PKMζ is essential for spinal plasticity underlying the maintenance of persistent pain. - Wikidata - awgldk - TriplyDB

PKMζ is essential for spinal plasticity underlying the maintenance of persistent pain.

PKMζ is essential for spinal plasticity underlying the maintenance of persistent pain.

http://www.wikidata.org/entity/Q40661071

about

Commonalities between pain and memory mechanisms and their meaning for understanding chronic pain PKMζ knockdown disrupts post-ischemic long-term potentiation via inhibiting postsynaptic expression of aminomethyl phosphonic acid receptors Memory maintenance by PKMζ--an evolutionary perspective Structural Models for the Design of PKMzeta Inhibitors with Neurobiological Indications.Topical combinations to treat microvascular dysfunction of chronic postischemia pain Phrenic long-term facilitation requires PKCθ activity within phrenic motor neurons.Contribution of PKMζ-dependent and independent amplification to components of experimental neuropathic pain Plasticity-Related PKMζ Signaling in the Insular Cortex Is Involved in the Modulation of Neuropathic Pain after Nerve Injury.Zeta Inhibitory Peptide as a Novel Therapy to Control Chronic Visceral Hypersensitivity in a Rat Model The pharmacology of nociceptor priming Spinal atypical protein kinase C activity is necessary to stabilize inactivity-induced phrenic motor facilitation.The thalamo-cortical complex network correlates of chronic pain.Consistent sex-dependent effects of PKMζ gene ablation and pharmacological inhibition on the maintenance of referred pain.ZIPping to pain relief: the role (or not) of PKMζ in chronic pain.How to erase memory traces of pain and fear PKMζ Is Not Required for Development of Postsurgical Pain.BDNF regulates atypical PKC at spinal synapses to initiate and maintain a centralized chronic pain state.PKM and the maintenance of memory.The p75NTR signaling cascade mediates mechanical hyperalgesia induced by nerve growth factor injected into the rat hind paw.The Effect of Electroacupuncture on PKMzeta in the ACC in Regulating Anxiety-Like Behaviors in Rats Experiencing Chronic Inflammatory Pain.Role of a novel nociceptor autocrine mechanism in chronic pain.The TrkA receptor mediates experimental thermal hyperalgesia produced by nerve growth factor: Modulation by the p75 neurotrophin receptor.Peripheral Synthesis of an Atypical Protein Kinase C Mediates the Enhancement of Excitability and the Development of Mechanical Hyperalgesia Produced by Nerve Growth Factor.How is chronic pain related to sympathetic dysfunction and autonomic dysreflexia following spinal cord injury?Spinal protein kinase Mζ contributes to the maintenance of peripheral inflammation-primed persistent nociceptive sensitization after plantar incision.Spinal PKCα inhibition and gene-silencing for pain relief: AMPAR trafficking at the synapses between primary afferents and sensory interneurons.

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P2860

PKMζ is essential for spinal plasticity underlying the maintenance of persistent pain.

http://www.wikidata.org/entity/Q40661071

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

PKMζ is essential for spinal plasticity underlying the maintenance of persistent pain.

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type

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PKMζ is essential for spinal plasticity underlying the maintenance of persistent pain.

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prefLabel

PKMζ is essential for spinal plasticity underlying the maintenance of persistent pain.

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PKMζ is essential for spinal plasticity underlying the maintenance of persistent pain.

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P2093

Andre Laferrière

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Anne Haldane

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Fernando Cervero

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Mark H Pitcher

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Naresh Kumar

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Terence J Coderre

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Todd C Sacktor

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Virginia Cornea

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Yue Huang

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P2860

PKMzeta maintains spatial, instrumental, and classically conditioned long-term memories

Persistent activation of the zeta isoform of protein kinase C in the maintenance of long-term potentiation

Protein kinase M zeta synthesis from a brain mRNA encoding an independent protein kinase C zeta catalytic domain. Implications for the molecular mechanism of memory

Protein kinase Mzeta is necessary and sufficient for LTP maintenance

Ectopic discharge in Abeta afferents as a source of neuropathic pain

Storage of spatial information by the maintenance mechanism of LTP

Preserved acute pain and reduced neuropathic pain in mice lacking PKCgamma

Neuronal plasticity: increasing the gain in pain

Quantitative assessment of tactile allodynia in the rat paw

A new and sensitive method for measuring thermal nociception in cutaneous hyperalgesia

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