Mycobacterium tuberculosis inhibits macrophage responses to IFN-gamma through myeloid differentiation factor 88-dependent and -independent mechanisms.
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Immunotherapy for tuberculosis: future prospectsMycobacterium tuberculosis: Manipulator of Protective ImmunityTuberculosis vaccines and prevention of infectionExosomes and Exosomal miRNA in Respiratory DiseasesTrans-cyclopropanation of mycolic acids on trehalose dimycolate suppresses Mycobacterium tuberculosis -induced inflammation and virulenceEspA acts as a critical mediator of ESX1-dependent virulence in Mycobacterium tuberculosis by affecting bacterial cell wall integrityEffect of deletion or overexpression of the 19-kilodalton lipoprotein Rv3763 on the innate response to Mycobacterium tuberculosisThe SecA2 secretion factor of Mycobacterium tuberculosis promotes growth in macrophages and inhibits the host immune responseComparative Proteomic Analysis of Aminoglycosides Resistant and Susceptible Mycobacterium tuberculosis Clinical Isolates for Exploring Potential Drug TargetsReversion of the ELISPOT test after treatment in Gambian tuberculosis cases.Dynamic changes in pro- and anti-inflammatory cytokine profiles and gamma interferon receptor signaling integrity correlate with tuberculosis disease activity and response to curative treatment.Microdissection of the cytokine milieu of pulmonary granulomas from tuberculous guinea pigs.TLR2-dependent inhibition of macrophage responses to IFN-gamma is mediated by distinct, gene-specific mechanismsSelective inhibition of IFNG-induced autophagy by Mir155- and Mir31-responsive WNT5A and SHH signalingInduction of IFN-alphabeta enables Listeria monocytogenes to suppress macrophage activation by IFN-gamma.In vivo expression of innate immunity markers in patients with Mycobacterium tuberculosis infection.CD36 deficiency attenuates experimental mycobacterial infection.Mycobacterium tuberculosis EsxH inhibits ESCRT-dependent CD4+ T-cell activationThe adaptor molecule CARD9 is essential for tuberculosis control.Mycobacterium tuberculosis, macrophages, and the innate immune response: does common variation matter?Exosomes released from M. tuberculosis infected cells can suppress IFN-γ mediated activation of naïve macrophages.Suboptimal activation of antigen-specific CD4+ effector cells enables persistence of M. tuberculosis in vivo.Analysis of phagosomal proteomes: from latex-bead to bacterial phagosomesRegulation of antigen presentation by Mycobacterium tuberculosis: a role for Toll-like receptorsGenome-wide transcriptional profiling of peripheral blood leukocytes from cattle infected with Mycobacterium bovis reveals suppression of host immune genes.The stress-response factor SigH modulates the interaction between Mycobacterium tuberculosis and host phagocytesLipoproteins of bacterial pathogens.Global gene expression and systems biology analysis of bovine monocyte-derived macrophages in response to in vitro challenge with Mycobacterium bovisAntagonistic crosstalk between type I and II interferons and increased host susceptibility to bacterial infectionsBeyond macrophages: the diversity of mononuclear cells in tuberculosis.LAG3 expression in active Mycobacterium tuberculosis infectionsDevelopment of a secondary immune response to Mycobacterium tuberculosis is independent of Toll-like receptor 2.Host defense and recruitment of Foxp3⁺ T regulatory cells to the lungs in chronic Mycobacterium tuberculosis infection requires toll-like receptor 2Cell-mediated immune responses in tuberculosis.Mycobacterium tuberculosis triggers host type I IFN signaling to regulate IL-1β production in human macrophagesMycobacterium tuberculosis Hip1 dampens macrophage proinflammatory responses by limiting toll-like receptor 2 activationMycobacterial glycolipids di-O-acylated trehalose and tri-O-acylated trehalose downregulate inducible nitric oxide synthase and nitric oxide production in macrophages.Vitamin D is required for IFN-gamma-mediated antimicrobial activity of human macrophagesMannose-capped Lipoarabinomannan from Mycobacterium tuberculosis induces IL-37 production via upregulating ERK1/2 and p38 in human type II alveolar epithelial cells.ESX-1 exploits type I IFN-signalling to promote a regulatory macrophage phenotype refractory to IFNγ-mediated autophagy and growth restriction of intracellular mycobacteria.
P2860
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P2860
Mycobacterium tuberculosis inhibits macrophage responses to IFN-gamma through myeloid differentiation factor 88-dependent and -independent mechanisms.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Mycobacterium tuberculosis inh ...... t and -independent mechanisms.
@en
type
label
Mycobacterium tuberculosis inh ...... t and -independent mechanisms.
@en
prefLabel
Mycobacterium tuberculosis inh ...... t and -independent mechanisms.
@en
P2093
P1476
Mycobacterium tuberculosis inh ...... t and -independent mechanisms.
@en
P2093
Alejandra Jaeger
Alejandra Solache
Barry R Bloom
Eric J Rubin
John T Belisle
Preston J Hill
Sarah M Fortune
P304
P356
10.4049/JIMMUNOL.172.10.6272
P407
P50
P577
2004-05-01T00:00:00Z