Plasma cell-specific transcription factor XBP-1s binds to and transactivates the Epstein-Barr virus BZLF1 promoter.
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Regulation of the latent-lytic switch in Epstein-Barr virusInterplay of Murine Gammaherpesvirus 68 with NF-kappaB Signaling of the HostHCV causes chronic endoplasmic reticulum stress leading to adaptation and interference with the unfolded protein responseThe B-cell specific transcription factor, Oct-2, promotes Epstein-Barr virus latency by inhibiting the viral immediate-early protein, BZLF1Gammaherpesvirus-driven plasma cell differentiation regulates virus reactivation from latently infected B lymphocytes.IL-21 imposes a type II EBV gene expression on type III and type I B cells by the repression of C- and activation of LMP-1-promoter.Hypoxia-inducible factor-1α plays roles in Epstein-Barr virus's natural life cycle and tumorigenesis by inducing lytic infection through direct binding to the immediate-early BZLF1 gene promoter.Epstein-Barr virus (EBV) Rta-mediated EBV and Kaposi's sarcoma-associated herpesvirus lytic reactivations in 293 cells.The murine gammaherpesvirus immediate-early Rta synergizes with IRF4, targeting expression of the viral M1 superantigen to plasma cellsViral latency and its regulation: lessons from the gamma-herpesviruses.Efficient replication of Epstein-Barr virus in stratified epithelium in vitro.Down-regulation of BLIMP1α by the EBV oncogene, LMP-1, disrupts the plasma cell differentiation program and prevents viral replication in B cells: implications for the pathogenesis of EBV-associated B-cell lymphomas.Tegument protein control of latent herpesvirus establishment and animation.Bortezomib induction of C/EBPβ mediates Epstein-Barr virus lytic activation in Burkitt lymphoma.The ZIIR element of the Epstein-Barr virus BZLF1 promoter plays a central role in establishment and maintenance of viral latencyThe Epstein-Barr virus BRRF1 protein, Na, induces lytic infection in a TRAF2- and p53-dependent mannerMCAF1 and Rta-activated BZLF1 transcription in Epstein-Barr virus.Interleukin 21 signaling in B cells is required for efficient establishment of murine gammaherpesvirus latencyMaintenance of Epstein-Barr Virus Latent Status by a Novel Mechanism, Latent Membrane Protein 1-Induced Interleukin-32, via the Protein Kinase Cδ Pathway.An Epstein-Barr Virus (EBV) mutant with enhanced BZLF1 expression causes lymphomas with abortive lytic EBV infection in a humanized mouse model.Amplification of JNK signaling is necessary to complete the murine gammaherpesvirus 68 lytic replication cycle.Frequency and phenotype of human immunodeficiency virus envelope-specific B cells from patients with broadly cross-neutralizing antibodies.Contribution of myocyte enhancer factor 2 family transcription factors to BZLF1 expression in Epstein-Barr virus reactivation from latency.The product of Kaposi's sarcoma-associated herpesvirus immediate early gene K4.2 regulates immunoglobulin secretion and calcium homeostasis by interacting with and inhibiting pERP1.Epstein-Barr virus induces global changes in cellular mRNA isoform usage that are important for the maintenance of latency.The unfolded protein response and autophagy: herpesviruses rule!NF-κB as a target for oncogenic viruses.The role of X-box binding protein-1 in tumorigenicity.Epstein-Barr virus utilizes Ikaros in regulating its latent-lytic switch in B cells.Switching of EBV cycles between latent and lytic states.Dynamic Epstein-Barr virus gene expression on the path to B-cell transformation.Regulation of Epstein-Barr virus reactivation from latency.The expanding roles of endoplasmic reticulum stress in virus replication and pathogenesis.Activation of the B cell antigen receptor triggers reactivation of latent Kaposi's sarcoma-associated herpesvirus in B cells.Identification of Novel Kaposi's Sarcoma-Associated Herpesvirus Orf50 Transcripts: Discovery of New RTA Isoforms with Variable Transactivation Potential.Cellular differentiation regulator BLIMP1 induces Epstein-Barr virus lytic reactivation in epithelial and B cells by activating transcription from both the R and Z promoters.Epstein-barr virus blocks the autophagic flux and appropriates the autophagic machinery to enhance viral replication.Epstein-Barr virus: more than 50 years old and still providing surprises.Murine gammaherpesvirus 68 reactivation from B cells requires IRF4 but not XBP-1.Histone acetyltransferase-deficient p300 mutants in diffuse large B cell lymphoma have altered transcriptional regulatory activities and are required for optimal cell growth.
P2860
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P2860
Plasma cell-specific transcription factor XBP-1s binds to and transactivates the Epstein-Barr virus BZLF1 promoter.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Plasma cell-specific transcrip ...... ein-Barr virus BZLF1 promoter.
@en
type
label
Plasma cell-specific transcrip ...... ein-Barr virus BZLF1 promoter.
@en
prefLabel
Plasma cell-specific transcrip ...... ein-Barr virus BZLF1 promoter.
@en
P2860
P356
P1433
P1476
Plasma cell-specific transcrip ...... ein-Barr virus BZLF1 promoter.
@en
P2093
Chia Chi Sun
David A Thorley-Lawson
P2860
P304
13566-13577
P356
10.1128/JVI.01055-07
P407
P577
2007-09-26T00:00:00Z