Upregulation of Twist-1 by NF-kappaB blocks cytotoxicity induced by chemotherapeutic drugs.
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Hyaluronan-CD44 interaction promotes c-Src-mediated twist signaling, microRNA-10b expression, and RhoA/RhoC up-regulation, leading to Rho-kinase-associated cytoskeleton activation and breast tumor cell invasionThe NF-κB Pathway and Cancer Stem CellsNovel Molecular Markers for Breast CancerThe emerging role of Twist proteins in hematopoietic cells and hematological malignanciesNormal and disease-related biological functions of Twist1 and underlying molecular mechanismsImmunomodulatory effects of low dose chemotherapy and perspectives of its combination with immunotherapyAKT-ions with a TWIST between EMT and METGene expression profiling of pulmonary fibrosis identifies Twist1 as an antiapoptotic molecular "rectifier" of growth factor signalingLoss of BRMS1 promotes a mesenchymal phenotype through NF-κB-dependent regulation of Twist1.NF-κB regulates mesenchymal transition for the induction of non-small cell lung cancer initiating cells.In vitro biophysical, microspectroscopic and cytotoxic evaluation of metastatic and non-metastatic cancer cells in responses to anti-cancer drug.Cellular processes of v-Src transformation revealed by gene profiling of primary cells--implications for human cancer.TCRP1 expression is associated with platinum sensitivity in human lung and ovarian cancer cells.Expression of the Breast Cancer Metastasis Suppressor 1 (BRMS1) maintains in vitro chemosensitivity of breast cancer cells.Downregulation of microRNA miR-520h by E1A contributes to anticancer activity.TWIST and ovarian cancer stem cells: implications for chemoresistance and metastasisTWIST1 associates with NF-κB subunit RELA via carboxyl-terminal WR domain to promote cell autonomous invasion through IL8 productionSuppression of Her2/neu expression through ILK inhibition is regulated by a pathway involving TWIST and YB-1Oncosuppressive suicide gene virotherapy "PVH1-yCD/5-FC" for pancreatic peritoneal carcinomatosis treatment: NFκB and Akt/PI3K involvementModulation of oxidative stress by twist oncoproteinsPDGFRα and β play critical roles in mediating Foxq1-driven breast cancer stemness and chemoresistance.Tamoxifen inhibits ER-negative breast cancer cell invasion and metastasis by accelerating Twist1 degradation.A New Paradigm to Mitigate Osteosarcoma by Regulation of MicroRNAs and Suppression of the NF-κB Signaling Cascade.Expression of Y-box-binding protein YB-1 allows stratification into long- and short-term survivors of head and neck cancer patientsOverexpression of TWIST2 correlates with poor prognosis in head and neck squamous cell carcinomas.Epithelial-mesenchymal transition induced by TNF-α requires NF-κB-mediated transcriptional upregulation of Twist1Oncogenic KRas suppresses inflammation-associated senescence of pancreatic ductal cellsA20 suppresses hepatocellular carcinoma proliferation and metastasis through inhibition of Twist1 expressionNF-kappaB Is Involved in the Regulation of EMT Genes in Breast Cancer Cells.Twist is transcriptionally induced by activation of STAT3 and mediates STAT3 oncogenic functionTwist2 is a valuable prognostic biomarker for colorectal cancer.Redox regulation of multidrug resistance in cancer chemotherapy: molecular mechanisms and therapeutic opportunitiesZoledronic acid reverses the epithelial-mesenchymal transition and inhibits self-renewal of breast cancer cells through inactivation of NF-κBRecently identified biomarkers that promote lymph node metastasis in head and neck squamous cell carcinomaInterhelical loops within the bHLH domain are determinant in maintaining TWIST1-DNA complexes.ZnCl2 sustains the adriamycin-induced cell death inhibited by high glucose.TGF-β reduces DNA ds-break repair mechanisms to heighten genetic diversity and adaptability of CD44+/CD24- cancer cells.Multiple biological functions of Twist1 in various cancers.Role of TLR2-dependent inflammation in metastatic progression.Phosphorylation of basic helix-loop-helix transcription factor Twist in development and disease.
P2860
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P2860
Upregulation of Twist-1 by NF-kappaB blocks cytotoxicity induced by chemotherapeutic drugs.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Upregulation of Twist-1 by NF- ...... ced by chemotherapeutic drugs.
@en
Upregulation of Twist-1 by NF- ...... ced by chemotherapeutic drugs.
@en-gb
type
label
Upregulation of Twist-1 by NF- ...... ced by chemotherapeutic drugs.
@en
Upregulation of Twist-1 by NF- ...... ced by chemotherapeutic drugs.
@en-gb
prefLabel
Upregulation of Twist-1 by NF- ...... ced by chemotherapeutic drugs.
@en
Upregulation of Twist-1 by NF- ...... ced by chemotherapeutic drugs.
@en-gb
P2093
P2860
P50
P921
P356
P1476
Upregulation of Twist-1 by NF- ...... ced by chemotherapeutic drugs.
@en
P2093
Can G Pham
Christian Kuntzen
Guido Franzoso
James R Knabb
P2860
P304
P356
10.1128/MCB.01219-06
P407
P577
2007-04-02T00:00:00Z