Resveratrol inhibits cardiac hypertrophy via AMP-activated protein kinase and Akt. - Wikidata - awgldk - TriplyDB

Resveratrol inhibits cardiac hypertrophy via AMP-activated protein kinase and Akt.

Resveratrol inhibits cardiac hypertrophy via AMP-activated protein kinase and Akt.

http://www.wikidata.org/entity/Q40452406

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Resveratrol inhibits mTOR signaling by promoting the interaction between mTOR and DEPTOR Resveratrol and Cardiovascular Diseases Redox Signaling in Diabetic Nephropathy: Hypertrophy versus Death Choices in Mesangial Cells and Podocytes Sirtuin 1 and sirtuin 3: physiological modulators of metabolism Therapeutic targeting of autophagy: potential and concerns in treating cardiovascular disease Experimental studies of the molecular pathways regulated by exercise and resveratrol in heart, skeletal muscle and the vasculature AMPK: An emerging target for modification of injury-induced pain plasticity.Resveratrol improves survival, hemodynamics and energetics in a rat model of hypertension leading to heart failure Metabolic shifts during aging and pathology Cardiac myocyte follistatin-like 1 functions to attenuate hypertrophy following pressure overload Resveratrol Reverses Functional Chagas Heart Disease in Mice Activation of AMPK inhibits cardiomyocyte hypertrophy by modulating of the FOXO1/MuRF1 signaling pathway in vitro Validation of anti-aging drugs by treating age-related diseases.Exogenous NAD blocks cardiac hypertrophic response via activation of the SIRT3-LKB1-AMP-activated kinase pathway Resveratrol, an activator of SIRT1, upregulates sarcoplasmic calcium ATPase and improves cardiac function in diabetic cardiomyopathy Concerted regulation of cGMP and cAMP phosphodiesterases in early cardiac hypertrophy induced by angiotensin II Is alcohol beneficial or harmful for cardioprotection?Role of AMP-activated protein kinase in ferritin H gene expression by resveratrol in human T cells.The FoxO family in cardiac function and dysfunction.The Role of SIRT1 in Diabetic Kidney Disease.Resveratrol inhibits development of experimental endometriosis in vivo and reduces endometrial stromal cell invasiveness in vitro.Cellular and mitochondrial effects of alcohol consumption.Resveratrol potentiates glucose-stimulated insulin secretion in INS-1E beta-cells and human islets through a SIRT1-dependent mechanism.Resveratrol-activated SIRT1 in liver and pancreatic β-cells: a Janus head looking to the same direction of metabolic homeostasis Resveratrol retards progression of diabetic nephropathy through modulations of oxidative stress, proinflammatory cytokines, and AMP-activated protein kinase.Continued postnatal administration of resveratrol prevents diet-induced metabolic syndrome in rat offspring born growth restricted.Resveratrol inhibits mTOR signaling by targeting DEPTOR.Mitochondrial oxidative stress corrupts coronary collateral growth by activating adenosine monophosphate activated kinase-α signaling.EGCG Blocked Phenylephrin-Induced Hypertrophy in H9C2 Cardiomyocytes, by Activating AMPK-Dependent Pathway Hormesis does not make sense except in the light of TOR-driven aging.Translating cell survival and cell longevity into treatment strategies with SIRT1 Targeting cardiovascular disease with novel SIRT1 pathways.Perspectives of Targeting mTORC1-S6K1 in Cardiovascular Aging SIRT1: new avenues of discovery for disorders of oxidative stress Mechanisms linking obesity, chronic kidney disease, and fatty liver disease: the roles of fetuin-A, adiponectin, and AMPK.Regulation of NAD(P)H oxidases by AMPK in cardiovascular systems AMPK deficiency in cardiac muscle results in dilated cardiomyopathy in the absence of changes in energy metabolism.SIRT1-PGC1α-NFκB Pathway of Oxidative and Inflammatory Stress during Trypanosoma cruzi Infection: Benefits of SIRT1-Targeted Therapy in Improving Heart Function in Chagas Disease.Targeting AMPK: From Ancient Drugs to New Small-Molecule Activators.Resveratrol as a calorie restriction mimetic: therapeutic implications.

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P2860

Resveratrol inhibits cardiac hypertrophy via AMP-activated protein kinase and Akt.

http://www.wikidata.org/entity/Q40452406

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

@zh-cn

name

Resveratrol inhibits cardiac hypertrophy via AMP-activated protein kinase and Akt.

@en

type

label

Resveratrol inhibits cardiac hypertrophy via AMP-activated protein kinase and Akt.

@en

prefLabel

Resveratrol inhibits cardiac hypertrophy via AMP-activated protein kinase and Akt.

@en

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Journal of Biological Chemistry

P1476

Resveratrol inhibits cardiac hypertrophy via AMP-activated protein kinase and Akt.

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P2093

Anita Y M Chan

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Carrie-Lynn M Soltys

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Jason R B Dyck

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Peter E Light

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Shairaz Baksh

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Vernon W Dolinsky

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P2860

Inhibition of glycogen synthase kinase-3 by insulin mediated by protein kinase B

Resveratrol improves health and survival of mice on a high-calorie diet

A calcineurin-dependent transcriptional pathway for cardiac hypertrophy

Expression of an active LKB1 complex in cardiac myocytes results in decreased protein synthesis associated with phenylephrine-induced hypertrophy

Myocardial ischemia and increased heart work modulate the phosphorylation state of eukaryotic elongation factor-2

Why is left ventricular hypertrophy so predictive of morbidity and mortality?

Adiponectin-mediated modulation of hypertrophic signals in the heart

Calcineurin/NFAT coupling participates in pathological, but not physiological, cardiac hypertrophy.

Akt/protein kinase B promotes organ growth in transgenic mice.

5'-AMP-activated protein kinase (AMPK) is induced by low-oxygen and glucose deprivation conditions found in solid-tumor microenvironments.

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24194-24201

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10.1074/JBC.M802869200

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35

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283

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18562309

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3259789

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