Cutting edge: guinea pigs with a natural C3a-receptor defect exhibit decreased bronchoconstriction in allergic airway disease: evidence for an involvement of the C3a anaphylatoxin in the pathogenesis of asthma.
about
Anaphylatoxin C3a receptors in asthmaComplement C3a and C4a increased in plasma of patients with aspirin-induced asthmaRegulation of C3a receptor signaling in human mast cells by G protein coupled receptor kinases.Regulation of human mast cell and basophil function by anaphylatoxins C3a and C5a.Lack of association of C3 gene with uveitis: additional insights into the genetic profile of uveitis regarding complement pathway genes.Understanding asthma using animal modelsComplement and the alternative pathway play an important role in LPS/D-GalN-induced fulminant hepatic failure.A protective role for C5a in the development of allergic asthma associated with altered levels of B7-H1 and B7-DC on plasmacytoid dendritic cellsRole and mechanism of action of complement in regulating T cell immunity.Complement system in lung disease.A regulatory role for the C5a anaphylatoxin in type 2 immunity in asthma.Factor B of the alternative complement pathway regulates development of airway hyperresponsiveness and inflammationEarly neoplastic progression is complement independent.The role of the anaphylatoxins in health and disease.Allergens induce enhanced bronchoconstriction and leukotriene production in C5 deficient mice.C5 modulates airway hyperreactivity and pulmonary eosinophilia during enhanced respiratory syncytial virus disease by decreasing C3a receptor expressionInterleukin-13 in asthma pathogenesis.New insights into the role of the complement pathway in allergy and asthma.Complement activation and cardiac surgery: a novel target for improving outcomes.Complement C3a regulates Muc5ac expression by airway Clara cells independently of Th2 responses.Essential role for the C5a receptor in regulating the effector phase of synovial infiltration and joint destruction in experimental arthritis.The impact of comorbid atopic disease on asthma: clinical expression and treatment.The anaphylatoxin C3a downregulates the Th2 response to epicutaneously introduced antigen.Complement activation pathways: a bridge between innate and adaptive immune responses in asthma.Transcriptional control of complement receptor gene expression.In vitro allergen challenge of peripheral blood induces differential gene expression in mononuclear cells of asthmatic patients: inhibition of cytosolic phospholipase A2alpha overcomes the asthma-associated response.A protective role for the fifth complement component (c5) in allergic airway disease.C3a receptor deficiency accelerates the onset of renal injury in the MRL/lpr mouse.Complement activation and disease: protective effects of hyperbilirubinaemia.A complex role for complement in allergic asthma.The role of complement in the diagnosis and management of allergic rhinitis and allergic asthma.The transcription factors AP-1 and Ets are regulators of C3a receptor expression.Th17 cytokines are critical for respiratory syncytial virus-associated airway hyperreponsiveness through regulation by complement C3a and tachykinins.Old dogs-new tricks: immunoregulatory properties of C3 and C5 cleavage fragments.Association study of the C3 gene with adult and childhood asthma.Complement-mediated regulation of the IL-17A axis is a central genetic determinant of the severity of experimental allergic asthma.Airway hyper-reactivity mediated by B-1 cell immunoglobulin M antibody generating complement C5a at 1 day post-immunization in a murine hapten model of non-atopic asthma.A novel promoter polymorphism in the gene encoding complement component 5 receptor 1 on chromosome 19q13.3 is not associated with asthma and atopy in three independent populations.Mannose-binding lectin levels in children with asthma.Contribution of anaphylatoxins to allergic inflammation in human lungs.
