Multipotent hematopoietic cells susceptible to alternative double-strand break repair pathways that promote genome rearrangements.
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The ubiquitin ligase FBXW7 modulates leukemia-initiating cell activity by regulating MYC stabilityOxidative stress, bone marrow failure, and genome instability in hematopoietic stem cellsThe consequences of Rad51 overexpression for normal and tumor cellsDeregulation of DNA double-strand break repair in multiple myeloma: implications for genome stabilityHematopoietic stem cell quiescence promotes error-prone DNA repair and mutagenesisRsf-1, a chromatin remodeling protein, induces DNA damage and promotes genomic instability.siRNA screening identifies differences in the Fanconi anemia pathway in BALB/c-Trp53+/- with susceptibility versus C57BL/6-Trp53+/- mice with resistance to mammary tumors.Double-strand break repair by interchromosomal recombination: an in vivo repair mechanism utilized by multiple somatic tissues in mammals.Autophagy confers DNA damage repair pathways to protect the hematopoietic system from nuclear radiation injury.Impact of Charged Particle Exposure on Homologous DNA Double-Strand Break Repair in Human Blood-Derived Cells.Apoptotic susceptibility to DNA damage of pluripotent stem cells facilitates pharmacologic purging of teratoma riskDNA repair in murine embryonic stem cells and differentiated cellsHeterozygous PALB2 c.1592delT mutation channels DNA double-strand break repair into error-prone pathways in breast cancer patients.Cdk12 is essential for embryonic development and the maintenance of genomic stability.Human induced pluripotent cells resemble embryonic stem cells demonstrating enhanced levels of DNA repair and efficacy of nonhomologous end-joining.RAD51 overexpression is a negative prognostic marker for colorectal adenocarcinoma.NF-κB-dependent DNA damage-signaling differentially regulates DNA double-strand break repair mechanisms in immature and mature human hematopoietic cells.TRANSCRIPTIONAL AND PHOSPHO-PROTEOMIC SCREENS REVEAL STEM CELL ACTIVATION OF INSULIN-RESISTANCE AND TRANSFORMATION PATHWAYS FOLLOWING A SINGLE MINIMALLY TOXIC EPISODE OF ROS.The Universal Non-Neuronal Nature of Parkinson's Disease: A Theory.
P2860
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P2860
Multipotent hematopoietic cells susceptible to alternative double-strand break repair pathways that promote genome rearrangements.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Multipotent hematopoietic cell ...... promote genome rearrangements.
@en
type
label
Multipotent hematopoietic cell ...... promote genome rearrangements.
@en
prefLabel
Multipotent hematopoietic cell ...... promote genome rearrangements.
@en
P2860
P356
P1433
P1476
Multipotent hematopoietic cell ...... promote genome rearrangements.
@en
P2093
Christine Richardson
Richard Francis
P2860
P304
P356
10.1101/GAD.1522807
P577
2007-05-01T00:00:00Z