Defects in cGMP-PKG pathway contribute to impaired NO-dependent responses in hepatic stellate cells upon activation.
about
Intravenous and gastric cerium dioxide nanoparticle exposure disrupts microvascular smooth muscle signalingNO-independent stimulators and activators of soluble guanylate cyclase: discovery and therapeutic potentialThe cGMP signaling pathway as a therapeutic target in heart failure with preserved ejection fractionPathophysiology of portal hypertensionHepatic stellate cells: protean, multifunctional, and enigmatic cells of the liverPlatelet-derived growth factor signaling through ephrin-b2 regulates hepatic vascular structure and function.Nitric oxide augments mesenchymal stem cell ability to repair liver fibrosis.Inducible NOS mediates CNP-induced relaxation of intestinal myofibroblasts.Role of estrogen receptor β selective agonist in ameliorating portal hypertension in rats with CCl4-induced liver cirrhosisNitric oxide promotes caspase-independent hepatic stellate cell apoptosis through the generation of reactive oxygen speciesStellate cell contraction: role, regulation, and potential therapeutic target.Minocycline attenuates cirrhotic cardiomyopathy and portal hypertension in a rat model: Possible involvement of nitric oxide pathwayHemodynamic alterations in cirrhosis and portal hypertension.Endothelin and hepatic wound healing.Endothelial dysfunction in the regulation of cirrhosis and portal hypertension.Current concepts on the role of nitric oxide in portal hypertension.Antifibrotic effects of ZK14, a novel nitric oxide-donating biphenyldicarboxylate derivative, on rat HSC-T6 cells and CCl4-induced hepatic fibrosis.Everolimus is a potent inhibitor of activated hepatic stellate cell functions in vitro and in vivo, while demonstrating anti-angiogenic activities.Molecular pathophysiology of portal hypertension.Protein kinase G signaling disrupts Rac1-dependent focal adhesion assembly in liver specific pericytesNitric oxide-mediated inhibition of taurocholate uptake involves S-nitrosylation of NTCPActivated pericyte attenuates endothelial functions: nitric oxide-cGMP rescues activated pericyte-associated endothelial dysfunctions.Fabrication and characterization of an inorganic gold and silica nanoparticle mediated drug delivery system for nitric oxide.Liraglutide improves liver microvascular dysfunction in cirrhosis: Evidence from translational studies.The soluble guanylate cyclase stimulator riociguat reduces fibrogenesis and portal pressure in cirrhotic rats.Taurocholic acid is an active promoting factor, not just a biomarker of progression of liver cirrhosis: evidence from a human metabolomic study and in vitro experiments
P2860
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P2860
Defects in cGMP-PKG pathway contribute to impaired NO-dependent responses in hepatic stellate cells upon activation.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Defects in cGMP-PKG pathway co ...... tellate cells upon activation.
@en
type
label
Defects in cGMP-PKG pathway co ...... tellate cells upon activation.
@en
prefLabel
Defects in cGMP-PKG pathway co ...... tellate cells upon activation.
@en
P2093
P2860
P356
P1476
Defects in cGMP-PKG pathway co ...... stellate cells upon activation
@en
P2093
Daniel A Langer
Gianrico Farrugia
Jay Gadgil
Roman E Perri
Suvro Chatterjee
Vijay H Shah
P2860
P304
P356
10.1152/AJPGI.00297.2005
P577
2005-11-03T00:00:00Z