TCR engagement in the absence of cell cycle progression leads to T cell anergy independent of p27(Kip1).
about
Molecular mechanisms for adaptive tolerance and other T cell anergy modelsIdentification of DNA methyltransferase 3a as a T cell receptor-induced regulator of Th1 and Th2 differentiation.Anergic T cells are metabolically anergic.Induction of T cell anergy: integration of environmental cues and infectious tolerance.Functional characterization of human T cell hyporesponsiveness induced by CTLA4-Ig.mTOR at the crossroads of T cell proliferation and tolerance.Interleukin 2 plays a central role in Th2 differentiation.Specific gut commensal flora locally alters T cell tuning to endogenous ligands.IL-2 signaling prevents T cell anergy by inhibiting the expression of anergy-inducing genesmTOR: taking cues from the immune microenvironment.Egr-2 and Egr-3 are negative regulators of T cell activation.p21(Cip1) up-regulated during histone deacetylase inhibitor-induced CD4(+) T-cell anergy selectively associates with mitogen-activated protein kinases.The kinases aurora B and mTOR regulate the G1-S cell cycle progression of T lymphocytes.
P2860
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P2860
TCR engagement in the absence of cell cycle progression leads to T cell anergy independent of p27(Kip1).
description
2001 nî lūn-bûn
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2001年の論文
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2001年学术文章
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2001年学术文章
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name
TCR engagement in the absence ...... ergy independent of p27(Kip1).
@en
type
label
TCR engagement in the absence ...... ergy independent of p27(Kip1).
@en
prefLabel
TCR engagement in the absence ...... ergy independent of p27(Kip1).
@en
P2093
P2860
P1476
TCR engagement in the absence ...... ergy independent of p27(Kip1).
@en
P2093
Bruniquel D
Schwartz RH
P2860
P304
P356
10.1002/1521-4141(200112)31:12<3737::AID-IMMU3737>3.0.CO;2-G
P407
P577
2001-12-01T00:00:00Z