Identification of genes required for the spontaneous repair of partial-thickness cartilage defects in immature rats.

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Articular cartilage and joint development from embryogenesis to adulthood.Effect of Choline on the Composition and Degradation Enzyme of Extracellular Matrix of Mice Chondrocytes Exposed to Fluoride.

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P2860

Identification of genes required for the spontaneous repair of partial-thickness cartilage defects in immature rats.

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

@yue

2015年論文

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2015年論文

@zh-hk

2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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name

Identification of genes requir ...... lage defects in immature rats.

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type

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label

Identification of genes requir ...... lage defects in immature rats.

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Identification of genes requir ...... lage defects in immature rats.

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P1476

Identification of genes requir ...... lage defects in immature rats.

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P2093

Arata Nakajima

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Kazuhisa Takahashi

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Koichi Nakagawa

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Masahiko Saito

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Naoshi Ikegawa

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Satoshi Yamaguchi

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Taisuke Fukawa

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Takahisa Sasho

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Yorikazu Akatsu

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Yuta Muramatsu

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P2860

Role of hypoxia-inducible factor-1 alpha in the regulation of plasminogen activator activity in rat knee joint chondrocytes

The surface of articular cartilage contains a progenitor cell population.

Aberrant hypertrophy in Smad3-deficient murine chondrocytes is rescued by restoring transforming growth factor beta-activated kinase 1/activating transcription factor 2 signaling: a potential clinical implication for osteoarthritis.

Human developmental chondrogenesis as a basis for engineering chondrocytes from pluripotent stem cells.

IL1β induces mesenchymal stem cells migration and leucocyte chemotaxis through NF-κB.

Expression of a truncated, kinase-defective TGF-beta type II receptor in mouse skeletal tissue promotes terminal chondrocyte differentiation and osteoarthritis.

Modulation of TGF-beta signaling by proinflammatory cytokines in articular chondrocytes.

TGF-beta/Smad3 signals repress chondrocyte hypertrophic differentiation and are required for maintaining articular cartilage

Development of partial-thickness articular cartilage injury in a rabbit model.

Bovine lactoferricin-induced anti-inflammation is, in part, via up-regulation of interleukin-11 by secondary activation of STAT3 in human articular cartilage

P304

190-199

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scholarly article

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10.3109/03008207.2015.1121250

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P478

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Takane Suzuki

Ryuichiro Akagi

P577

2015-12-31T00:00:00Z

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P698

26719950

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P921

cartilage

Identification of genes required for the spontaneous repair of partial-thickness cartilage defects in immature rats.

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P2860

P2860

Identification of genes required for the spontaneous repair of partial-thickness cartilage defects in immature rats.

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type

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P1433

P1476

P2093

P2860

P304

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P356

P433

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P50

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P698

P921

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P1476

P2093

P2860

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P31

P356

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P478

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P577

P698

P921