Chronically increased oxidative stress in fibroblasts from Alzheimer's disease patients causes early senescence and renders resistance to apoptosis by oxidative stress.

Chronically increased oxidative stress in fibroblasts from Alzheimer's disease patients causes early senescence and renders resistance to apoptosis by oxidative stress.

Item
http://www.wikidata.org/entity/Q40368198
Inactivation of CDK/pRb pathway normalizes survival pattern of lymphoblasts expressing the FTLD-progranulin mutation c.709-1G>AChronic treatment with resveratrol induces redox stress- and ataxia telangiectasia-mutated (ATM)-dependent senescence in p53-positive cancer cells.Sporadic Alzheimer disease fibroblasts display an oxidative stress phenotypeDynamin-like protein 1 reduction underlies mitochondrial morphology and distribution abnormalities in fibroblasts from sporadic Alzheimer's disease patients.The role of abnormal mitochondrial dynamics in the pathogenesis of Alzheimer's diseaseAlterations in microRNA expression in stress-induced cellular senescence.Peripheral mitochondrial dysfunction in Alzheimer's disease: focus on lymphocytes.Human dermal fibroblasts in psychiatry research.Integrated High-Content Quantification of Intracellular ROS Levels and Mitochondrial Morphofunction.Familial Alzheimer's Disease Lymphocytes Respond Differently Than Sporadic Cells to Oxidative Stress: Upregulated p53-p21 Signaling Linked with Presenilin 1 Mutants.Mitochondrial Bioenergetics Is Altered in Fibroblasts from Patients with Sporadic Alzheimer's Disease.Ubisol-Q10 (a Nanomicellar Water-Soluble Formulation of CoQ10) Treatment Inhibits Alzheimer-Type Behavioral and Pathological Symptoms in a Double Transgenic Mouse (TgAPEswe, PSEN1dE9) Model of Alzheimer's Disease.The increased density of p38 mitogen-activated protein kinase-immunoreactive microglia in the sensorimotor cortex of aged TgCRND8 mice is associated predominantly with smaller dense-core amyloid plaques.Mitochondrial permeability transition pore contributes to mitochondrial dysfunction in fibroblasts of patients with sporadic Alzheimer's disease
P2860

Chronically increased oxidative stress in fibroblasts from Alzheimer's disease patients causes early senescence and renders resistance to apoptosis by oxidative stress.

Item
http://www.wikidata.org/entity/Q40368198
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Chronically increased oxidativ ...... apoptosis by oxidative stress.
@en
type
label
Chronically increased oxidativ ...... apoptosis by oxidative stress.
@en
prefLabel
Chronically increased oxidativ ...... apoptosis by oxidative stress.
@en
P1433
P1476
P2093
P304
P31
P356
P433
P478
P577
P698
P921
P356
P1433
P1476
Chronically increased oxidativ ...... apoptosis by oxidative stress.
@en
P2093
P304
P31
P356
P433
P478
P577
P698
P921