Inhibition of cell proliferation and cell cycle progression by specific inhibition of basal JNK activity: evidence that mitotic Bcl-2 phosphorylation is JNK-independent.
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The functional contrariety of JNKJNK Inhibition Inhibits Lateral Line Neuromast Hair Cell Development.G1 to S phase transition protein 1 induces apoptosis signal-regulating kinase 1 activation by dissociating 14-3-3 from ASK1Role of somatostatin receptor-2 in gentamicin-induced auditory hair cell loss in the Mammalian inner earThe apoptotic mechanism of action of the sphingosine kinase 1 selective inhibitor SKI-178 in human acute myeloid leukemia cell lines.Effect of JNK inhibitor SP600125 on hair cell regeneration in zebrafish (Danio rerio) larvae.Ras/p38 and PI3K/Akt but not Mek/Erk signaling mediate BDNF-induced neurite formation on neonatal cochlear spiral ganglion explantsc-Jun-NH2-kinase-1 inhibition leads to antitumor activity in ovarian cancer.The phosphorylation status and anti-apoptotic activity of Bcl-2 are regulated by ERK and protein phosphatase 2A on the mitochondria.Up-regulation of SEPT9_v1 stabilizes c-Jun-N-terminal kinase and contributes to its pro-proliferative activity in mammary epithelial cells.A human ribonuclease induces apoptosis associated with p21WAF1/CIP1 induction and JNK inactivation.Activation of the Jun N-terminal kinase pathway by friend spleen focus-forming virus and its role in the growth and survival of friend virus-induced erythroleukemia cells.Activation of c-Jun N-terminal kinase (JNK) during mitosis in retinal progenitor cells.Virtual target screening: validation using kinase inhibitors.Selective repression of low-density lipoprotein receptor expression by SP600125: coupling of histone H3-Ser10 phosphorylation and Sp1 occupancyCop1 constitutively regulates c-Jun protein stability and functions as a tumor suppressor in mice.Calcium-activated K+ channels increase cell proliferation independent of K+ conductance.SP600125 suppresses Cdk1 and induces endoreplication directly from G2 phase, independent of JNK inhibition.Identification and characterization of novel immunomodulatory bursal-derived pentapeptide-II (BPP-II)Posttranslational modification of vesicular stomatitis virus glycoprotein, but not JNK inhibition, is the antiviral mechanism of SP600125High-throughput RNAi screening of human kinases identifies predictors of clinical outcome in colorectal cancer patients treated with oxaliplatin.Aberrant expression of IL-22 receptor 1 and autocrine IL-22 stimulation contribute to tumorigenicity in ALK+ anaplastic large cell lymphoma.TSLP signaling network revealed by SILAC-based phosphoproteomicsThromboxane A2 Receptor Inhibition Suppresses Multiple Myeloma Cell Proliferation by Inducing p38/c-Jun N-terminal Kinase (JNK) Mitogen-activated Protein Kinase (MAPK)-mediated G2/M Progression Delay and Cell Apoptosis.Ajuba family proteins link JNK to Hippo signaling.JNK inhibitor SP600125 promotes the formation of polymerized tubulin, leading to G2/M phase arrest, endoreduplication, and delayed apoptosis.Basal p38 mitogen-activated protein kinase regulates unliganded glucocorticoid receptor function in airway smooth muscle cells.β1 integrin- and JNK-dependent tumor growth upon hypofractionated radiation.Glycogen synthase kinase 3beta is a negative regulator of growth factor-induced activation of the c-Jun N-terminal kinase.The role of mitogen- and stress-activated protein kinase pathways in melanoma.Purinergic inhibition of Na⁺,K⁺,Cl⁻ cotransport in C11-MDCK cells: Role of stress-activated protein kinases.Role of p21 in SP600125-induced cell cycle arrest, endoreduplication, and apoptosis.Sphingosylphosphorylcholine induces proliferation of human adipose tissue-derived mesenchymal stem cells via activation of JNK.Microtubule destabilising agents: far more than just antimitotic anticancer drugs.Roles of phosphorylated JNK in esophageal squamous cell carcinomas of Kazakh ethnic.The nonsteroidal anti-inflammatory drug celecoxib suppresses the growth and induces apoptosis of human glioblastoma cells via the NF-κB pathway.The effect of the combined action of roscovitine and Paclitaxel on the apoptotic and cell cycle regulatory mechanisms in colon and anaplastic thyroid cancer cells.Apoptosis signal-regulating kinase1 is inducible by protein kinase Cδ and contributes to phorbol ester-mediated G1 phase arrest through persistent JNK activation.Inhibition of basal p38 or JNK activity enhances epithelial barrier function through differential modulation of claudin expression.Identification of the major phosphorylation site in Bcl-xL induced by microtubule inhibitors and analysis of its functional significance.
P2860
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P2860
Inhibition of cell proliferation and cell cycle progression by specific inhibition of basal JNK activity: evidence that mitotic Bcl-2 phosphorylation is JNK-independent.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Inhibition of cell proliferati ...... horylation is JNK-independent.
@en
type
label
Inhibition of cell proliferati ...... horylation is JNK-independent.
@en
prefLabel
Inhibition of cell proliferati ...... horylation is JNK-independent.
@en
P2093
P2860
P356
P1476
Inhibition of cell proliferati ...... horylation is JNK-independent.
@en
P2093
Brydon L Bennett
Christopher S Lyle
Jeffrey A Muir
Timothy C Chambers
Toria B Obey
William A Gaarde
P2860
P304
11957-11966
P356
10.1074/JBC.M304935200
P407
P577
2004-01-02T00:00:00Z