Protumorigenic overexpression of stathmin/Op18 by gain-of-function mutation in p53 in human hepatocarcinogenesis.
about
Stathmin is dispensable for tumor onset in miceCharacterization of human septic sera induced gene expression modulation in human myocytes.Mutant p53 gain-of-function in cancer.Overexpression of stathmin1 in the diffuse type of gastric cancer and its roles in proliferation and migration of gastric cancer cells.Increased expression of stathmin and elongation factor 1α in precancerous nodules with telomere dysfunction in hepatitis B viral cirrhotic patients.Bioinformatics analysis of key genes and pathways for hepatocellular carcinoma transformed from cirrhosisA cohort study of STMN1 expression in colorectal cancer: body mass index and prognosis.Nicotine-mediated invasion and migration of non-small cell lung carcinoma cells by modulating STMN3 and GSPT1 genes in an ID1-dependent manner.MiR-101 is involved in human breast carcinogenesis by targeting Stathmin1.Stathmin regulates keratinocyte proliferation and migration during cutaneous regeneration.Stathmin1 regulates p27 expression, proliferation and drug resistance, resulting in poor clinical prognosis in cholangiocarcinoma.RETRACTED: STMN-1 is a potential marker of lymph node metastasis in distal esophageal adenocarcinomas and silencing its expression can reverse malignant phenotype of tumor cells.Upregulation of the mitochondrial transport protein, Tim50, by mutant p53 contributes to cell growth and chemoresistanceThe prognostic value and therapeutic target role of stathmin-1 in urinary bladder cancerProteomics identification of ITGB3 as a key regulator in reactive oxygen species-induced migration and invasion of colorectal cancer cellsmicroRNA-101 is a potent inhibitor of autophagymiR-1228 promotes the proliferation and metastasis of hepatoma cells through a p53 forward feedback loopTransactivation of the TIEG1 confers growth inhibition of transforming growth factor-β-susceptible hepatocellular carcinoma cells.Mutant p53: one name, many proteins.Deregulation of FoxM1b leads to tumour metastasis.Gain-of-Function Activity of Mutant p53 in Lung Cancer through Up-Regulation of Receptor Protein Tyrosine Kinase AxlGenotyping of stathmin and its association with clinical factors and survival in patients with ovarian cancerStathmin regulates mutant p53 stability and transcriptional activity in ovarian cancer.The consequence of oncomorphic TP53 mutations in ovarian cancer.Aurora kinase inhibitor PHA-739358 suppresses growth of hepatocellular carcinoma in vitro and in a xenograft mouse model.High-resolution flow cytometry: a suitable tool for monitoring aneuploid prostate cancer cells after TMZ and TMZ-BioShuttle treatment.An underlying prognosis predictor of hepatocellular carcinoma: Oncoprotein 18.Stathmin: a protein with many tasks. New biomarker and potential target in cancer.Epigenetic silencing of miR-210 increases the proliferation of gastric epithelium during chronic Helicobacter pylori infection.Elevated STMN1 Expression Correlates with Poor Prognosis in Patients with Pancreatic Ductal Adenocarcinoma.Stathmin1 overexpression in hypopharyngeal squamous cell carcinoma: A new promoter in FaDu cell proliferation and migration.High expression of stathmin 1 is a strong prognostic marker in oral squamous cell carcinoma patients treated by docetaxel-containing regimens.Stathmin in pancreatic neuroendocrine neoplasms: a marker of proliferation and PI3K signaling.Transcriptional Regulation by Wild-Type and Cancer-Related Mutant Forms of p53.Gain-of-function mutant p53 downregulates miR-223 contributing to chemoresistance of cultured tumor cells.Allele specific gain-of-function activity of p53 mutants in lung cancer cells.Nuclear accumulation of seven in absentia homologue-2 supports motility and proliferation of liver cancer cells.p53 mutants induce transcription of NF-κB2 in H1299 cells through CBP and STAT binding on the NF-κB2 promoter and gain of function activity.The transcriptional coactivators megakaryoblastic leukemia 1/2 mediate the effects of loss of the tumor suppressor deleted in liver cancer 1.Overexpression of far upstream element binding proteins: a mechanism regulating proliferation and migration in liver cancer cells.
