Radiation to stromal fibroblasts increases invasiveness of pancreatic cancer cells through tumor-stromal interactions. - Wikidata - awgldk - TriplyDB

Radiation to stromal fibroblasts increases invasiveness of pancreatic cancer cells through tumor-stromal interactions.

Radiation to stromal fibroblasts increases invasiveness of pancreatic cancer cells through tumor-stromal interactions.

http://www.wikidata.org/entity/Q40559132

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Mesenchymal Stem/Stromal Cells in Stromal Evolution and Cancer Progression c-Jun N-Terminal Kinases Mediate a Wide Range of Targets in the Metastatic Cascade Radiotherapy and the tumor stroma: the importance of dose and fractionation Identifying new small molecule anti-invasive compounds for glioma treatment The effect of environmental chemicals on the tumor microenvironment Stromal fibroblasts in cancer initiation and progression The senescence-associated secretory phenotype: the dark side of tumor suppression Organotypic culture model of pancreatic cancer demonstrates that stromal cells modulate E-cadherin, beta-catenin, and Ezrin expression in tumor cells.PAK4 interacts with p85 alpha: implications for pancreatic cancer cell migration Quantitative analysis of hTERT mRNA levels in cells microdissected from cytological specimens.Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty acids enhancing the invasiveness of pancreatic cancer cells.Smad4-mediated TGF-beta signaling in tumorigenesis.Tumor-stroma interactions Prospectively isolated cancer-associated CD10(+) fibroblasts have stronger interactions with CD133(+) colon cancer cells than with CD133(-) cancer cells.Recruitment of CD34(+) fibroblasts in tumor-associated reactive stroma: the reactive microvasculature hypothesis.Revisiting the hallmarks of cancer.Modeling progression in radiation-induced lung adenocarcinomas.Senescent cells as a source of inflammatory factors for tumor progression.Overexpression of c-met in the early stage of pancreatic carcinogenesis; altered expression is not sufficient for progression from chronic pancreatitis to pancreatic cancer Fibroblast heterogeneity in the cancer wound.A reciprocal role of prostate cancer on stromal DNA damage.MicroRNA, hsa-miR-200c, is an independent prognostic factor in pancreatic cancer and its upregulation inhibits pancreatic cancer invasion but increases cell proliferation.Verteporfin-based photodynamic therapy overcomes gemcitabine insensitivity in a panel of pancreatic cancer cell lines.Gene expression levels as predictive markers of outcome in pancreatic cancer after gemcitabine-based adjuvant chemotherapy.CD166/ALCAM expression is characteristic of tumorigenicity and invasive and migratory activities of pancreatic cancer cells Perspectives on tissue interactions in development and disease.Pancreatic cancer cells enhance the ability of collagen internalization during epithelial-mesenchymal transition.Escherichia coli producing colibactin triggers premature and transmissible senescence in mammalian cells.CD271⁺ subpopulation of pancreatic stellate cells correlates with prognosis of pancreatic cancer and is regulated by interaction with cancer cells Evidences of early senescence in multiple myeloma bone marrow mesenchymal stromal cells.Stromal mediation of radiation carcinogenesis.Role of heparanase in radiation-enhanced invasiveness of pancreatic carcinoma.MicroRNA expression as a predictive marker for gemcitabine response after surgical resection of pancreatic cancer.Pancreatic satellite cells derived galectin-1 increase the progression and less survival of pancreatic ductal adenocarcinoma.Radiation-enhancement of MDA-MB-231 breast cancer cell invasion prevented by a cyclooxygenase-2 inhibitor.Simultaneous irradiation of fibroblasts and carcinoma cells repress the secretion of soluble factors able to stimulate carcinoma cell migration.Quantitative proteomic analysis of single or fractionated radiation-induced proteins in human breast cancer MDA-MB-231 cells Hypoxia-inducible factor 1α (HIF-1α) and reactive oxygen species (ROS) mediates radiation-induced invasiveness through the SDF-1α/CXCR4 pathway in non-small cell lung carcinoma cells.1,25(OH)2D3 Deficiency Induces Colon Inflammation via Secretion of Senescence-Associated Inflammatory Cytokines Cancer-associated fibroblasts from human NSCLC survive ablative doses of radiation but their invasive capacity is reduced.

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P2860

Radiation to stromal fibroblasts increases invasiveness of pancreatic cancer cells through tumor-stromal interactions.

http://www.wikidata.org/entity/Q40559132

description

2004 nî lūn-bûn

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

@zh-mo

2004年論文

@zh-tw

2004年论文

@wuu

2004年论文

@zh

2004年论文

@zh-cn

name

Radiation to stromal fibroblas ...... gh tumor-stromal interactions.

@en

type

label

Radiation to stromal fibroblas ...... gh tumor-stromal interactions.

@en

prefLabel

Radiation to stromal fibroblas ...... gh tumor-stromal interactions.

@en

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0008-5472.CAN-03-2464

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Cancer Research

P1476

Radiation to stromal fibroblas ...... gh tumor-stromal interactions.

@en

P2093

Eishi Nagai

http://www.w3.org/2001/XMLSchema#string

Kazuhiro Mizumoto

http://www.w3.org/2001/XMLSchema#string

Kenoki Ohuchida

http://www.w3.org/2001/XMLSchema#string

Kunio Matsumoto

http://www.w3.org/2001/XMLSchema#string

Li-Wu Qian

http://www.w3.org/2001/XMLSchema#string

Masao Tanaka

http://www.w3.org/2001/XMLSchema#string

Mitsuhiko Murakami

http://www.w3.org/2001/XMLSchema#string

Norihiro Sato

http://www.w3.org/2001/XMLSchema#string

Toshikazu Nakamura

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3215-3222

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scholarly article

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10.1158/0008-5472.CAN-03-2464

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9

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64

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2004-05-01T00:00:00Z

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8579873

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15126362

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