Infection and injury of neurons by West Nile encephalitis virus.
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Caspase-12 controls West Nile virus infection via the viral RNA receptor RIG-I.Chemokine receptor CCR5 promotes leukocyte trafficking to the brain and survival in West Nile virus infectionThe Israeli strain IS-98-ST1 of West Nile virus as viral model for West Nile encephalitis in the Old WorldThe Interferon-Stimulated Gene Ifi27l2a Restricts West Nile Virus Infection and Pathogenesis in a Cell-Type- and Region-Specific MannerDual Function of Ccr5 during Langat Virus Encephalitis: Reduction in Neutrophil-Mediated Central Nervous System Inflammation and Increase in T Cell-Mediated Viral ClearanceWest Nile Virus Infection in the Central Nervous SystemSpinal Cord Ventral Horns and Lymphoid Organ Involvement in Powassan Virus Infection in a Mouse ModelNuclear localization of Japanese encephalitis virus core protein enhances viral replication.Complement activation is required for induction of a protective antibody response against West Nile virus infectionNeuronal CXCL10 directs CD8+ T-cell recruitment and control of West Nile virus encephalitis.Alpha/beta interferon protects against lethal West Nile virus infection by restricting cellular tropism and enhancing neuronal survivalCD8+ T Cells Require Perforin To Clear West Nile Virus from Infected NeuronsWest Nile Virus Discriminates between DC-SIGN and DC-SIGNR for Cellular Attachment and InfectionPKR and RNase L Contribute to Protection against Lethal West Nile Virus Infection by Controlling Early Viral Spread in the Periphery and Replication in NeuronsPathogenesis of West Nile Virus Infection: a Balance between Virulence, Innate and Adaptive Immunity, and Viral EvasionCaspase 3-Dependent Cell Death of Neurons Contributes to the Pathogenesis of West Nile Virus EncephalitisInhibition of apoptosis prevents West Nile virus induced cell deathNovel Strategy for Treatment of Viral Central Nervous System Infection by Using a Cell-Permeating Inhibitor of c-Jun N-Terminal KinaseCell-Specific IRF-3 Responses Protect against West Nile Virus Infection by Interferon-Dependent and -Independent MechanismsEffect of exogenous interferon and an interferon inducer on western equine encephalitis virus disease in a hamster modelAxonal transport mediates West Nile virus entry into the central nervous system and induces acute flaccid paralysisWest Nile Virus-Induced Neuroinflammation: Glial Infection and Capsid Protein-Mediated NeurovirulenceWest Nile Virus Infection Activates the Unfolded Protein Response, Leading to CHOP Induction and ApoptosisIdentification of Novel Small-Molecule Inhibitors of West Nile Virus InfectionFas Ligand Interactions Contribute to CD8+ T-Cell-Mediated Control of West Nile Virus Infection in the Central Nervous SystemDifferential Effects of Mutations in NS4B on West Nile Virus Replication and Inhibition of Interferon SignalingDrak2 contributes to West Nile virus entry into the brain and lethal encephalitisCXCR4 antagonism increases T cell trafficking in the central nervous system and improves survival from West Nile virus encephalitisTumor Necrosis Factor Alpha Protects against Lethal West Nile Virus Infection by Promoting Trafficking of Mononuclear Leukocytes into the Central Nervous SystemWest Nile virus–induced acute flaccid paralysis is prevented by monoclonal antibody treatment when administered after infection of spinal cord neuronsAxonal degeneration as a self-destructive defense mechanism against neurotropic virus infectionThe structural immunology of antibody protection against West Nile virusPersistent West Nile Virus Associated with a Neurological Sequela in Hamsters Identified by Motor Unit Number EstimationWest Nile virus infection modulates human brain microvascular endothelial cells tight junction proteins and cell adhesion molecules: Transmigration across the in vitro blood-brain barrierThe Immune Adaptor Molecule SARM Modulates Tumor Necrosis Factor Alpha Production and Microglia Activation in the Brainstem and Restricts West Nile Virus PathogenesisProgress on the development of therapeutics against West Nile virusApoptosis in animal models of virus-induced diseaseIPS-1 Is Essential for the Control of West Nile Virus Infection and ImmunityThe interferon-inducible gene viperin restricts West Nile virus pathogenesisChemokine Receptor Ccr7 Restricts Fatal West Nile Virus Encephalitis
P2860
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P2860
Infection and injury of neurons by West Nile encephalitis virus.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Infection and injury of neurons by West Nile encephalitis virus.
@en
type
label
Infection and injury of neurons by West Nile encephalitis virus.
@en
prefLabel
Infection and injury of neurons by West Nile encephalitis virus.
@en
P2860
P1433
P1476
Infection and injury of neurons by West Nile encephalitis virus.
@en
P2093
David Gottlieb
P2860
P304
13203-13213
P356
10.1128/JVI.77.24.13203-13213.2003
P407
P577
2003-12-01T00:00:00Z