Telmisartan inhibits AGE-induced C-reactive protein production through downregulation of the receptor for AGE via peroxisome proliferator-activated receptor-gamma activation.
about
Contribution of the toxic advanced glycation end-products-receptor axis in nonalcoholic steatohepatitis-related hepatocellular carcinomaRole of receptor for advanced glycation end products (RAGE) in liver diseaseTelmisartan inhibits cell proliferation by blocking nuclear translocation of ProHB-EGF C-terminal fragment in colon cancer cellsThe "Metabolic Memory" Theory and the Early Treatment of Hyperglycemia in Prevention of Diabetic ComplicationsToxic AGE (TAGE) Theory for the Pathophysiology of the Onset/Progression of NAFLD and ALDInvolvement of the TAGE-RAGE system in non-alcoholic steatohepatitis: Novel treatment strategiesOpen-label, Randomized Crossover Study Between Telmisartan and Valsartan on Improving Insulin Resistance and Adipocytokines in Nondiabetic Patients with Mild HypertensionMetabolic actions of angiotensin receptor antagonists: PPAR-gamma agonist actions or a class effect?The AGE/RAGE axis in diabetes-accelerated atherosclerosis.Anti-inflammatory activity of telmisartan in rat models of experimentally-induced chronic inflammation: Comparative study with dexamethasone.Angiotensin-receptor blockers as therapy for mild-to-moderate hypertension-associated non-alcoholic steatohepatitisPPARgamma and its ligands: therapeutic implications in cardiovascular disease.Potential of the angiotensin receptor blockers (ARBs) telmisartan, irbesartan, and candesartan for inhibiting the HMGB1/RAGE axis in prevention and acute treatment of stroke.Telmisartan: a different angiotensin II receptor blocker protecting a different population?Advanced glycation end products, oxidative stress and diabetic nephropathy.What is the role of the receptor for advanced glycation end products-ligand axis in liver injury?Cardiovascular disease and high-mobility group box 1--is a new inflammatory killer in town?Tissue level of advanced glycation end products is an independent determinant of high-sensitivity C-reactive protein levels in haemodialysis patients.Telmisartan, an angiotensin II type 1 receptor blocker, inhibits advanced glycation end-product (AGE)-elicited hepatic insulin resistance via peroxisome proliferator-activated receptor-gamma activation.Involvement of TAGE-RAGE System in the Pathogenesis of Diabetic Retinopathy.Olmesartan protects endothelial cells against oxidative stress-mediated cellular injury.Ω3-Polyunsaturated fatty acids prevent lipoperoxidation, modulate antioxidant enzymes, and reduce lipid content but do not alter glycogen metabolism in the livers of diabetic rats fed on a high fat thermolyzed diet.
P2860
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P2860
Telmisartan inhibits AGE-induced C-reactive protein production through downregulation of the receptor for AGE via peroxisome proliferator-activated receptor-gamma activation.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Telmisartan inhibits AGE-induc ...... ted receptor-gamma activation.
@en
type
label
Telmisartan inhibits AGE-induc ...... ted receptor-gamma activation.
@en
prefLabel
Telmisartan inhibits AGE-induc ...... ted receptor-gamma activation.
@en
P2093
P1433
P1476
Telmisartan inhibits AGE-induc ...... ted receptor-gamma activation.
@en
P2093
K Nakamura
M Takeuchi
S Yamagishi
T Imaizumi
P2888
P304
P356
10.1007/S00125-006-0437-7
P577
2006-09-27T00:00:00Z