Porphyromonas gingivalis induces murine macrophage foam cell formation.
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Entry of Porphyromonas gingivalis outer membrane vesicles into epithelial cells causes cellular functional impairmentBiogenesis and function of Porphyromonas gingivalis outer membrane vesiclesCytokine profiling of macrophages exposed to Porphyromonas gingivalis, its lipopolysaccharide, or its FimA protein.Porphyromonas gingivalis GroEL induces osteoclastogenesis of periodontal ligament cells and enhances alveolar bone resorption in rats.Lipoproteins and lipoprotein metabolism in periodontal disease.Hemagglutinin B is involved in the adherence of Porphyromonas gingivalis to human coronary artery endothelial cells.Immune dysregulation mediated by the oral microbiome: potential link to chronic inflammation and atherosclerosis.Vulnerable populations and the association between periodontal and chronic kidney diseaseCD36/SR-B2-TLR2 Dependent Pathways Enhance Porphyromonas gingivalis Mediated Atherosclerosis in the Ldlr KO Mouse Model.Functional Advantages of Porphyromonas gingivalis Vesicles.Dihydroaustrasulfone Alcohol (WA-25) Impedes Macrophage Foam Cell Formation by Regulating the Transforming Growth Factor-β1 Pathway.Oral inflammatory diseases and systemic inflammation: role of the macrophage.Differential Responses of Pattern Recognition Receptors to Outer Membrane Vesicles of Three Periodontal PathogensBiological foundation for periodontitis as a potential risk factor for atherosclerosis.Mechanisms involved in the association between periodontal diseases and cardiovascular diseaseCommonality in chronic inflammatory diseases: periodontitis, diabetes, and coronary artery disease.Innate immune signaling and Porphyromonas gingivalis-accelerated atherosclerosis.Toll-like receptor 2 plays a critical role in the progression of atherosclerosis that is independent of dietary lipids.The periodontal host response with diabetes.Nasal immunization with Porphyromonas gingivalis outer membrane protein decreases P. gingivalis-induced atherosclerosis and inflammation in spontaneously hyperlipidemic miceEffects of Porphyromonas gingivalis lipopolysaccharide on the expression of key genes involved in cholesterol metabolism in macrophages.Are reactive oxygen species always detrimental to pathogens?Role of extracellular membrane vesicles in the pathogenesis of various diseases, including cancer, renal diseases, atherosclerosis, and arthritis.Signaling events in pathogen-induced macrophage foam cell formation.Commentary: changing paradigms in the oral disease-systemic disease relationship.Metabolic Remodeling, Inflammasome Activation, and Pyroptosis in Macrophages Stimulated by Porphyromonas gingivalis and Its Outer Membrane Vesicles.Homocysteine Metabolism, Atherosclerosis, and Diseases of Aging.Collagen-binding proteins of Streptococcus mutans and related streptococci.Outer membrane vesicles - offensive weapons or good Samaritans?Porphyromonas gingivalis lipopolysaccharide alters atherosclerotic-related gene expression in oxidized low-density-lipoprotein-induced macrophages and foam cells.Outer Membrane Vesicles Prime and Activate Macrophage Inflammasomes and Cytokine Secretion In Vitro and In Vivo.Role of MyD88-dependent and MyD88-independent signaling in Porphyromonas gingivalis-elicited macrophage foam cell formation.Deposition and hydrolysis of serine dipeptide lipids of Bacteroidetes bacteria in human arteries: relationship to atherosclerosis.Lipoxin A₄ inhibits porphyromonas gingivalis-induced aggregation and reactive oxygen species production by modulating neutrophil-platelet interaction and CD11b expression.Immunization with malondialdehyde-modified low-density lipoprotein (LDL) reduces atherosclerosis in LDL receptor-deficient mice challenged with Porphyromonas gingivalis.Porphyromonas gingivalis outer membrane vesicles enter human epithelial cells via an endocytic pathway and are sorted to lysosomal compartmentsScavenger receptor A is expressed by macrophages in response to Porphyromonas gingivalis, and participates in TNF-alpha expression.Severe periodontitis in young adults is associated with sub-clinical atherosclerosis.Induction of Porphyromonas gingivalis GroEL signaling via binding to Toll-like receptors 2 and 4.Linkage of Infection to Adverse Systemic Complications: Periodontal Disease, Toll-Like Receptors, and Other Pattern Recognition Systems.
P2860
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P2860
Porphyromonas gingivalis induces murine macrophage foam cell formation.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Porphyromonas gingivalis induces murine macrophage foam cell formation.
@en
type
label
Porphyromonas gingivalis induces murine macrophage foam cell formation.
@en
prefLabel
Porphyromonas gingivalis induces murine macrophage foam cell formation.
@en
P2093
P1476
Porphyromonas gingivalis induces murine macrophage foam cell formation.
@en
P2093
Hiroshi Miyakawa
Howard K Kuramitsu
Mingshan Qi
P304
P356
10.1016/J.MICPATH.2003.07.002
P577
2003-12-01T00:00:00Z