Response to rotenone is glucose-sensitive in a model of human acute lymphoblastic leukemia: involvement of oxidative stress mechanism, DJ-1, Parkin, and PINK-1 proteins.
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Fructose sensitizes Jurkat cells oxidative stress-induced apoptosis via caspase-dependent and caspase-independent mechanisms.Mitophagy in TGEV infection counteracts oxidative stress and apoptosis.A combined approach against tumorigenesis using glucose deprivation and mitochondrial complex 1 inhibition by rotenone.Neuroprotective Effect of the LRRK2 Kinase Inhibitor PF-06447475 in Human Nerve-Like Differentiated Cells Exposed to Oxidative Stress Stimuli: Implications for Parkinson's Disease.Vitamin E synthetic derivate-TPGS-selectively induces apoptosis in jurkat t cells via oxidative stress signaling pathways: implications for acute lymphoblastic leukemia.
P2860
Response to rotenone is glucose-sensitive in a model of human acute lymphoblastic leukemia: involvement of oxidative stress mechanism, DJ-1, Parkin, and PINK-1 proteins.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
2014年论文
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2014年论文
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name
Response to rotenone is glucos ...... , Parkin, and PINK-1 proteins.
@en
type
label
Response to rotenone is glucos ...... , Parkin, and PINK-1 proteins.
@en
prefLabel
Response to rotenone is glucos ...... , Parkin, and PINK-1 proteins.
@en
P2860
P356
P1476
Response to rotenone is glucos ...... 1, Parkin, and PINK-1 proteins
@en
P2093
Marlene Jimenez-Del-Rio
Miguel Mendivil-Perez
P2860
P304
P356
10.1155/2014/457154
P577
2014-05-11T00:00:00Z