Structural effects of an LQT-3 mutation on heart Na+ channel gating.
about
Voltage-gated sodium channel-associated proteins and alternative mechanisms of inactivation and blockSolution Structure of the NaV1.2 C-terminal EF-hand DomainDistinct functional defect of three novel Brugada syndrome related cardiac sodium channel mutationsCardiac Na Channels: Structure to FunctionMechanistic links between Na+ channel (SCN5A) mutations and impaired cardiac pacemaking in sick sinus syndrome.Sodium channel inactivation: molecular determinants and modulation.Using computational modeling to predict arrhythmogenesis and antiarrhythmic therapy.Mutation of sodium channel SCN3A in a patient with cryptogenic pediatric partial epilepsyA carboxyl-terminal hydrophobic interface is critical to sodium channel function. Relevance to inherited disorders.Calmodulin mediates Ca2+ sensitivity of sodium channels.Calmodulin limits pathogenic Na+ channel persistent current.Distribution of cardiac sodium channels in clusters potentiates ephaptic interactions in the intercalated disc.
P2860
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P2860
Structural effects of an LQT-3 mutation on heart Na+ channel gating.
description
2004 nî lūn-bûn
@nan
2004年の論文
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2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
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2004年论文
@zh-cn
name
Structural effects of an LQT-3 mutation on heart Na+ channel gating.
@en
type
label
Structural effects of an LQT-3 mutation on heart Na+ channel gating.
@en
prefLabel
Structural effects of an LQT-3 mutation on heart Na+ channel gating.
@en
P2093
P2860
P1433
P1476
Structural effects of an LQT-3 mutation on heart Na+ channel gating.
@en
P2093
P2860
P304
P356
10.1016/S0006-3495(04)74251-4
P407
P577
2004-03-01T00:00:00Z