Crosstalk between ATF4 and MTA1/HDAC1 promotes osteosarcoma progression.
about
Increased phosphorylation of eIF2α in chronic myeloid leukemia cells stimulates secretion of matrix modifying enzymes.ATF4 promotes angiogenesis and neuronal cell death and confers ferroptosis in a xCT-dependent manner.The oxido-metabolic driver ATF4 enhances temozolamide chemo-resistance in human gliomasPSAT1 is regulated by ATF4 and enhances cell proliferation via the GSK3β/β-catenin/cyclin D1 signaling pathway in ER-negative breast cancer.Surviving Stress: Modulation of ATF4-Mediated Stress Responses in Normal and Malignant Cells.
P2860
Crosstalk between ATF4 and MTA1/HDAC1 promotes osteosarcoma progression.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年学术文章
@wuu
2016年学术文章
@zh-cn
2016年学术文章
@zh-hans
2016年学术文章
@zh-my
2016年学术文章
@zh-sg
2016年學術文章
@yue
2016年學術文章
@zh
2016年學術文章
@zh-hant
name
Crosstalk between ATF4 and MTA1/HDAC1 promotes osteosarcoma progression.
@en
type
label
Crosstalk between ATF4 and MTA1/HDAC1 promotes osteosarcoma progression.
@en
prefLabel
Crosstalk between ATF4 and MTA1/HDAC1 promotes osteosarcoma progression.
@en
P2093
P2860
P356
P1433
P1476
Crosstalk between ATF4 and MTA1/HDAC1 promotes osteosarcoma progression.
@en
P2093
Caihong Yang
Jiang Wang
Jin-Ming Zhang
P2860
P304
P356
10.18632/ONCOTARGET.6940
P407
P577
2016-01-18T00:00:00Z