P2860
Q21093657-FF50FF44-1E01-45EB-8AF5-F66AD52CC68EQ28193417-8AF5DAD1-9D96-4B06-835F-71056D7DFC7DQ31028372-B6F8718E-582E-4661-BC26-403800374396Q33625259-20199B3D-3365-4B38-9237-D8940EA9C061Q33678499-B9DC54E8-0299-4E3B-BF41-8AD4D8C65731Q33701214-2A8FD5D6-4A0A-4394-9A62-E8AEABE313F5Q34070523-BB9EC381-8F3B-45C2-B997-84CB33390A7BQ34072341-1C68E0FF-999E-4CB9-85C1-8667E633B1F8Q34072846-23C8663A-80ED-4E59-881A-1A8F31037441Q34305533-C1956618-5C5F-4BE7-BBA9-C97F6D10C1FFQ34416391-7D262F25-5BB7-4E4A-BFD4-5A57E3FE9072Q34650129-58518B49-C724-4301-805F-192B6DD46970Q34928831-766FC0C6-9C6B-4261-9617-A5EE1C6B52CCQ34983578-D358A51D-21C3-46F5-98DB-BFCCCB7FD887Q35129075-CD57389E-299D-4193-A2CE-92D14C344740Q35634924-D559722C-047F-4D93-8E00-C16E8DF44068Q35652268-DC34EC30-50E7-4684-9B97-F780FCBC15BEQ36223658-86B45124-CB28-4301-9383-5A62F40CB6CFQ36269099-6D7C2D55-0B8B-477E-9ED0-BD31AD52DF17Q36330548-467590F6-507D-4976-9966-8284ABCB9BD4Q36371879-BEB1D293-71A9-49FC-A7CF-8196D990C00EQ36799761-08B0C698-5F2E-44B1-858C-5E90A760D542Q36849001-90E37ADE-2764-4860-B6E6-BE8DFF03B0F8Q36868284-08E9EEE6-8815-4F08-A4BD-ED71FBA7A50EQ36961063-F59D8E1B-D67E-485D-B45A-7C63728BBAA7Q37035482-2A226B38-43F5-46EB-9B1E-02370F3FE70BQ37144283-AC45A992-1CFA-4EAE-B448-D439BCD2546BQ37230515-52841996-F6DA-4A91-AC9C-DE185B9540A3Q37609484-2A94E7AA-DB6F-4AE8-99DE-D8CB653D397FQ37733520-3DE2549C-0F9E-4B86-9D5E-879C8DE5690DQ37822299-E6AD9F34-F98E-4F25-9594-0787F33E72F1Q38319410-35A37EB3-AD4F-4645-AA1C-BFB373130679Q38625810-2424A941-926A-4BD5-BE1B-BAEF80CDB201Q38991048-2093253C-6761-4502-BFB8-4CC611AFEB49Q39850367-892D430C-81F1-480E-819D-245C9B203189Q42423633-537CFC95-4FF4-431C-9EE5-3B6CBE454815Q45067688-E83B747F-028E-4F8F-BBCB-21B3D4AC1884Q46398747-637EBEE6-303F-4A4F-83AC-A6440ECD6BC9Q46457418-839A4CD3-E21B-46B1-92A5-AEB89D45099BQ46815105-3D705C59-12E9-485F-B176-067656274876
P2860
Cutting edge: guinea pigs with a natural C3a-receptor defect exhibit decreased bronchoconstriction in allergic airway disease: evidence for an involvement of the C3a anaphylatoxin in the pathogenesis of asthma.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年学术文章
@wuu
2000年学术文章
@zh-cn
2000年学术文章
@zh-hans
2000年学术文章
@zh-my
2000年学术文章
@zh-sg
2000年學術文章
@yue
2000年學術文章
@zh
2000年學術文章
@zh-hant
name
Cutting edge: guinea pigs with ...... in the pathogenesis of asthma.
@en
type
label
Cutting edge: guinea pigs with ...... in the pathogenesis of asthma.
@en
prefLabel
Cutting edge: guinea pigs with ...... in the pathogenesis of asthma.
@en
P2093
P1476
Cutting edge: guinea pigs with ...... in the pathogenesis of asthma.
@en
P2093
Hoymann HG
Meier-Wiedenbach I
Meyer zu Vilsendorf A
P304
P356
10.4049/JIMMUNOL.165.10.5401
P407
P577
2000-11-01T00:00:00Z