The role of hyperthermia and metabolism as mechanisms of tolerance to methamphetamine neurotoxicity.
about
Mephedrone, an abused psychoactive component of 'bath salts' and methamphetamine congener, does not cause neurotoxicity to dopamine nerve endings of the striatumMethamphetamine toxicity and messengers of deathThe effects of methamphetamine self-administration on cortical monoaminergic deficits induced by subsequent high-dose methamphetamine administrationsMethamphetamine preconditioning alters midbrain transcriptional responses to methamphetamine-induced injury in the rat striatumMethamphetamine self-administration is associated with persistent biochemical alterations in striatal and cortical dopaminergic terminals in the ratLow concentrations of methamphetamine can protect dopaminergic cells against a larger oxidative stress injury: mechanistic studyMethamphetamine sensitization attenuates the febrile and neuroinflammatory response to a subsequent peripheral immune stimulusMethamphetamine self-administration attenuates hippocampal serotonergic deficits: role of brain-derived neurotrophic factor.Disparity in the temporal appearance of methamphetamine-induced apoptosis and depletion of dopamine terminal markers in the striatum of mice.A neurotoxic regimen of methamphetamine exacerbates the febrile and neuroinflammatory response to a subsequent peripheral immune stimulus.Methamphetamine preconditioning causes differential changes in striatal transcriptional responses to large doses of the drug.Long-term protective effects of methamphetamine preconditioning against single-day methamphetamine toxic challenges.Prior methamphetamine self-administration attenuates the dopaminergic deficits caused by a subsequent methamphetamine exposure.Prior methamphetamine self-administration attenuates serotonergic deficits induced by subsequent high-dose methamphetamine administrations.Perseverative behavior in rats with methamphetamine-induced neurotoxicity.Methamphetamine preconditioning: differential protective effects on monoaminergic systems in the rat brainRepeated exposure to MDMA provides neuroprotection against subsequent MDMA-induced serotonin depletion in brain.Methamphetamine-induced dopaminergic deficits and refractoriness to subsequent treatment.Preconditioning provides neuroprotection in models of CNS disease: paradigms and clinical significance.Methamphetamine addiction: involvement of CREB and neuroinflammatory signaling pathways.Chronic methamphetamine self-administration disrupts cortical control of cognition.Current understanding of methamphetamine-associated dopaminergic neurodegeneration and psychotoxic behaviors.Attenuated microglial activation mediates tolerance to the neurotoxic effects of methamphetamine.Differential neurochemical consequences of an escalating dose-binge regimen followed by single-day multiple-dose methamphetamine challenges.Methamphetamine self-administration modulates glutamate neurophysiology.
P2860
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P2860
The role of hyperthermia and metabolism as mechanisms of tolerance to methamphetamine neurotoxicity.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
The role of hyperthermia and m ...... methamphetamine neurotoxicity.
@en
type
label
The role of hyperthermia and m ...... methamphetamine neurotoxicity.
@en
prefLabel
The role of hyperthermia and m ...... methamphetamine neurotoxicity.
@en
P2093
P1476
The role of hyperthermia and m ...... methamphetamine neurotoxicity.
@en
P2093
Annette E Fleckenstein
Diana G Wilkins
Kamisha L Johnson-Davis
P304
P356
10.1016/J.EJPHAR.2003.09.063
P407
P577
2003-12-01T00:00:00Z