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Up-regulation of Mcl-1 and Bak by coronavirus infection of human, avian and animal cells modulates apoptosis and viral replicationMulti-organ lesions in suckling mice infected with SARS-associated mammalian reovirus linked with apoptosis induced by viral proteins μ1 and σ1The open reading frame 3a protein of severe acute respiratory syndrome-associated coronavirus promotes membrane rearrangement and cell death.Interactions of SARS coronavirus nucleocapsid protein with the host cell proteasome subunit p42.Equine torovirus (BEV) induces caspase-mediated apoptosis in infected cellsPathology and pathogenesis of severe acute respiratory syndromePalmitoylation of SARS-CoV S protein is necessary for partitioning into detergent-resistant membranes and cell-cell fusion but not interaction with M protein.The cytoplasmic tail of the severe acute respiratory syndrome coronavirus spike protein contains a novel endoplasmic reticulum retrieval signal that binds COPI and promotes interaction with membrane protein.Induction of apoptosis by the severe acute respiratory syndrome coronavirus 7a protein is dependent on its interaction with the Bcl-XL protein.Regulation of cell death during infection by the severe acute respiratory syndrome coronavirus and other coronaviruses.Severe acute respiratory syndrome coronavirus triggers apoptosis via protein kinase R but is resistant to its antiviral activityThe replication of a mouse adapted SARS-CoV in a mouse cell line stably expressing the murine SARS-CoV receptor mACE2 efficiently induces the expression of proinflammatory cytokinesNucleoprotein of influenza A virus negatively impacts antiapoptotic protein API5 to enhance E2F1-dependent apoptosis and virus replication.Correlation between TGF-β1 expression and proteomic profiling induced by severe acute respiratory syndrome coronavirus papain-like protease.SARS-CoV nucleocapsid protein induced apoptosis of COS-1 mediated by the mitochondrial pathway.7a protein of severe acute respiratory syndrome coronavirus inhibits cellular protein synthesis and activates p38 mitogen-activated protein kinase.Adenovirus-mediated expression of the C-terminal domain of SARS-CoV spike protein is sufficient to induce apoptosis in Vero E6 cells.Overexpression of 7a, a protein specifically encoded by the severe acute respiratory syndrome coronavirus, induces apoptosis via a caspase-dependent pathway.The SARS coronavirus nucleocapsid protein induces actin reorganization and apoptosis in COS-1 cells in the absence of growth factors.Accelerated induction of apoptosis in insect cells by baculovirus-expressed SARS-CoV membrane protein.The SARS-coronavirus membrane protein induces apoptosis via interfering with PDK1-PKB/Akt signalling.
P2860
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P2860
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
SARS coronavirus induces apoptosis in Vero E6 cells.
@en
type
label
SARS coronavirus induces apoptosis in Vero E6 cells.
@en
prefLabel
SARS coronavirus induces apoptosis in Vero E6 cells.
@en
P2093
P2860
P356
P1476
SARS coronavirus induces apoptosis in Vero E6 cells.
@en
P2093
Congyi Zheng
Gengfu Xiao
Huimin Yan
Jiamin Zhang
Yuanyang Hu
P2860
P304
P356
10.1002/JMV.20094
P577
2004-07-01T00:00:00Z