STAT3 as a mediator of BCR-ABL1-independent resistance in chronic myeloid leukemia.
about
Mechanisms of Resistance to ABL Kinase Inhibition in Chronic Myeloid Leukemia and the Development of Next Generation ABL Kinase Inhibitors.Combined targeting of STAT3 and STAT5: a novel approach to overcome drug resistance in chronic myeloid leukemia.Development of artificial bone marrow fibre scaffolds to study resistance to anti-leukaemia agents.Transcriptional Regulation of Emergency Granulopoiesis in Leukemia.
P2860
STAT3 as a mediator of BCR-ABL1-independent resistance in chronic myeloid leukemia.
description
2014 nî lūn-bûn
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2014年の論文
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2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
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2014年论文
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2014年论文
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name
STAT3 as a mediator of BCR-ABL1-independent resistance in chronic myeloid leukemia.
@en
type
label
STAT3 as a mediator of BCR-ABL1-independent resistance in chronic myeloid leukemia.
@en
prefLabel
STAT3 as a mediator of BCR-ABL1-independent resistance in chronic myeloid leukemia.
@en
P2093
P2860
P356
P1433
P1476
STAT3 as a mediator of BCR-ABL1-independent resistance in chronic myeloid leukemia.
@en
P2093
A M Eiring
M W Deininger
P T Gunning
P2860
P2888
P356
10.1038/LEUSUP.2014.3
P433
P577
2014-12-17T00:00:00Z
P6179
1049086911