends replicative capacity of human nucleus pulposus cells, and protects against serum starvation-induced apoptosis and cell cycle arrest.KAI1 promoter activity is dependent on p53, junB and AP2: evidence for a possible mechanism underlying loss of KAI1 expression in cancer cells.Rap1 reverses transcriptional repression of TGF-beta type II receptor by a mechanism involving AP-1 in the human pancreatic cancer cell line, UK Pan-1.The defective transforming phenotype of c-Jun Ala(63/73) is rescued by mutation of the C-terminal phosphorylation site.Herpes simplex virus type 1 infection stimulates p38/c-Jun N-terminal mitogen-activated protein kinase pathways and activates transcription factor AP-1.Fra-1 expression in malignant and benign thyroid tumor.Role of mitogen-activated protein kinases and c-Jun/AP-1 trans-activating activity in the regulation of protease mRNAs and the malignant phenotype in NIH 3T3 fibroblasts.JunD regulates transcription of the tissue inhibitor of metalloproteinases-1 and interleukin-6 genes in activated hepatic stellate cells.Two UVC-induced stress response pathways in HeLa cells identified by cDNA microarray.Activation of activator protein 1 and extracellular signal-regulated kinases in human hepatocellular transformation.miR-126-5p by direct targeting of JNK-interacting protein-2 (JIP-2) plays a key role in Theileria-infected macrophage virulence.Cell context reveals a dual role for Maf in oncogenesis.High levels of phosphorylated c-Jun, Fra-1, Fra-2 and ATF-2 proteins correlate with malignant phenotypes in the multistage mouse skin carcinogenesis model.
P2860
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P2860
Stepwise transformation of rat embryo fibroblasts: c-Jun, JunB, or JunD can cooperate with Ras for focus formation, but a c-Jun-containing heterodimer is required for immortalization.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Stepwise transformation of rat ...... required for immortalization.
@en
type
label
Stepwise transformation of rat ...... required for immortalization.
@en
prefLabel
Stepwise transformation of rat ...... required for immortalization.
@en
P2093
P2860
P356
P1476
Stepwise transformation of rat ...... required for immortalization.
@en
P2093
P2860
P304
P356
10.1128/MCB.16.5.1881
P407
P577
1996-05-01T00:00:00Z