Transient and long-lasting openings of the mitochondrial permeability transition pore can be monitored directly in intact cells by changes in mitochondrial calcein fluorescence.
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Inhibition of the Mitochondrial Permeability Transition for Cytoprotection: Direct versus Indirect MechanismsApolipoprotein O is mitochondrial and promotes lipotoxicity in heartThe permeability transition pore triggers Bax translocation to mitochondria during neuronal apoptosisMitochondria, calcium and cell death: a deadly triad in neurodegenerationCyclosporin A corrects mitochondrial dysfunction and muscle apoptosis in patients with collagen VI myopathiesCell death disguised: The mitochondrial permeability transition pore as the c-subunit of the F(1)F(O) ATP synthaseMitochondrial reactive oxygen species: a double edged sword in ischemia/reperfusion vs preconditioningFrom ATP to PTP and Back: A Dual Function for the Mitochondrial ATP SynthaseNovel quinazolinone MJ-29 triggers endoplasmic reticulum stress and intrinsic apoptosis in murine leukemia WEHI-3 cells and inhibits leukemic micePhoto-induction and automated quantification of reversible mitochondrial permeability transition pore opening in primary mouse myotubesAlterations in voltage-sensing of the mitochondrial permeability transition pore in ANT1-deficient cells.The oat mitochondrial permeability transition and its implication in victorin binding and induced cell deathThe mitochondrial permeability transition pore: molecular nature and role as a target in cardioprotectionCaMKII induces permeability transition through Drp1 phosphorylation during chronic β-AR stimulationVoltage-dependent anion channels are dispensable for mitochondrial-dependent cell deathThe Mitochondrial Permeability Transition Pore Regulator Cyclophilin D Exhibits Tissue-Specific Control of Metabolic Homeostasis.Calcium-independent phospholipase A2 (iPLA2 beta)-mediated ceramide generation plays a key role in the cross-talk between the endoplasmic reticulum (ER) and mitochondria during ER stress-induced insulin-secreting cell apoptosisHexokinase II knockdown results in exaggerated cardiac hypertrophy via increased ROS productionDisruption of actin filaments induces mitochondrial Ca2+ release to the cytoplasm and [Ca2+]c changes in Arabidopsis root hairs.Superoxide flashes: early mitochondrial signals for oxidative stress-induced apoptosis.Permeability transition pore-mediated mitochondrial superoxide flashes mediate an early inhibitory effect of amyloid beta1-42 on neural progenitor cell proliferation.Fluctuations in mitochondrial membrane potential caused by repetitive gating of the permeability transition porePhotodynamic therapy-induced apoptosis in epidermoid carcinoma cells. Reactive oxygen species and mitochondrial inner membrane permeabilization.Modulation of mitochondrial Ca2+ by nitric oxide in cultured bovine vascular endothelial cells.Characteristics and function of cardiac mitochondrial nitric oxide synthase.Unconjugated Bilirubin exerts Pro-Apoptotic Effect on Platelets via p38-MAPK activationEffect of transient and permanent permeability transition pore opening on NAD(P)H localization in intact cells.Platelet protective efficacy of 3,4,5 trisubstituted isoxazole analogue by inhibiting ROS-mediated apoptosis and platelet aggregation.Involvement of VDAC, Bax and ceramides in the efflux of AIF from mitochondria during curcumin-induced apoptosis.Regulation of the mitochondrial permeability transition pore by the outer membrane does not involve the peripheral benzodiazepine receptor (Translocator Protein of 18 kDa (TSPO)).Ca2+ overload and mitochondrial permeability transition pore activation in living delta-sarcoglycan-deficient cardiomyocytes.PLCδ1 protein rescues ischemia-reperfused heart by the regulation of calcium homeostasisTargeting chelatable iron as a therapeutic modality in Parkinson's disease.Melatonin preserves the transient mitochondrial permeability transition for protection during mitochondrial Ca(2+) stress in astrocyte.Optogenetic control of mitochondrial metabolism and Ca2+ signaling by mitochondria-targeted opsinsAzidothymidine-triphosphate impairs mitochondrial dynamics by disrupting the quality control system.Identification of specific pluripotent stem cell death--inducing small molecules by chemical screening.Granzyme B can cause mitochondrial depolarization and cell death in the absence of BID, BAX, and BAK.Melatonin enhances hydrogen peroxide-induced apoptosis in human promyelocytic leukaemia HL-60 cells.Mitochondria as all-round players of the calcium game.
P2860
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P2860
Transient and long-lasting openings of the mitochondrial permeability transition pore can be monitored directly in intact cells by changes in mitochondrial calcein fluorescence.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Transient and long-lasting ope ...... hondrial calcein fluorescence.
@en
type
label
Transient and long-lasting ope ...... hondrial calcein fluorescence.
@en
prefLabel
Transient and long-lasting ope ...... hondrial calcein fluorescence.
@en
P2093
P2860
P1433
P1476
Transient and long-lasting ope ...... hondrial calcein fluorescence.
@en
P2093
P2860
P304
P356
10.1016/S0006-3495(99)77239-5
P407
P577
1999-02-01T00:00:00Z