Chronic treatment with resveratrol induces redox stress- and ataxia telangiectasia-mutated (ATM)-dependent senescence in p53-positive cancer cells.
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Multiple molecular targets of resveratrol: Anti-carcinogenic mechanismsThe role of cullin 5-containing ubiquitin ligasesCancer prevention with promising natural products: mechanisms of action and molecular targetsResveratrol Directly Binds to Mitochondrial Complex I and Increases Oxidative Stress in Brain Mitochondria of Aged MiceXenohormetic and anti-aging activity of secoiridoid polyphenols present in extra virgin olive oil: a new family of gerosuppressant agentsResveratrol inhibits the growth of gastric cancer by inducing G1 phase arrest and senescence in a Sirt1-dependent mannerResveratrol inhibits cisplatin-induced epithelial-to-mesenchymal transition in ovarian cancer cell linesArsenic trioxide and resveratrol show synergistic anti-leukemia activity and neutralized cardiotoxicityInvolvement of autophagy induction in penta-1,2,3,4,6-O-galloyl-β-D-glucose-induced senescence-like growth arrest in human cancer cellsResveratrol sequentially induces replication and oxidative stresses to drive p53-CXCR2 mediated cellular senescence in cancer cells.Resveratrol suppresses colitis and colon cancer associated with colitis.Therapy-induced senescence in cancerAnti-angiogenic effects of resveratrol mediated by decreased VEGF and increased TSP1 expression in melanoma-endothelial cell co-cultureDirect activation of ATM by resveratrol under oxidizing conditions.Metformin and the ATM DNA damage response (DDR): accelerating the onset of stress-induced senescence to boost protection against cancerHigh Potency of a Novel Resveratrol Derivative, 3,3',4,4'-Tetrahydroxy-trans-stilbene, against Ovarian Cancer Is Associated with an Oxidative Stress-Mediated Imbalance between DNA Damage Accumulation and RepairInduced pluripotent stem cells from ataxia-telangiectasia recapitulate the cellular phenotype.Mobilization of Intracellular Copper by Gossypol and Apogossypolone Leads to Reactive Oxygen Species-Mediated Cell Death: Putative Anticancer MechanismReactive oxygen species: a double-edged sword in oncogenesis.Multifaceted approach to resveratrol bioactivity: Focus on antioxidant action, cell signaling and safety.The anticancer effects of resveratrol: modulation of transcription factors.Oxygen regulates molecular mechanisms of cancer progression and metastasis.Cell death mechanisms of plant-derived anticancer drugs: beyond apoptosis.The role of reactive oxygen species and subsequent DNA-damage response in the emergence of resistance towards resveratrol in colon cancer models.Inhibition of HDAC increases the senescence induced by natural polyphenols in glioma cells.Induction of DNA damage and p21-dependent senescence by Riccardin D is a novel mechanism contributing to its growth suppression in prostate cancer cells in vitro and in vivo.Plant polyphenol induced cell death in human cancer cells involves mobilization of intracellular copper ions and reactive oxygen species generation: a mechanism for cancer chemopreventive action.Sulfate metabolites provide an intracellular pool for resveratrol generation and induce autophagy with senescence.Resveratrol metabolites inhibit human metastatic colon cancer cells progression and synergize with chemotherapeutic drugs to induce cell death.Cellular and Molecular Targets of Resveratrol on Lymphoma and Leukemia Cells.Resveratrol acts as a topoisomerase II poison in human glioma cells.Penta-1,2,3,4,6-O-galloyl-beta-D-glucose induces senescence-like terminal S-phase arrest in human hepatoma and breast cancer cells.Resveratrol enhances anti-proliferative effect of VACM-1/cul5 in T47D cancer cells.Selenium compounds activate early barriers of tumorigenesis.Haplodeficiency of Ataxia Telangiectasia Mutated Accelerates Heart Failure After Myocardial Infarction.Active NF-E2-related factor (Nrf2) contributes to keep endothelial NO synthase (eNOS) in the coupled state: role of reactive oxygen species (ROS), eNOS, and heme oxygenase (HO-1) levels.Resveratrol prevents the expression of glaucoma markers induced by chronic oxidative stress in trabecular meshwork cellsPlant polyphenols mobilize nuclear copper in human peripheral lymphocytes leading to oxidatively generated DNA breakage: implications for an anticancer mechanism.Oligosaccharide G19 inhibits U-87 MG human glioma cells growth in vitro and in vivo by targeting epidermal growth factor (EGF) and activating p53/p21 signaling.
P2860
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P2860
Chronic treatment with resveratrol induces redox stress- and ataxia telangiectasia-mutated (ATM)-dependent senescence in p53-positive cancer cells.
description
2007 nî lūn-bûn
@nan
2007 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Chronic treatment with resvera ...... in p53-positive cancer cells.
@ast
Chronic treatment with resvera ...... in p53-positive cancer cells.
@en
Chronic treatment with resvera ...... ataxia telangiectasia-mutated
@nl
type
label
Chronic treatment with resvera ...... in p53-positive cancer cells.
@ast
Chronic treatment with resvera ...... in p53-positive cancer cells.
@en
Chronic treatment with resvera ...... ataxia telangiectasia-mutated
@nl
prefLabel
Chronic treatment with resvera ...... in p53-positive cancer cells.
@ast
Chronic treatment with resvera ...... in p53-positive cancer cells.
@en
Chronic treatment with resvera ...... ataxia telangiectasia-mutated
@nl
P2860
P356
P1476
Chronic treatment with resvera ...... e in p53-positive cancer cells
@en
P2093
Yvonne D C Schilder
P2860
P304
26759-26766
P356
10.1074/JBC.M703229200
P407
P577
2007-07-11T00:00:00Z