Cisplatin-induced cell death is EGFR/src/ERK signaling dependent in mouse proximal tubule cells. - Wikidata - awgldk - TriplyDB

Cisplatin-induced cell death is EGFR/src/ERK signaling dependent in mouse proximal tubule cells.

Cisplatin-induced cell death is EGFR/src/ERK signaling dependent in mouse proximal tubule cells.

http://www.wikidata.org/entity/Q40554483

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An integrated view of cisplatin-induced nephrotoxicity and ototoxicity Membrane Transition Temperature Determines Cisplatin Response Differential effects of tannic acid on cisplatin induced nephrotoxicity in rats.IP(3) receptor antagonist, 2-APB, attenuates cisplatin induced Ca2+-influx in HeLa-S3 cells and prevents activation of calpain and induction of apoptosis.Triphala inhibits both in vitro and in vivo xenograft growth of pancreatic tumor cells by inducing apoptosis.Mechanistic elucidation of the antitumor properties of withaferin a in breast cancer.Renalase prevents AKI independent of amine oxidase activity Pharmacological inhibition of Src kinase protects against acute kidney injury in a murine model of renal ischemia/reperfusion.Protective effect of rutin supplementation against cisplatin-induced Nephrotoxicity in rats Pathophysiology of cisplatin-induced acute kidney injury.LIN-3/EGF promotes the programmed cell death of specific cells in Caenorhabditis elegans by transcriptional activation of the pro-apoptotic gene egl-1.Doxorubicin induces cytotoxicity through upregulation of pERK-dependent ATF3.OTOTOXIC EFFECTS OF CARBOPLATIN IN ORGANOTYPIC CULTURES IN CHINCHILLAS AND RATS Erk in kidney diseases.Epidermal growth factor receptor inhibition with erlotinib partially prevents cisplatin-induced nephrotoxicity in rats Inhibition of PKCδ reduces cisplatin-induced nephrotoxicity without blocking chemotherapeutic efficacy in mouse models of cancer Hyperactive EGF receptor, Jaks and Stat3 signaling promote enhanced colony-forming ability, motility and migration of cisplatin-resistant ovarian cancer cells.Induction of anterior gradient 2 (AGR2) plays a key role in insulin-like growth factor-1 (IGF-1)-induced breast cancer cell proliferation and migration.6-Formylindolo(3,2-b)Carbazole (FICZ) Modulates the Signalsome Responsible for RA-Induced Differentiation of HL-60 Myeloblastic Leukemia Cells.Activation of ERK1/2 pathway mediates oxidant-induced decreases in mitochondrial function in renal cells.Lower blood pressure and risk of cisplatin nephrotoxicity: a retrospective cohort study.Purinergic P2Y2 Receptor Control of Tissue Factor Transcription in Human Coronary Artery Endothelial Cells: NEW AP-1 TRANSCRIPTION FACTOR SITE AND NEGATIVE REGULATOR.PD98059 Protects Brain against Cells Death Resulting from ROS/ERK Activation in a Cardiac Arrest Rat Model Cisplatin cytotoxicity of auditory cells requires secretions of proinflammatory cytokines via activation of ERK and NF-kappaB.A Comprehensive Review on the Genetic Regulation of Cisplatin-induced Nephrotoxicity ERK-mediated suppression of cilia in cisplatin-induced tubular cell apoptosis and acute kidney injury.Erythropoietin (EPO)-receptor signaling induces cell death of primary myeloma cells in vitro.Effects of hydroxyl radical scavenging on cisplatin-induced p53 activation, tubular cell apoptosis and nephrotoxicity.Role of CFTR in oxidative stress and suicidal death of renal cells during cisplatin-induced nephrotoxicity.How ERK1/2 activation controls cell proliferation and cell death: Is subcellular localization the answer?Activation and involvement of p53 in cisplatin-induced nephrotoxicity.Benzyl Isothiocyanate potentiates p53 signaling and antitumor effects against breast cancer through activation of p53-LKB1 and p73-LKB1 axes.ERK and cell death: mechanisms of ERK-induced cell death--apoptosis, autophagy and senescence.Signalling mechanisms involved in renal pathological changes during cisplatin-induced nephropathy.Biliverdin reductase: a target for cancer therapy?Pharmacotherapeutic options for treating adverse effects of Cisplatin chemotherapy.Silencing of fused toes homolog enhances cisplatin sensitivity in cervical cancer cells by inhibiting epidermal growth factor receptor-mediated repair of DNA damage.Folate-mediated chemotherapy and diagnostics: An updated review and outlook.MAPK signaling in cisplatin-induced death: predominant role of ERK1 over ERK2 in human hepatocellular carcinoma cells.C-phycocyanin attenuates cisplatin-induced nephrotoxicity in mice.

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Cisplatin-induced cell death is EGFR/src/ERK signaling dependent in mouse proximal tubule cells.

http://www.wikidata.org/entity/Q40554483

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AJPRENAL.00112.2004

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American Journal of Physiology - Renal Physiology

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Cisplatin-induced cell death i ...... n mouse proximal tubule cells.

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Hideaki Kaneto

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Istvan Arany

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Judit K Megyesi

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Peter M Price

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Robert L Safirstein

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P2860

Cell survival promoted by the Ras-MAPK signaling pathway by transcription-dependent and -independent mechanisms

Signal transduction by the JNK group of MAP kinases

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Involvement of c-Jun N-terminal kinase in oxidative stress-mediated suppression of insulin gene expression

Opposing effects of ERK and JNK-p38 MAP kinases on apoptosis

MAP kinase pathways.

Epidermal growth factor receptor: mechanisms of activation and signalling.

Pharmacokinetic and pharmacodynamic properties of EGFR inhibitors under clinical investigation.

Protein kinase C-alpha and ERK1/2 mediate mitochondrial dysfunction, decreases in active Na+ transport, and cisplatin-induced apoptosis in renal cells.

Enhancement of drug sensitivity of human malignancies by epidermal growth factor.

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