The aberrant fusion proteins PML-RAR alpha and PLZF-RAR alpha contribute to the overexpression of cyclin A1 in acute promyelocytic leukemia.
about
Loss of expression of HDAC-recruiting methyl-CpG-binding domain proteins in human cancerCyclin-A1 represents a new immunogenic targetable antigen expressed in acute myeloid leukemia stem cells with characteristics of a cancer-testis antigenA Limited Role for the Cell Cycle Regulator Cyclin A1 in Murine LeukemogenesisCancer-testis antigen cyclin A1 is broadly expressed in ovarian cancer and is associated with prolonged time to tumor progression after platinum-based therapy.Altered myelopoiesis and the development of acute myeloid leukemia in transgenic mice overexpressing cyclin A1.Gene and protein analysis reveals that p53 pathway is functionally inactivated in cytogenetically normal Acute Myeloid Leukemia and Acute Promyelocytic LeukemiaThe cyclin A1-CDK2 complex regulates DNA double-strand break repair.Dynamics of epigenetic modifications in leukemia.Translocation products in acute myeloid leukemia activate the Wnt signaling pathway in hematopoietic cells.Dissecting the role of p53 phosphorylation in homologous recombination provides new clues for gain-of-function mutants.
P2860
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P2860
The aberrant fusion proteins PML-RAR alpha and PLZF-RAR alpha contribute to the overexpression of cyclin A1 in acute promyelocytic leukemia.
description
2000 nî lūn-bûn
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name
The aberrant fusion proteins P ...... acute promyelocytic leukemia.
@en
type
label
The aberrant fusion proteins P ...... acute promyelocytic leukemia.
@en
prefLabel
The aberrant fusion proteins P ...... acute promyelocytic leukemia.
@en
P2093
P1433
P1476
The aberrant fusion proteins P ...... acute promyelocytic leukemia.
@en
P2093
P304
P407
P577
2000-12-01T00:00:00Z