Severe acute respiratory syndrome coronavirus evades antiviral signaling: role of nsp1 and rational design of an attenuated strain.
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NS5 of dengue virus mediates STAT2 binding and degradationRegulation of Stress Responses and Translational Control by CoronavirusSuppression of coronavirus replication by cyclophilin inhibitorsCoronavirus virulence genes with main focus on SARS-CoV envelope geneCoronavirus gene 7 counteracts host defenses and modulates virus virulenceCoronavirus Infection Modulates the Unfolded Protein Response and Mediates Sustained Translational RepressionMurine Coronavirus Mouse Hepatitis Virus Is Recognized by MDA5 and Induces Type I Interferon in Brain Macrophages/MicrogliaStructure of Alphacoronavirus Transmissible Gastroenteritis Virus nsp1 Has Implications for Coronavirus nsp1 Function and EvolutionInfluenza A Virus Protein PA-X Contributes to Viral Growth and Suppression of the Host Antiviral and Immune ResponsesDynamic innate immune responses of human bronchial epithelial cells to severe acute respiratory syndrome-associated coronavirus infection.SARS-CoV pathogenesis is regulated by a STAT1 dependent but a type I, II and III interferon receptor independent mechanism.Pathogenicity of severe acute respiratory coronavirus deletion mutants in hACE-2 transgenic mice.Coronaviruses post-SARS: update on replication and pathogenesis.Global mRNA degradation during lytic gammaherpesvirus infection contributes to establishment of viral latency.Mechanisms of severe acute respiratory syndrome pathogenesis and innate immunomodulation.Pathogenesis of murine coronavirus in the central nervous system.Accessory protein 5a is a major antagonist of the antiviral action of interferon against murine coronavirus.Porcine epidemic diarrhea virus nucleocapsid protein antagonizes beta interferon production by sequestering the interaction between IRF3 and TBK1.Dependence of coronavirus RNA replication on an NH2-terminal partial nonstructural protein 1 in cis.The SARS-coronavirus-host interactome: identification of cyclophilins as target for pan-coronavirus inhibitorsSARS coronavirus nsp1 protein induces template-dependent endonucleolytic cleavage of mRNAs: viral mRNAs are resistant to nsp1-induced RNA cleavageAutocrine interferon priming in macrophages but not dendritic cells results in enhanced cytokine and chemokine production after coronavirus infectionAlphacoronavirus transmissible gastroenteritis virus nsp1 protein suppresses protein translation in mammalian cells and in cell-free HeLa cell extracts but not in rabbit reticulocyte lysateMurine coronavirus ubiquitin-like domain is important for papain-like protease stability and viral pathogenesis.Attenuation of mouse hepatitis virus by deletion of the LLRKxGxKG region of Nsp1.Identification of residues of SARS-CoV nsp1 that differentially affect inhibition of gene expression and antiviral signaling.The SARS coronavirus papain like protease can inhibit IRF3 at a post activation step that requires deubiquitination activity.Interferon priming enables cells to partially overturn the SARS coronavirus-induced block in innate immune activationSARS coronavirus papain-like protease inhibits the type I interferon signaling pathway through interaction with the STING-TRAF3-TBK1 complex.The nsp1, nsp13, and M proteins contribute to the hepatotropism of murine coronavirus JHM.WU.Cytokine systems approach demonstrates differences in innate and pro-inflammatory host responses between genetically distinct MERS-CoV isolates.Coronavirus nonstructural protein 1: Common and distinct functions in the regulation of host and viral gene expression.Mechanisms of innate immune evasion in re-emerging RNA viruses.Genetic evidence of a long-range RNA-RNA interaction between the genomic 5' untranslated region and the nonstructural protein 1 coding region in murine and bovine coronavirusesIdentification of the Mechanisms Causing Reversion to Virulence in an Attenuated SARS-CoV for the Design of a Genetically Stable Vaccine.Nonstructural Protein 11 of Porcine Reproductive and Respiratory Syndrome Virus Suppresses Both MAVS and RIG-I Expression as One of the Mechanisms to Antagonize Type I Interferon Production.Severe acute respiratory syndrome coronavirus nsp1 facilitates efficient propagation in cells through a specific translational shutoff of host mRNA.Severe Acute Respiratory Syndrome Coronavirus ORF7a Inhibits Bone Marrow Stromal Antigen 2 Virion Tethering through a Novel Mechanism of Glycosylation Interference.Middle East respiratory syndrome coronavirus ORF4b protein inhibits type I interferon production through both cytoplasmic and nuclear targets.Induction of alternatively activated macrophages enhances pathogenesis during severe acute respiratory syndrome coronavirus infection
P2860
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P2860
Severe acute respiratory syndrome coronavirus evades antiviral signaling: role of nsp1 and rational design of an attenuated strain.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Severe acute respiratory syndr ...... esign of an attenuated strain.
@en
type
label
Severe acute respiratory syndr ...... esign of an attenuated strain.
@en
prefLabel
Severe acute respiratory syndr ...... esign of an attenuated strain.
@en
P2093
P2860
P356
P1433
P1476
Severe acute respiratory syndr ...... esign of an attenuated strain.
@en
P2093
Marc G Wathelet
Matthew B Frieman
Melissa Orr
P2860
P304
11620-11633
P356
10.1128/JVI.00702-07
P407
P577
2007-08-22T00:00:00Z