LOX Fails to Substitute for RANKL in Osteoclastogenesis.

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Osteoimmunology: The Conceptual Framework Unifying the Immune and Skeletal Systems.Established Models and New Paradigms for Hypoxia-Driven Cancer-Associated Bone Disease.Functionally distinct disease-associated fibroblast subsets in rheumatoid arthritis.

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P2860

LOX Fails to Substitute for RANKL in Osteoclastogenesis.

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http://www.wikidata.org/entity/Q40547452

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2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

@zh-cn

name

LOX Fails to Substitute for RANKL in Osteoclastogenesis.

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type

Item

label

LOX Fails to Substitute for RANKL in Osteoclastogenesis.

@en

prefLabel

LOX Fails to Substitute for RANKL in Osteoclastogenesis.

@en

P1476

LOX Fails to Substitute for RANKL in Osteoclastogenesis.

@en

P2093

Asuka Terashima

http://www.w3.org/2001/XMLSchema#string

Hiroshi Takayanagi

http://www.w3.org/2001/XMLSchema#string

Hisataka Yasuda

http://www.w3.org/2001/XMLSchema#string

Kazuo Okamoto

http://www.w3.org/2001/XMLSchema#string

Koki Hamada

http://www.w3.org/2001/XMLSchema#string

Masayuki Tsukasaki

http://www.w3.org/2001/XMLSchema#string

Noriko Komatsu

http://www.w3.org/2001/XMLSchema#string

Stephanie J Win

http://www.w3.org/2001/XMLSchema#string

Tadashi Okamura

http://www.w3.org/2001/XMLSchema#string

Takeshi Nitta

http://www.w3.org/2001/XMLSchema#string

P2860

Central control of fever and female body temperature by RANKL/RANK

Osteoimmunology: shared mechanisms and crosstalk between the immune and bone systems

OPGL is a key regulator of osteoclastogenesis, lymphocyte development and lymph-node organogenesis

RANKL Is Necessary and Sufficient to Initiate Development of Antigen-Sampling M Cells in the Intestinal Epithelium

Induction and activation of the transcription factor NFATc1 (NFAT2) integrate RANKL signaling in terminal differentiation of osteoclasts

Tumor necrosis factor alpha stimulates osteoclast differentiation by a mechanism independent of the ODF/RANKL-RANK interaction

Osteoclast differentiation independent of the TRANCE-RANK-TRAF6 axis.

RANK is the intrinsic hematopoietic cell surface receptor that controls osteoclastogenesis and regulation of bone mass and calcium metabolism

The hypoxic cancer secretome induces pre-metastatic bone lesions through lysyl oxidase.

TNF-alpha induces osteoclastogenesis by direct stimulation of macrophages exposed to permissive levels of RANK ligand

P304

434-439

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scholarly article

P356

10.1002/JBMR.2990

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http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

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Josef M Penninger

Kazuki Nagashima

P577

2016-09-08T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

27606829

http://www.w3.org/2001/XMLSchema#string

LOX Fails to Substitute for RANKL in Osteoclastogenesis.

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P2860

P2860

LOX Fails to Substitute for RANKL in Osteoclastogenesis.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698