P2860
Q21134036-950F238A-C690-4DDE-9464-837AFF069016Q30487504-46E25BDB-9034-408C-82A1-C22E55CAF668Q33687048-4F0E45BE-6409-414B-A233-13D5C07A9D6EQ33723723-42B8C3F3-39EC-4BD0-819C-A8D6F3DDD652Q33728805-20C06615-772A-41F0-ADB2-B9C3227C686CQ33833557-D45D4DA3-A321-4D36-96AC-88D1331E27C6Q33837712-79AD99FA-9F04-440F-BC17-DA423504ED91Q34000136-D58D86BC-81A0-469F-8D27-BB885F805BD6Q34446981-D41554D2-F8B0-4AAB-AB0F-F98CC2B992D3Q34997445-7AD1B74D-FAC9-4335-93AF-F2697411A214Q35051799-6EE1A970-987C-4B3B-81E4-7C75E87B44AFQ35085500-1A2C3A76-190A-464E-A12B-220DF18C9280Q35086367-FDC9A747-941C-4522-8E3A-73DD203FFD12Q35214846-A7A57B09-8082-4A10-858C-723AB3E66B45Q35497344-629B7640-22F1-4431-8AC8-7FD0C8B2D789Q35626306-712AF54A-877C-47B6-A293-278443C0E1CEQ35677296-94088427-22AB-46EB-ABF1-D1AFEC59A546Q35929155-1EC63151-E8E7-4980-AFD7-A3CF47DFE448Q36070574-92C72A79-EEE7-4CCC-84D6-65CC4F792A85Q36108360-697220DA-2C1A-48DD-84D8-7A2BC100F643Q36481940-614F6C33-2B28-4B9D-A51A-7667DF3677DFQ36774388-82947A20-D45C-4150-B0FC-579D6C7F78BBQ36870100-4964912D-6169-4BB9-9D40-A5B259F31BFBQ37225303-82AFA45C-93E6-4B03-BABA-2CF6D30ED9FAQ37328115-CCE13143-3EE9-4A53-84D4-9CBC6FDB1CE0Q37430234-DFD755AB-9FC3-4874-B295-A47DE437FDC1Q37565193-9FB22472-7996-4296-AD35-3D60B0802F66Q37942978-AF1415A5-3AAF-46EF-8E39-15CA8D7C115BQ38304465-36B9C4C1-B9C6-43CA-A252-07E7E2959B63Q38387283-A7166F8E-90F4-4991-87B3-F73871F057AAQ38729446-D9CB2A6B-F7BD-43A6-8791-F7280F8BDF8CQ38817928-276396F4-1304-47D5-97A5-06B76BA0861AQ38952182-B2B5EE34-FC36-42F9-8EE2-7EF6DF457D30Q39005896-B476D921-46C4-4BE4-9F21-572357202417Q39166823-CBD3F84D-AAD6-4D88-A4C3-86A020719595Q39276191-2D0912CD-1445-40AB-ABD3-18281F1E0A44Q39398502-A39BA4C9-91F3-4491-8887-B5D16EF41645Q39422471-559B063A-F1C0-4DC4-A67D-AD6774094590Q39433431-F8AD19ED-8520-40DB-9EBE-E5728BF7D8C7Q39827518-38F6EAB3-E1E1-421D-A95D-27D03F6F971A
P2860
Protumorigenic overexpression of stathmin/Op18 by gain-of-function mutation in p53 in human hepatocarcinogenesis.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Protumorigenic overexpression ...... in human hepatocarcinogenesis.
@en
type
label
Protumorigenic overexpression ...... in human hepatocarcinogenesis.
@en
prefLabel
Protumorigenic overexpression ...... in human hepatocarcinogenesis.
@en
P2093
P2860
P356
P1433
P1476
Protumorigenic overexpression ...... in human hepatocarcinogenesis
@en
P2093
Antje Brauckhoff
Martina Keith
Peter Schirmacher
Sebastian Vreden
Stephan Singer
Volker Ehemann
P2860
P304
P356
10.1002/HEP.21736
P407
P50
P577
2007-09-01T00:00:00Z