Imatinib mesylate (STI571) decreases the vascular endothelial growth factor plasma concentration in patients with chronic myeloid leukemia.
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VEGF and FGF prime vascular tube morphogenesis and sprouting directed by hematopoietic stem cell cytokines.Exosomes released by K562 chronic myeloid leukemia cells promote angiogenesis in a Src-dependent fashion.Clinical relevance of vascular endothelial growth factor (VEGFA) and VEGF receptor (VEGFR2) gene polymorphism on the treatment outcome following imatinib therapy.Human cytomegalovirus-encoded chemokine receptor US28 promotes tumorigenesis.Alternative vascularization mechanisms in cancer: Pathology and therapeutic implications.Role of exosomes released by chronic myelogenous leukemia cells in angiogenesis.Imatinib mesylate (Gleevec) in advanced breast cancer-expressing C-Kit or PDGFR-beta: clinical activity and biological correlationsAntiangiogenic cancer treatment: The great discovery and greater complexity (Review).Sp1 phosphorylation and its regulation of gene transcription.Targeting the vascular endothelial growth factor in hematologic malignancies.Allium Roseum L. Extract Exerts Potent Suppressive Activities on Chronic Myeloid Leukemia K562 Cell Viability Through the Inhibition of BCR-ABL, PI3K/Akt, and ERK1/2 Pathways and the Abrogation of VEGF Secretion.Transcriptional regulation of survivin by c-Myc in BCR/ABL-transformed cells: implications in anti-leukaemic strategy.Downregulation of VEGF-A, STAT5 and AKT in acute myeloid leukemia blasts of patients treated with SU5416.Bilateral Central Retinal Vein Occlusion as Presenting Feature of Chronic Myeloid Leukemia.Oxidative stress promotes transcriptional up-regulation of Fyn in BCR-ABL1-expressing cells.Defective blood dendritic cells in chronic myeloid leukemia correlate with high plasmatic VEGF and are not normalized by imatinib mesylate.Suppression of vascular endothelial growth factor (VEGF) expression by targeting the Bcr-Abl oncogene and protein tyrosine kinase activity in Bcr-Abl-positive leukaemia cells.Lepirudin: is the approved dosing schedule too high?Elevated plasma levels of vascular endothelial growth factor is associated with marked splenomegaly in chronic myeloid leukemia.Imatinib has the potential to exert its antileukemia effects by down-regulating hERG1 K+ channels in chronic myelogenous leukemia.
P2860
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P2860
Imatinib mesylate (STI571) decreases the vascular endothelial growth factor plasma concentration in patients with chronic myeloid leukemia.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Imatinib mesylate (STI571) dec ...... with chronic myeloid leukemia.
@en
type
label
Imatinib mesylate (STI571) dec ...... with chronic myeloid leukemia.
@en
prefLabel
Imatinib mesylate (STI571) dec ...... with chronic myeloid leukemia.
@en
P2093
P921
P1433
P1476
Imatinib mesylate (STI571) dec ...... with chronic myeloid leukemia
@en
P2093
Christine Bourcier
François-Xavier Mahon
Gilles Pagès
Jill-Patrice Cassuto
Laurence Legros
P304
P356
10.1182/BLOOD-2003-08-2695
P407
P577
2004-02-19T00:00:00Z