Melatonin preserves the transient mitochondrial permeability transition for protection during mitochondrial Ca(2+) stress in astrocyte.

Melatonin preserves the transient mitochondrial permeability transition for protection during mitochondrial Ca(2+) stress in astrocyte.

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http://www.wikidata.org/entity/Q33833058
Heat stress induced apoptosis is triggered by transcription-independent p53, Ca(2+) dyshomeostasis and the subsequent Bax mitochondrial translocation.Melatonin Therapy in Patients with Alzheimer's Disease.Melatonin antioxidative defense: therapeutical implications for aging and neurodegenerative processes.Mitochondria and chloroplasts as the original sites of melatonin synthesis: a hypothesis related to melatonin's primary function and evolution in eukaryotes.Melatonin and the theories of aging: a critical appraisal of melatonin's role in antiaging mechanisms.Protective role of melatonin in mitochondrial dysfunction and related disorders.Mitochondrial Ca2+ and regulation of the permeability transition pore.2',3'-Cyclic nucleotide 3'-phosphodiesterase as a messenger of protection of the mitochondrial function during melatonin treatment in aging.Melatonin improved rat cardiac mitochondria and survival rate in septic heart injury.mtDNA T8993G mutation-induced mitochondrial complex V inhibition augments cardiolipin-dependent alterations in mitochondrial dynamics during oxidative, Ca(2+), and lipid insults in NARP cybrids: a potential therapeutic target for melatonin.Long-term Aβ exposure augments mCa2+-independent mROS-mediated depletion of cardiolipin for the shift of a lethal transient mitochondrial permeability transition to its permanent mode in NARP cybrids: a protective targeting of melatonin.Melatonin and the electron transport chain.Melatonin prevents ultraviolet radiation-induced alterations in plasma membrane potential and intracellular pH in human keratinocytes.Melatonin prevents the dynamin-related protein 1-dependent mitochondrial fission and oxidative insult in the cortical neurons after 1-methyl-4-phenylpyridinium treatment.Melatonin protects against rotenone-induced cell injury via inhibition of Omi and Bax-mediated autophagy in Hela cells.Melatonin enhances antioxidative enzyme gene expression (CAT, GPx, SOD), prevents their UVR-induced depletion, and protects against the formation of DNA damage (8-hydroxy-2'-deoxyguanosine) in ex vivo human skin.
P2860

Melatonin preserves the transient mitochondrial permeability transition for protection during mitochondrial Ca(2+) stress in astrocyte.

Item
http://www.wikidata.org/entity/Q33833058
description
2011 nî lūn-bûn
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2011 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մարտին հրատարակված գիտական հոդված
@hy
2011年の論文
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2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Melatonin preserves the transi ...... al Ca(2+) stress in astrocyte.
@ast
Melatonin preserves the transi ...... al Ca(2+) stress in astrocyte.
@en
type
label
Melatonin preserves the transi ...... al Ca(2+) stress in astrocyte.
@ast
Melatonin preserves the transi ...... al Ca(2+) stress in astrocyte.
@en
prefLabel
Melatonin preserves the transi ...... al Ca(2+) stress in astrocyte.
@ast
Melatonin preserves the transi ...... al Ca(2+) stress in astrocyte.
@en
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Melatonin preserves the transi ...... al Ca(2+) stress in astrocyte.
@en
P2093
P2860
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P31
P356
10.1111/J.1600-079X.2011.00861.X
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P698
P921