Chemokine stimulation of monocyte matrix metalloproteinase-9 requires endogenous TNF-alpha.
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Cathepsin D specifically cleaves the chemokines macrophage inflammatory protein-1 alpha, macrophage inflammatory protein-1 beta, and SLC that are expressed in human breast cancerHigh matrix metalloproteinase production correlates with immune activation and leukocyte migration in leprosy reactional lesions.Viral expression of CCL2 is sufficient to induce demyelination in RAG1-/- mice infected with a neurotropic coronavirus.TNFR1-mediated signaling is important to induce the improvement of liver fibrosis by bone marrow cell infusion.Breast cancer: coordinated regulation of CCL2 secretion by intracellular glycosaminoglycans and chemokine motifs.Effects of monocyte-endothelium interactions on the expression of type IV collagenases in monocytes.Lack of TNF-α-induced MMP-9 production and abnormal E-cadherin redistribution associated with compromised fusion in MCP-1-null macrophagesReduced expression of inducible gelatinase B/matrix metalloproteinase-9 in monocytes from patients with myelodysplastic syndrome: Correlation of inducible levels with the percentage of cytogenetically marked cells and with marrow cellularity.Isolevuglandin-type lipid aldehydes induce the inflammatory response of macrophages by modifying phosphatidylethanolamines and activating the receptor for advanced glycation endproducts.Modified huo-luo-xiao-ling dan suppresses adjuvant arthritis by inhibiting chemokines and matrix-degrading enzymes.Regulation of MMP-9 gene expression for the development of novel molecular targets against cancer and inflammatory diseases.Hypoxia, leukocytes, and the pulmonary circulation.Plasminogen is an important regulator in the pathogenesis of a murine model of asthmaRole of chemokines in tumor growth.Role of bone marrow-derived cells in angiogenesis: focus on macrophages and pericytes.Plasticity in tumor-promoting inflammation: impairment of macrophage recruitment evokes a compensatory neutrophil responseCirculating levels of matrix metalloproteinases and tissue inhibitors of matrix metalloproteinases during Japanese encephalitis virus infection.Melanoma-derived conditioned media efficiently induce the differentiation of monocytes to macrophages that display a highly invasive gene signature.Type conversion of secretomes in a 3D TAM2 and HCC cell co-culture system and functional importance of CXCL2 in HCC.Innate-adaptive crosstalk: how dendritic cells shape immune responses in the CNS.Mesenchymal stem cell-derived CCL-9 and CCL-5 promote mammary tumor cell invasion and the activation of matrix metalloproteinases.Tumor-associated macrophages and stromal TNF-α regulate collagen structure in a breast tumor model as visualized by second harmonic generation.Migratory neighbors and distant invaders: tumor-associated niche cellsIL-17 expression by breast-cancer-associated macrophages: IL-17 promotes invasiveness of breast cancer cell lines.Role of eotaxin-1 signaling in ovarian cancerMonocyte chemoattractant protein-1/CCL2 as a biomarker in acute coronary syndromes.CCL5 as an adjuvant for cancer immunotherapy.The Tumor-Promoting Flow of Cells Into, Within and Out of the Tumor Site: Regulation by the Inflammatory Axis of TNFα and Chemokines.Collaborating with the enemy: function of macrophages in the development of neoplastic disease.The potential to target CCL5/CCR5 in breast cancer.The associations between the MCP-1 -2518 A/G polymorphism and ischemic heart disease and ischemic stroke: a meta-analysis of 28 research studies involving 21,524 individuals.The role of dietary fatty acid intake in inflammatory gene expression: a critical review.Chemokines in the cancer microenvironment and their relevance in cancer immunotherapy.Divergent and synergistic regulation of matrix metalloprotease production by cytokines in combination with C-C chemokines.Effects of estradiol and progestogens on tumor-necrosis factor-alpha-induced changes of biochemical markers for breast cancer growth and metastasis.Inflammatory stimuli from macrophages and cancer cells synergistically promote tumor growth and angiogenesis.Burn injury reveals altered phenotype in mannan-binding lectin-deficient mice.Morphologic features and development of granulomatous vasculitis in feline infectious peritonitis.CP-481,715, a potent and selective CCR1 antagonist with potential therapeutic implications for inflammatory diseases.Cell type-specific release of matrix-metallo-proteinase-9 by bacterial chemoattractant in human blood phagocytic leukocytes.
P2860
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P2860
Chemokine stimulation of monocyte matrix metalloproteinase-9 requires endogenous TNF-alpha.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
2002年论文
@zh
2002年论文
@zh-cn
name
Chemokine stimulation of monoc ...... requires endogenous TNF-alpha.
@en
type
label
Chemokine stimulation of monoc ...... requires endogenous TNF-alpha.
@en
prefLabel
Chemokine stimulation of monoc ...... requires endogenous TNF-alpha.
@en
P2860
P1476
Chemokine stimulation of monoc ...... requires endogenous TNF-alpha.
@en
P2093
Kate A Scott
Stephen C Robinson
P2860
P304
P356
10.1002/1521-4141(200202)32:2<404::AID-IMMU404>3.0.CO;2-X
P407
P577
2002-02-01T00:00:00Z