The sarcolemmal calcium pump, alpha-1 syntrophin, and neuronal nitric-oxide synthase are parts of a macromolecular protein complex.
about
Regulation of cardiovascular cellular processes by S-nitrosylationSyntrophin mutation associated with long QT syndrome through activation of the nNOS-SCN5A macromolecular complexBlood pressure is regulated by an alpha1D-adrenergic receptor/dystrophin signalosomeSpecific role of neuronal nitric-oxide synthase when tethered to the plasma membrane calcium pump in regulating the beta-adrenergic signal in the myocardiumCardiac nitric oxide synthase-1 localization within the cardiomyocyte is accompanied by the adaptor protein, CAPONS-nitrosylation in cardiovascular signalingCompromised redox homeostasis, altered nitroso-redox balance, and therapeutic possibilities in atrial fibrillationNitric oxide synthases, S-nitrosylation and cardiovascular health: from molecular mechanisms to therapeutic opportunities (review)Channelopathies from mutations in the cardiac sodium channel protein complexAbnormal Ca(2+) cycling in failing ventricular myocytes: role of NOS1-mediated nitroso-redox balanceDefective interactions of protein partner with ion channels and transporters as alternative mechanisms of membrane channelopathiesα1-Syntrophin Variant Identified in Drug-Induced Long QT Syndrome Increases Late Sodium CurrentInteraction of alpha1-syntrophin with multiple isoforms of heterotrimeric G protein alpha subunitsMolecular interactions of the plasma membrane calcium ATPase 2 at pre- and post-synaptic sites in rat cerebellumThe Subcellular Localisation of Neuronal Nitric Oxide Synthase Determines the Downstream Effects of NO on Myocardial FunctionUp-regulation of miR-31 in human atrial fibrillation begets the arrhythmia by depleting dystrophin and neuronal nitric oxide synthaseDigenic inheritance novel mutations in SCN5a and SNTA1 increase late I(Na) contributing to LQT syndrome.Platelet PMCA- and SERCA-type Ca2+ -ATPase expression in diabetes: a novel signature of abnormal megakaryocytopoiesis.Alpha1-syntrophin mutations identified in sudden infant death syndrome cause an increase in late cardiac sodium currentMechanochemotransduction during cardiomyocyte contraction is mediated by localized nitric oxide signaling.Defining a new paradigm for human arrhythmia syndromes: phenotypic manifestations of gene mutations in ion channel- and transporter-associated proteins.Alpha-dystrobrevin-1 recruits alpha-catulin to the alpha1D-adrenergic receptor/dystrophin-associated protein complex signalosome.Diseases caused by mutations in Nav1.5 interacting proteinsThe plasma membrane Ca²+ ATPase and the plasma membrane sodium calcium exchanger cooperate in the regulation of cell calcium.Plasma membrane Ca2+ ATPases as dynamic regulators of cellular calcium handling.Plasma membrane calcium ATPase proteins as novel regulators of signal transduction pathways.Plasma membrane calcium ATPases as novel candidates for therapeutic agent developmentDistinct pathophysiological mechanisms of cardiomyopathy in hearts lacking dystrophin or the sarcoglycan complex.Nitric Oxide Protects L-Type Calcium Channel of Cardiomyocyte during Long-Term Isoproterenol Stimulation in Tail-Suspended Rats.Plasma membrane calcium pump (PMCA4)-neuronal nitric-oxide synthase complex regulates cardiac contractility through modulation of a compartmentalized cyclic nucleotide microdomain.Isoform-specific up-regulation of plasma membrane Ca2+ATPase expression during colon and gastric cancer cell differentiationThe regulation of the Na/Ca exchanger and plasmalemmal Ca2+ ATPase by other proteins.Plasma membrane calcium ATPase 4b inhibits nitric oxide generation through calcium-induced dynamic interaction with neuronal nitric oxide synthase.PSD-95 mediates membrane clustering of the human plasma membrane Ca2+ pump isoform 4b.The role of biglycan in the heart.The neurobiology of the dystrophin-associated glycoprotein complex.A novel mechanism for the treatment of angina, arrhythmias, and diastolic dysfunction: inhibition of late I(Na) using ranolazine.Endothelium-driven myocardial growth or nitric oxide at the crossroads.Calcium pumps in health and disease.Calcium signaling dysfunction in heart disease.
P2860
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P2860
The sarcolemmal calcium pump, alpha-1 syntrophin, and neuronal nitric-oxide synthase are parts of a macromolecular protein complex.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
The sarcolemmal calcium pump, ...... acromolecular protein complex.
@en
type
label
The sarcolemmal calcium pump, ...... acromolecular protein complex.
@en
prefLabel
The sarcolemmal calcium pump, ...... acromolecular protein complex.
@en
P2093
P356
P1476
The sarcolemmal calcium pump, ...... acromolecular protein complex.
@en
P2093
Aly O Zaki
Angel L Armesilla
Cassandra L Hagarty
Elizabeth J Cartwright
Fiona H McIntyre
Judith C Williams
Ludwig Neyses
Mamta H Buch
Michael Emerson
Sybille Schomburg
P304
23341-23348
P356
10.1074/JBC.M513341200
P407
P577
2006-05-30T00:00:00Z