Platelet-activating factor antagonists enhance intracellular degradation of amyloid-β42 in neurons via regulation of cholesterol ester hydrolases.

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Role of Cholesterol and Phospholipids in Amylin Misfolding, Aggregation and Etiology of Islet Amyloidosis Current Understanding of Platelet-Activating Factor Signaling in Central Nervous System Diseases.Enhanced neuronal degradation of amyloid-β oligomers allows synapse regeneration.

P2860

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P2860

Platelet-activating factor antagonists enhance intracellular degradation of amyloid-β42 in neurons via regulation of cholesterol ester hydrolases.

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http://www.wikidata.org/entity/Q40624800

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2014 nî lūn-bûn

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2014年の論文

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2014年論文

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2014年論文

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2014年论文

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name

Platelet-activating factor ant ...... cholesterol ester hydrolases.

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Platelet-activating factor ant ...... cholesterol ester hydrolases.

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Platelet-activating factor ant ...... cholesterol ester hydrolases.

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P1476

Platelet-activating factor ant ...... cholesterol ester hydrolases.

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P2093

Alun Williams

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Charlotte Simmons

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Clive Bate

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Victoria Ingham

http://www.w3.org/2001/XMLSchema#string

P2860

Amyloid-beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

Soluble amyloid beta peptide concentration as a predictor of synaptic change in Alzheimer's disease

Accumulation of amyloid beta-protein in the low-density membrane domain accurately reflects the extent of beta-amyloid deposition in the brain

Evidence that phospholipase A2 activity is required for Golgi complex and trans Golgi network membrane tubulation

Natural oligomers of the Alzheimer amyloid-beta protein induce reversible synapse loss by modulating an NMDA-type glutamate receptor-dependent signaling pathway

Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins

A beta oligomers - a decade of discovery

Soluble pool of Abeta amyloid as a determinant of severity of neurodegeneration in Alzheimer's disease

Cystatin C-cathepsin B axis regulates amyloid beta levels and associated neuronal deficits in an animal model of Alzheimer's disease.

Caveolae are highly immobile plasma membrane microdomains, which are not involved in constitutive endocytic trafficking

P2888

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10.1186/ALZRT245

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English

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Platelet-activating factor antagonists enhance intracellular degradation of amyloid-β42 in neurons via regulation of cholesterol ester hydrolases.

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P2860

P2860

Platelet-activating factor antagonists enhance intracellular degradation of amyloid-β42 in neurons via regulation of cholesterol ester hydrolases.

description

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type

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P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P407

P433

P478

P577

P5875

P6179

P698

P932

P356

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P407

P433

P478

P577

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P6179

P698

P932