Leishmania promastigotes activate PI3K/Akt signalling to confer host cell resistance to apoptosis.
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Proteomic analysis of the secretome of Leishmania donovaniChronic infection by Leishmania amazonensis mediated through MAPK ERK mechanismsHost-Cell Survival and Death During Chlamydia InfectionDeficiency of p110δ isoform of the phosphoinositide 3 kinase leads to enhanced resistance to Leishmania donovaniGrowth factor and Th2 cytokine signaling pathways converge at STAT6 to promote arginase expression in progressive experimental visceral leishmaniasisIdentification of Leishmania proteins preferentially released in infected cells using change mediated antigen technology (CMAT).Mammalian Innate Immune Response to a Leishmania-Resident RNA Virus Increases Macrophage Survival to Promote Parasite PersistenceSleeping with the enemy: how intracellular pathogens cope with a macrophage lifestyle.Intracellular pathogen Leishmania donovani activates hypoxia inducible factor-1 by dual mechanism for survival advantage within macrophage.Deception and manipulation: the arms of leishmania, a successful parasiteHost-mediated Leishmania donovani treatment using AR-12 encapsulated in acetalated dextran microparticles.MicroRNA expression profile in human macrophages in response to Leishmania major infectionLeishmania infantum Induces Mild Unfolded Protein Response in Infected Macrophages.FcgammaRIII mediates immunoglobulin G-induced interleukin-10 and is required for chronic Leishmania mexicana lesions.Leishmania donovani Exploits Myeloid Cell Leukemia 1 (MCL-1) Protein to Prevent Mitochondria-dependent Host Cell Apoptosis.Suicide prevention: disruption of apoptotic pathways by protozoan parasites.The amastigote forms of Leishmania are experts at exploiting host cell processes to establish infection and persist.The Leishmania surface protease GP63 cleaves multiple intracellular proteins and actively participates in p38 mitogen-activated protein kinase inactivation.Leishmania infection inhibits cycloheximide-induced macrophage apoptosis in a strain-dependent manner.Infection with Leishmania major induces a cellular stress response in macrophagesModulation of mammalian apoptotic pathways by intracellular protozoan parasites.Secreted virulence factors and immune evasion in visceral leishmaniasis.The role of vitamin D in the control of Leishmania infection.Macrophage cell death in microbial infections.The human parasite Leishmania amazonensis downregulates iNOS expression via NF-κB p50/p50 homodimer: role of the PI3K/Akt pathway.Resistance to apoptosis and autophagy leads to enhanced survival in Sertoli cells.A BODIPY-embedding miltefosine analog linked to cell-penetrating Tat(48-60) peptide favors intracellular delivery and visualization of the antiparasitic drug.Leishmania donovani prevents oxidative burst-mediated apoptosis of host macrophages through selective induction of suppressors of cytokine signaling (SOCS) proteins.Wedelolactone inhibits LPS-induced pro-inflammation via NF-kappaB pathway in RAW 264.7 cells.mTOR signaling pathway regulates the IL-12/IL-10 axis in Leishmania donovani infection.Leishmania donovani infection down-regulates TLR2-stimulated IL-12p40 and activates IL-10 in cells of macrophage/monocytic lineage by modulating MAPK pathways through a contact-dependent mechanism.Ammonium trichloro [1,2-ethanediolato-O,O']-tellurate cures experimental visceral leishmaniasis by redox modulation of Leishmania donovani trypanothione reductase and inhibiting host integrin linked PI3K/Akt pathway.Apoptotic induction induces Leishmania aethiopica and L. mexicana spreading in terminally differentiated THP-1 cells.The role of PD-1 in regulation of macrophage apoptosis and its subversion by Leishmania donovani.Leishmania donovani inhibits macrophage apoptosis and pro-inflammatory response through AKT-mediated regulation of β-catenin and FOXO-1Leishmania mexicana amastigotes inhibit p38 and JNK and activate PI3K/AKT: role in the inhibition of apoptosis of dendritic cells.Apoptotic-like Leishmania exploit the host's autophagy machinery to reduce T-cell-mediated parasite elimination.Neutrophil extracellular traps release induced by Leishmania: role of PI3Kγ, ERK, PI3Kσ, PKC, and [Ca2+].Systems Approach Reveals Nuclear Factor Erythroid 2-Related Factor 2/Protein Kinase R Crosstalk in Human Cutaneous Leishmaniasis.Activation of PI3K/Akt signaling has a dominant negative effect on IL-12 production by macrophages infected with Leishmania amazonensis promastigotes
P2860
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P2860
Leishmania promastigotes activate PI3K/Akt signalling to confer host cell resistance to apoptosis.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Leishmania promastigotes activ ...... cell resistance to apoptosis.
@en
type
label
Leishmania promastigotes activ ...... cell resistance to apoptosis.
@en
prefLabel
Leishmania promastigotes activ ...... cell resistance to apoptosis.
@en
P2093
P1476
Leishmania promastigotes activ ...... cell resistance to apoptosis.
@en
P2093
Aaron Ruhland
Nicole Leal
Peter E Kima
P356
10.1111/J.1462-5822.2006.00769.X
P577
2006-08-02T00:00:00Z