Snail and Slug are major determinants of ovarian cancer invasiveness at the transcription level.
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Mesenchyme Forkhead 1 (FOXC2) plays a key role in metastasis and is associated with aggressive basal-like breast cancersHER-2 overexpression differentially alters transforming growth factor-beta responses in luminal versus mesenchymal human breast cancer cellsTight Junctions and the Tumor MicroenvironmentEmerging role of cancer stem cells in the biology and treatment of ovarian cancer: basic knowledge and therapeutic possibilities for an innovative approachSLUG: Critical regulator of epithelial cell identity in breast development and cancerA comprehensive panel of three-dimensional models for studies of prostate cancer growth, invasion and drug responsesNeuraminidase-1: a novel therapeutic target in multistage tumorigenesisSlug deficiency enhances self-renewal of hematopoietic stem cells during hematopoietic regenerationResveratrol inhibits cisplatin-induced epithelial-to-mesenchymal transition in ovarian cancer cell linesWithaferin a alone and in combination with cisplatin suppresses growth and metastasis of ovarian cancer by targeting putative cancer stem cellsThe LIM protein AJUBA recruits protein arginine methyltransferase 5 to mediate SNAIL-dependent transcriptional repressionSnai2 expression enhances ultraviolet radiation-induced skin carcinogenesisTGF-beta-induced epithelial to mesenchymal transitionInduction of Slug by Chronic Exposure to Single-Walled Carbon Nanotubes Promotes Tumor Formation and Metastasis.An NF-kappaB and slug regulatory loop active in early vertebrate mesoderm.Potential Involvement of Snail Members in Neuronal Survival and Astrocytic Migration during the Gecko Spinal Cord RegenerationSNAI2/Slug promotes growth and invasion in human gliomas.Hydrogen peroxide mediates EGF-induced down-regulation of E-cadherin expression via p38 MAPK and snail in human ovarian cancer cells.Estrogen regulates Snail and Slug in the down-regulation of E-cadherin and induces metastatic potential of ovarian cancer cells through estrogen receptor alpha.A three-dimensional in vitro model of tumor cell intravasation.Phenotypic plasticity of neoplastic ovarian epithelium: unique cadherin profiles in tumor progressionConstitutive activation of BMP signalling abrogates experimental metastasis of OVCA429 cells via reduced cell adhesionIndometacin ameliorates high glucose-induced proliferation and invasion via modulation of e-cadherin in pancreatic cancer cells.Clinicopathologic significance of slug expression in human intrahepatic cholangiocarcinoma.Epithelial-mesenchymal transition markers in pancreatic ductal adenocarcinomaSlug suppression induces apoptosis via Puma transactivation in rheumatoid arthritis fibroblast-like synoviocytes treated with hydrogen peroxide.The E-cadherin repressor slug and progression of human extrahepatic hilar cholangiocarcinoma.The EMT (epithelial-mesenchymal-transition)-related protein expression indicates the metastatic status and prognosis in patients with ovarian cancer.Expression of transcription factors snail, slug, and twist in human bladder carcinomaTWIST and ovarian cancer stem cells: implications for chemoresistance and metastasisDysregulation of JAM-A plays an important role in human tumor progressionProteomics profiling of Madin-Darby canine kidney plasma membranes reveals Wnt-5a involvement during oncogenic H-Ras/TGF-beta-mediated epithelial-mesenchymal transition.Interference of suppressor of cytokine signaling 3 promotes epithelial-mesenchymal transition in MHCC97H cells.Lymphatic or hematogenous dissemination: how does a metastatic tumor cell decide?Fibroblast growth factor 2 induces E-cadherin down-regulation via PI3K/Akt/mTOR and MAPK/ERK signaling in ovarian cancer cellsCD151-α3β1 integrin complexes suppress ovarian tumor growth by repressing slug-mediated EMT and canonical Wnt signaling.Slug enhances invasion ability of pancreatic cancer cells through upregulation of matrix metalloproteinase-9 and actin cytoskeleton remodeling.Prevalence of epithelial ovarian cancer stem cells correlates with recurrence in early-stage ovarian cancer.Adenomatous polyposis coli (APC) is essential for maintaining the integrity of the seminiferous epithelium.Pituitary tumor transforming gene induces epithelial to mesenchymal transition by regulation of Twist, Snail, Slug, and E-cadherin.
P2860
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P2860
Snail and Slug are major determinants of ovarian cancer invasiveness at the transcription level.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Snail and Slug are major deter ...... ss at the transcription level.
@en
type
label
Snail and Slug are major deter ...... ss at the transcription level.
@en
prefLabel
Snail and Slug are major deter ...... ss at the transcription level.
@en
P2093
P1433
P1476
Snail and Slug are major deter ...... ss at the transcription level.
@en
P2093
P304
P356
10.1016/J.YGYNO.2004.12.043
P407
P577
2005-04-01T00:00:00Z